---
DOAJ_listed: '1'
OA_place: publisher
OA_type: gold
_id: '21708'
abstract:
- lang: eng
text: On October 4, 2023, a proglacial lake named the South Lhonak lake was the
source of a catastrophic Glacier Lake Outburst Flood (GLOF) in the Teesta river
basin area, resulting in 24 fatalities and leaving over 70 persons missing. The
GLOF also destroyed 13 bridges and a major hydropower plant in the Chungthang
region. Over 60,000 individuals in four districts of Sikkim were impacted by this
GLOF event. This study examines the factors that led to the GLOF event. Our study
shows that the cause of this GLOF was initiated by a landslide, that dumped a
substantial amount (~ 38.31 million m3) of debris into the South Lhonak Lake.
Furthermore, the glacier that was connected to the lake, lost a big chunk of ice
mass (~ 7 million m3) due to calving. The combination of these two processes led
to the collapse of the left lateral moraine that consequently generated flood
waves which breached the terminal moraine dam of the lake. We recommend monitoring
land subsidence and calving events for large proglacial lakes to prevent the disastrous
consequences of such GLOFs in the future.
acknowledgement: This work was carried out independently without the support of any
funding agency or sponsors. The authors thank the SARPROZ team for providing an
evaluation license for the MTInSAR processing software.
article_number: '9741'
article_processing_charge: Yes
article_type: original
author:
- first_name: Litan Kumar
full_name: Mohanty, Litan Kumar
last_name: Mohanty
- first_name: Prateek
full_name: Gantayat, Prateek
id: 02734268-3e8d-11ef-80a1-cec4a088d004
last_name: Gantayat
- first_name: Ankur
full_name: Dixit, Ankur
last_name: Dixit
- first_name: Manik
full_name: Das Adhikari, Manik
last_name: Das Adhikari
- first_name: Rahul
full_name: Biswas, Rahul
last_name: Biswas
- first_name: Vivek Kumar
full_name: Singh, Vivek Kumar
last_name: Singh
citation:
ama: Mohanty LK, GANTAYAT P, Dixit A, Das Adhikari M, Biswas R, Singh VK. Sequence
of events that led to the South Lhonak lake outburst flood in Sikkim, India. Scientific
Reports. 2026;16. doi:10.1038/s41598-026-35895-7
apa: Mohanty, L. K., GANTAYAT, P., Dixit, A., Das Adhikari, M., Biswas, R., &
Singh, V. K. (2026). Sequence of events that led to the South Lhonak lake outburst
flood in Sikkim, India. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-026-35895-7
chicago: Mohanty, Litan Kumar, PRATEEK GANTAYAT, Ankur Dixit, Manik Das Adhikari,
Rahul Biswas, and Vivek Kumar Singh. “Sequence of Events That Led to the South
Lhonak Lake Outburst Flood in Sikkim, India.” Scientific Reports. Springer
Nature, 2026. https://doi.org/10.1038/s41598-026-35895-7.
ieee: L. K. Mohanty, P. GANTAYAT, A. Dixit, M. Das Adhikari, R. Biswas, and V. K.
Singh, “Sequence of events that led to the South Lhonak lake outburst flood in
Sikkim, India,” Scientific Reports, vol. 16. Springer Nature, 2026.
ista: Mohanty LK, GANTAYAT P, Dixit A, Das Adhikari M, Biswas R, Singh VK. 2026.
Sequence of events that led to the South Lhonak lake outburst flood in Sikkim,
India. Scientific Reports. 16, 9741.
mla: Mohanty, Litan Kumar, et al. “Sequence of Events That Led to the South Lhonak
Lake Outburst Flood in Sikkim, India.” Scientific Reports, vol. 16, 9741,
Springer Nature, 2026, doi:10.1038/s41598-026-35895-7.
short: L.K. Mohanty, P. GANTAYAT, A. Dixit, M. Das Adhikari, R. Biswas, V.K. Singh,
Scientific Reports 16 (2026).
corr_author: '1'
date_created: 2026-04-12T22:01:48Z
date_published: 2026-03-24T00:00:00Z
date_updated: 2026-05-04T07:54:53Z
day: '24'
ddc:
- '550'
department:
- _id: FrPe
doi: 10.1038/s41598-026-35895-7
external_id:
pmid:
- '41876546'
file:
- access_level: open_access
checksum: cf13f61c38609ce6518d74562319c35f
content_type: application/pdf
creator: dernst
date_created: 2026-05-04T07:24:59Z
date_updated: 2026-05-04T07:24:59Z
file_id: '21785'
file_name: 2026_ScienceAdv_Mohanty.pdf
file_size: 17406006
relation: main_file
success: 1
file_date_updated: 2026-05-04T07:24:59Z
has_accepted_license: '1'
intvolume: ' 16'
language:
- iso: eng
license: https://creativecommons.org/licenses/by-nc-nd/4.0/
month: '03'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Sequence of events that led to the South Lhonak lake outburst flood in Sikkim,
India
tmp:
image: /images/cc_by_nc_nd.png
legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
(CC BY-NC-ND 4.0)
short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 16
year: '2026'
...
---
DOAJ_listed: '1'
OA_place: publisher
OA_type: gold
_id: '19366'
abstract:
- lang: eng
text: Staphylococcus aureus (S. aureus) is one of the most common causative agents
of mammary gland infection and mastitis, but the specific role of S. aureus-derived
extracellular vesicles (SaEVs) in mastitis has been poorly studied to date. Here,
we aimed to investigate the response of bovine monocyte-derived macrophages (boMdM)
to SaEVs of the genotype B (GTB) mastitis-related strain M5512B. Specifically,
we evaluated the effects on the actin cytoskeleton, gene expression, and the SaEV
proteomic cargo. Furthermore, we assessed to what extent the cellular and molecular
response of boMdM to SaEVs differed from peripheral mononuclear blood cells (PBMCs)
used for in vitro derivation of the former. We observed that SaEVs induced morphological
changes in boMdM, leading to a pro-inflammatory and pyroptosis-related increased
gene expression. Additionally, our study revealed that boMdM and PBMCs exhibited
stimulus-specific differing responses. The proteomic analysis of SaEVs identified
clusters of proteins related to virulence and antibiotic resistance, supporting
the theory that S. aureus might use EVs to evade host defences and colonize the
mammary gland. Our results bring new insights into how SaEVs might impact the
host during an S. aureus infection, which can be useful for future S. aureus vaccine
development.
acknowledgement: "The authors thank Michele Guastalla for his contributions to the
boMdM analyses and Stephan Handschin from the Scientific Center for Optical and
Electron Microscopy (ScopeM) of ETH Zurich for the TEM imaging. We gratefully acknowledge
the Functional Genomics Center Zurich (FGCZ) for performing the mass spectrometry
analysis for this study.\r\nOpen access funding provided by Swiss Federal Institute
of Technology Zurich. This work was supported by basic funding from ETH Zurich."
article_processing_charge: Yes
article_type: original
author:
- first_name: Mara D.
full_name: Saenz-De-Juano, Mara D.
last_name: Saenz-De-Juano
- first_name: Giulia
full_name: Silvestrelli, Giulia
id: 12632ae8-799e-11ef-94a2-e5a3b5ef49e9
last_name: Silvestrelli
- first_name: Samuel
full_name: Buri, Samuel
last_name: Buri
- first_name: Léa V.
full_name: Zinsli, Léa V.
last_name: Zinsli
- first_name: Mathias
full_name: Schmelcher, Mathias
last_name: Schmelcher
- first_name: Susanne E.
full_name: Ulbrich, Susanne E.
last_name: Ulbrich
citation:
ama: Saenz-De-Juano MD, Silvestrelli G, Buri S, Zinsli LV, Schmelcher M, Ulbrich
SE. Mastitis-related Staphylococcus aureus-derived extracellular vesicles induce
a pro-inflammatory response in bovine monocyte-derived macrophages. Scientific
Reports. 2025;15:6059. doi:10.1038/s41598-025-90466-6
apa: Saenz-De-Juano, M. D., Silvestrelli, G., Buri, S., Zinsli, L. V., Schmelcher,
M., & Ulbrich, S. E. (2025). Mastitis-related Staphylococcus aureus-derived
extracellular vesicles induce a pro-inflammatory response in bovine monocyte-derived
macrophages. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-025-90466-6
chicago: Saenz-De-Juano, Mara D., Giulia Silvestrelli, Samuel Buri, Léa V. Zinsli,
Mathias Schmelcher, and Susanne E. Ulbrich. “Mastitis-Related Staphylococcus Aureus-Derived
Extracellular Vesicles Induce a pro-Inflammatory Response in Bovine Monocyte-Derived
Macrophages.” Scientific Reports. Springer Nature, 2025. https://doi.org/10.1038/s41598-025-90466-6.
ieee: M. D. Saenz-De-Juano, G. Silvestrelli, S. Buri, L. V. Zinsli, M. Schmelcher,
and S. E. Ulbrich, “Mastitis-related Staphylococcus aureus-derived extracellular
vesicles induce a pro-inflammatory response in bovine monocyte-derived macrophages,”
Scientific Reports, vol. 15. Springer Nature, p. 6059, 2025.
ista: Saenz-De-Juano MD, Silvestrelli G, Buri S, Zinsli LV, Schmelcher M, Ulbrich
SE. 2025. Mastitis-related Staphylococcus aureus-derived extracellular vesicles
induce a pro-inflammatory response in bovine monocyte-derived macrophages. Scientific
Reports. 15, 6059.
mla: Saenz-De-Juano, Mara D., et al. “Mastitis-Related Staphylococcus Aureus-Derived
Extracellular Vesicles Induce a pro-Inflammatory Response in Bovine Monocyte-Derived
Macrophages.” Scientific Reports, vol. 15, Springer Nature, 2025, p. 6059,
doi:10.1038/s41598-025-90466-6.
short: M.D. Saenz-De-Juano, G. Silvestrelli, S. Buri, L.V. Zinsli, M. Schmelcher,
S.E. Ulbrich, Scientific Reports 15 (2025) 6059.
date_created: 2025-03-09T23:01:26Z
date_published: 2025-02-19T00:00:00Z
date_updated: 2025-09-30T10:58:59Z
day: '19'
ddc:
- '570'
department:
- _id: LoSw
doi: 10.1038/s41598-025-90466-6
external_id:
isi:
- '001426697000031'
pmid:
- '39972051'
file:
- access_level: open_access
checksum: 51b55ae299de1fa126016a11024b499a
content_type: application/pdf
creator: dernst
date_created: 2025-03-10T12:00:34Z
date_updated: 2025-03-10T12:00:34Z
file_id: '19380'
file_name: 2025_ScientificReports_SaenzdeJuano.pdf
file_size: 2780316
relation: main_file
success: 1
file_date_updated: 2025-03-10T12:00:34Z
has_accepted_license: '1'
intvolume: ' 15'
isi: 1
language:
- iso: eng
license: https://creativecommons.org/licenses/by/4.0/
month: '02'
oa: 1
oa_version: Published Version
page: '6059'
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Mastitis-related Staphylococcus aureus-derived extracellular vesicles induce
a pro-inflammatory response in bovine monocyte-derived macrophages
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 317138e5-6ab7-11ef-aa6d-ffef3953e345
volume: 15
year: '2025'
...
---
DOAJ_listed: '1'
OA_place: publisher
OA_type: gold
_id: '19529'
abstract:
- lang: eng
text: NRF2 is a transcription factor responsible for coordinating the expression
of over a thousand cytoprotective genes. Although NRF2 is constitutively expressed,
its stability is modulated by the redox-sensitive protein KEAP1 and other conditional
binding partner regulators. The new era of NRF2 research has highlighted the cooperation
between NRF2 and PIN1 in modifying its cytoprotective effect. Despite numerous
studies, the understanding of the PIN1-NRF2 interaction remains limited. Herein,
we described the binding interaction of PIN1 and three different 14-mer long phospho-peptides
mimicking NRF2 protein using computer-based, biophysical, and biochemical approaches.
According to our computational analyses, the residues positioned in the WW domain
of PIN1 (Ser16, Arg17, Ser18, Tyr23, Ser32, Gln33, and Trp34) were found to be
crucial for PIN1-NRF2 interactions. Biophysical FP assays were used to verify
the computational prediction. The data demonstrated that Pintide, a peptide predominantly
interacting with the PIN1 WW-domain, led to a significant reduction in the binding
affinity of the NRF2 mimicking peptides. Moreover, we evaluated the impact of
known PIN1 inhibitors (juglone, KPT-6566, and EGCG) on the PIN1-NRF2 interaction.
Among the inhibitors, KPT-6566 showed the most potent inhibitory effect on PIN1-NRF2
interaction within an IC50 range of 0.3–1.4 µM. Furthermore,
our mass spectrometry analyses showed that KPT-6566 appeared to covalently modify
PIN1 via conjugate addition, rather than disulfide exchange of the sulfonyl-acetate
moiety. Altogether, such inhibitors would also be highly valuable molecular probes
for further investigation of PIN1 regulation of NRF2 in the cellular context and
potentially pave the way for drug molecules that specifically inhibit the cytoprotective
effects of NRF2 in cancer.
acknowledgement: The authors would like to thank the Ministry of National Education
of Republic of Türkiye within the scope of the YLSY scholarship program for funding
(AO). This article is based upon work from COST Action CA20121, supported by COST
(European Cooperation in Science and Technology) (www.cost.eu) (https://benbedphar.org/about-benbedphar/).
The molecular dynamics simulations reported in this paper were performed at TUBITAK
ULAKBIM, High Performance and Grid Computing Center (TRUBA resources). The authors
thank Dr Sharad Mistry for his support in acquiring and processing the MS data.
article_number: '8907'
article_processing_charge: Yes
article_type: original
author:
- first_name: Adem
full_name: Ozleyen, Adem
last_name: Ozleyen
- first_name: Gizem Nur
full_name: Duran, Gizem Nur
last_name: Duran
- first_name: Serhat
full_name: Dönmez, Serhat
id: 7c624079-3200-11ee-973b-9fcc8a575580
last_name: Dönmez
- first_name: Mehmet
full_name: Ozbil, Mehmet
last_name: Ozbil
- first_name: Richard G.
full_name: Doveston, Richard G.
last_name: Doveston
- first_name: Tugba Boyunegmez
full_name: Tumer, Tugba Boyunegmez
last_name: Tumer
citation:
ama: Ozleyen A, Duran GN, Dönmez S, Ozbil M, Doveston RG, Tumer TB. Identification
and inhibition of PIN1-NRF2 protein–protein interactions through computational
and biophysical approaches. Scientific Reports. 2025;15. doi:10.1038/s41598-025-89342-0
apa: Ozleyen, A., Duran, G. N., Dönmez, S., Ozbil, M., Doveston, R. G., & Tumer,
T. B. (2025). Identification and inhibition of PIN1-NRF2 protein–protein interactions
through computational and biophysical approaches. Scientific Reports. Springer
Nature. https://doi.org/10.1038/s41598-025-89342-0
chicago: Ozleyen, Adem, Gizem Nur Duran, Serhat Dönmez, Mehmet Ozbil, Richard G.
Doveston, and Tugba Boyunegmez Tumer. “Identification and Inhibition of PIN1-NRF2
Protein–Protein Interactions through Computational and Biophysical Approaches.”
Scientific Reports. Springer Nature, 2025. https://doi.org/10.1038/s41598-025-89342-0.
ieee: A. Ozleyen, G. N. Duran, S. Dönmez, M. Ozbil, R. G. Doveston, and T. B. Tumer,
“Identification and inhibition of PIN1-NRF2 protein–protein interactions through
computational and biophysical approaches,” Scientific Reports, vol. 15.
Springer Nature, 2025.
ista: Ozleyen A, Duran GN, Dönmez S, Ozbil M, Doveston RG, Tumer TB. 2025. Identification
and inhibition of PIN1-NRF2 protein–protein interactions through computational
and biophysical approaches. Scientific Reports. 15, 8907.
mla: Ozleyen, Adem, et al. “Identification and Inhibition of PIN1-NRF2 Protein–Protein
Interactions through Computational and Biophysical Approaches.” Scientific
Reports, vol. 15, 8907, Springer Nature, 2025, doi:10.1038/s41598-025-89342-0.
short: A. Ozleyen, G.N. Duran, S. Dönmez, M. Ozbil, R.G. Doveston, T.B. Tumer, Scientific
Reports 15 (2025).
date_created: 2025-04-08T11:12:20Z
date_published: 2025-03-14T00:00:00Z
date_updated: 2025-09-30T11:33:37Z
day: '14'
ddc:
- '570'
department:
- _id: LeSa
doi: 10.1038/s41598-025-89342-0
external_id:
isi:
- '001445507400002'
pmid:
- '40087364'
file:
- access_level: open_access
checksum: 6124a10402a67b66364cfa9350d35b4b
content_type: application/pdf
creator: dernst
date_created: 2025-04-10T06:21:11Z
date_updated: 2025-04-10T06:21:11Z
file_id: '19537'
file_name: 2025_ScientificReports_Ozleyen.pdf
file_size: 5333058
relation: main_file
success: 1
file_date_updated: 2025-04-10T06:21:11Z
has_accepted_license: '1'
intvolume: ' 15'
isi: 1
language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Identification and inhibition of PIN1-NRF2 protein–protein interactions through
computational and biophysical approaches
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 317138e5-6ab7-11ef-aa6d-ffef3953e345
volume: 15
year: '2025'
...
---
_id: '12329'
abstract:
- lang: eng
text: In this article, we develop two independent and new approaches to model epidemic
spread in a network. Contrary to the most studied models, those developed here
allow for contacts with different probabilities of transmitting the disease (transmissibilities).
We then examine each of these models using some mean field type approximations.
The first model looks at the late-stage effects of an epidemic outbreak and allows
for the computation of the probability that a given vertex was infected. This
computation is based on a mean field approximation and only depends on the number
of contacts and their transmissibilities. This approach shares many similarities
with percolation models in networks. The second model we develop is a dynamic
model which we analyze using a mean field approximation which highly reduces the
dimensionality of the system. In particular, the original system which individually
analyses each vertex of the network is reduced to one with as many equations as
different transmissibilities. Perhaps the greatest contribution of this article
is the observation that, in both these models, the existence and size of an epidemic
outbreak are linked to the properties of a matrix which we call the R-matrix.
This is a generalization of the basic reproduction number which more precisely
characterizes the main routes of infection.
acknowledgement: Gonçalo Oliveira is supported by the NOMIS Foundation, Fundação Serrapilheira
1812-27395, by CNPq grants 428959/2018-0 and 307475/2018-2, and by FAPERJ through
the grant Jovem Cientista do Nosso Estado E-26/202.793/2019.
article_number: '468'
article_processing_charge: No
article_type: original
author:
- first_name: Arturo
full_name: Gómez, Arturo
last_name: Gómez
- first_name: Goncalo
full_name: Oliveira, Goncalo
id: 58abbde8-f455-11eb-a497-98c8fd71b905
last_name: Oliveira
citation:
ama: Gómez A, Oliveira G. New approaches to epidemic modeling on networks. Scientific
Reports. 2023;13. doi:10.1038/s41598-022-19827-9
apa: Gómez, A., & Oliveira, G. (2023). New approaches to epidemic modeling on
networks. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-022-19827-9
chicago: Gómez, Arturo, and Goncalo Oliveira. “New Approaches to Epidemic Modeling
on Networks.” Scientific Reports. Springer Nature, 2023. https://doi.org/10.1038/s41598-022-19827-9.
ieee: A. Gómez and G. Oliveira, “New approaches to epidemic modeling on networks,”
Scientific Reports, vol. 13. Springer Nature, 2023.
ista: Gómez A, Oliveira G. 2023. New approaches to epidemic modeling on networks.
Scientific Reports. 13, 468.
mla: Gómez, Arturo, and Goncalo Oliveira. “New Approaches to Epidemic Modeling on
Networks.” Scientific Reports, vol. 13, 468, Springer Nature, 2023, doi:10.1038/s41598-022-19827-9.
short: A. Gómez, G. Oliveira, Scientific Reports 13 (2023).
corr_author: '1'
date_created: 2023-01-22T23:00:55Z
date_published: 2023-01-10T00:00:00Z
date_updated: 2024-10-09T21:03:29Z
day: '10'
ddc:
- '510'
department:
- _id: TaHa
doi: 10.1038/s41598-022-19827-9
external_id:
isi:
- '001003345000051'
file:
- access_level: open_access
checksum: a8b83739f4a951e83e0b2a778f03b327
content_type: application/pdf
creator: dernst
date_created: 2023-01-23T07:53:23Z
date_updated: 2023-01-23T07:53:23Z
file_id: '12336'
file_name: 2023_ScientificReports_Gomez.pdf
file_size: 2167792
relation: main_file
success: 1
file_date_updated: 2023-01-23T07:53:23Z
has_accepted_license: '1'
intvolume: ' 13'
isi: 1
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: New approaches to epidemic modeling on networks
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 13
year: '2023'
...
---
_id: '13166'
abstract:
- lang: eng
text: Brachyury, a member of T-box gene family, is widely known for its major role
in mesoderm specification in bilaterians. It is also present in non-bilaterian
metazoans, such as cnidarians, where it acts as a component of an axial patterning
system. In this study, we present a phylogenetic analysis of Brachyury genes within
phylum Cnidaria, investigate differential expression and address a functional
framework of Brachyury paralogs in hydrozoan Dynamena pumila. Our analysis indicates
two duplication events of Brachyury within the cnidarian lineage. The first duplication
likely appeared in the medusozoan ancestor, resulting in two copies in medusozoans,
while the second duplication arose in the hydrozoan ancestor, resulting in three
copies in hydrozoans. Brachyury1 and 2 display a conservative expression pattern
marking the oral pole of the body axis in D. pumila. On the contrary, Brachyury3
expression was detected in scattered presumably nerve cells of the D. pumila larva.
Pharmacological modulations indicated that Brachyury3 is not under regulation
of cWnt signaling in contrast to the other two Brachyury genes. Divergence in
expression patterns and regulation suggest neofunctionalization of Brachyury3
in hydrozoans.
acknowledgement: "We thank N.A. Pertsov White Sea Biological Station of Moscow State
University for the help and support in obtaining samples and providing access to
all required facilities and equipment of the “Center of Microscopy WSBS MSU”. We
are grateful to Dr. Amro Hamdoun for pCS2+8 plasmid (Addgene plasmid # 34931).\r\nWork
in the Walentek lab is supported by the Deutsche Forschungsgemeinschaft (DFG) under
the Emmy Noether Programme (grant WA3365/2-2) and under Germany’s Excellence Strategy
(CIBSS-EXC-2189-Project ID 390939984). SK is supported by the project No. 0088-2021-0009
of the Koltzov Institute of Developmental Biology of the RAS. The study of molecular
patterning of D. pumila colony was funded by RFBR, project number 20-04-00978a (to
S.K.)."
article_number: '9382'
article_processing_charge: No
article_type: original
author:
- first_name: Alexandra A.
full_name: Vetrova, Alexandra A.
last_name: Vetrova
- first_name: Daria M.
full_name: Kupaeva, Daria M.
last_name: Kupaeva
- first_name: Alena
full_name: Kizenko, Alena
id: a521c60b-0815-11ed-9b02-b8bd522477c8
last_name: Kizenko
- first_name: Tatiana S.
full_name: Lebedeva, Tatiana S.
last_name: Lebedeva
- first_name: Peter
full_name: Walentek, Peter
last_name: Walentek
- first_name: Nikoloz
full_name: Tsikolia, Nikoloz
last_name: Tsikolia
- first_name: Stanislav V.
full_name: Kremnyov, Stanislav V.
last_name: Kremnyov
citation:
ama: Vetrova AA, Kupaeva DM, Kizenko A, et al. The evolutionary history of Brachyury
genes in Hydrozoa involves duplications, divergence, and neofunctionalization.
Scientific Reports. 2023;13. doi:10.1038/s41598-023-35979-8
apa: Vetrova, A. A., Kupaeva, D. M., Kizenko, A., Lebedeva, T. S., Walentek, P.,
Tsikolia, N., & Kremnyov, S. V. (2023). The evolutionary history of Brachyury
genes in Hydrozoa involves duplications, divergence, and neofunctionalization.
Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-023-35979-8
chicago: Vetrova, Alexandra A., Daria M. Kupaeva, Alena Kizenko, Tatiana S. Lebedeva,
Peter Walentek, Nikoloz Tsikolia, and Stanislav V. Kremnyov. “The Evolutionary
History of Brachyury Genes in Hydrozoa Involves Duplications, Divergence, and
Neofunctionalization.” Scientific Reports. Springer Nature, 2023. https://doi.org/10.1038/s41598-023-35979-8.
ieee: A. A. Vetrova et al., “The evolutionary history of Brachyury genes
in Hydrozoa involves duplications, divergence, and neofunctionalization,” Scientific
Reports, vol. 13. Springer Nature, 2023.
ista: Vetrova AA, Kupaeva DM, Kizenko A, Lebedeva TS, Walentek P, Tsikolia N, Kremnyov
SV. 2023. The evolutionary history of Brachyury genes in Hydrozoa involves duplications,
divergence, and neofunctionalization. Scientific Reports. 13, 9382.
mla: Vetrova, Alexandra A., et al. “The Evolutionary History of Brachyury Genes
in Hydrozoa Involves Duplications, Divergence, and Neofunctionalization.” Scientific
Reports, vol. 13, 9382, Springer Nature, 2023, doi:10.1038/s41598-023-35979-8.
short: A.A. Vetrova, D.M. Kupaeva, A. Kizenko, T.S. Lebedeva, P. Walentek, N. Tsikolia,
S.V. Kremnyov, Scientific Reports 13 (2023).
date_created: 2023-06-25T22:00:46Z
date_published: 2023-06-09T00:00:00Z
date_updated: 2023-08-02T06:17:18Z
day: '09'
ddc:
- '570'
department:
- _id: GradSch
doi: 10.1038/s41598-023-35979-8
external_id:
isi:
- '001006690200045'
pmid:
- '37296138'
file:
- access_level: open_access
checksum: baddf6b2fa9adf88263d4a3b0998f0f2
content_type: application/pdf
creator: dernst
date_created: 2023-06-26T09:58:53Z
date_updated: 2023-06-26T09:58:53Z
file_id: '13170'
file_name: 2023_ScientificReports_Vetrova.pdf
file_size: 4844149
relation: main_file
success: 1
file_date_updated: 2023-06-26T09:58:53Z
has_accepted_license: '1'
intvolume: ' 13'
isi: 1
language:
- iso: eng
month: '06'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: The evolutionary history of Brachyury genes in Hydrozoa involves duplications,
divergence, and neofunctionalization
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 13
year: '2023'
...
---
_id: '10731'
abstract:
- lang: eng
text: Motivated by COVID-19, we develop and analyze a simple stochastic model for
the spread of disease in human population. We track how the number of infected
and critically ill people develops over time in order to estimate the demand that
is imposed on the hospital system. To keep this demand under control, we consider
a class of simple policies for slowing down and reopening society and we compare
their efficiency in mitigating the spread of the virus from several different
points of view. We find that in order to avoid overwhelming of the hospital system,
a policy must impose a harsh lockdown or it must react swiftly (or both). While
reacting swiftly is universally beneficial, being harsh pays off only when the
country is patient about reopening and when the neighboring countries coordinate
their mitigation efforts. Our work highlights the importance of acting decisively
when closing down and the importance of patience and coordination between neighboring
countries when reopening.
acknowledgement: 'K.C. acknowledges support from ERC Consolidator Grant No. (863818:
ForM-SMart). A.P. acknowledges support from FWF Grant No. J-4220. M.A.N. acknowledges
support from Office of Naval Research grant N00014-16-1-2914 and from the John Templeton
Foundation.'
article_number: '1526'
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Jakub
full_name: Svoboda, Jakub
id: 130759D2-D7DD-11E9-87D2-DE0DE6697425
last_name: Svoboda
orcid: 0000-0002-1419-3267
- first_name: Josef
full_name: Tkadlec, Josef
last_name: Tkadlec
- first_name: Andreas
full_name: Pavlogiannis, Andreas
id: 49704004-F248-11E8-B48F-1D18A9856A87
last_name: Pavlogiannis
orcid: 0000-0002-8943-0722
- first_name: Krishnendu
full_name: Chatterjee, Krishnendu
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Martin A.
full_name: Nowak, Martin A.
last_name: Nowak
citation:
ama: Svoboda J, Tkadlec J, Pavlogiannis A, Chatterjee K, Nowak MA. Infection dynamics
of COVID-19 virus under lockdown and reopening. Scientific Reports. 2022;12(1).
doi:10.1038/s41598-022-05333-5
apa: Svoboda, J., Tkadlec, J., Pavlogiannis, A., Chatterjee, K., & Nowak, M.
A. (2022). Infection dynamics of COVID-19 virus under lockdown and reopening.
Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-022-05333-5
chicago: Svoboda, Jakub, Josef Tkadlec, Andreas Pavlogiannis, Krishnendu Chatterjee,
and Martin A. Nowak. “Infection Dynamics of COVID-19 Virus under Lockdown and
Reopening.” Scientific Reports. Springer Nature, 2022. https://doi.org/10.1038/s41598-022-05333-5.
ieee: J. Svoboda, J. Tkadlec, A. Pavlogiannis, K. Chatterjee, and M. A. Nowak, “Infection
dynamics of COVID-19 virus under lockdown and reopening,” Scientific Reports,
vol. 12, no. 1. Springer Nature, 2022.
ista: Svoboda J, Tkadlec J, Pavlogiannis A, Chatterjee K, Nowak MA. 2022. Infection
dynamics of COVID-19 virus under lockdown and reopening. Scientific Reports. 12(1),
1526.
mla: Svoboda, Jakub, et al. “Infection Dynamics of COVID-19 Virus under Lockdown
and Reopening.” Scientific Reports, vol. 12, no. 1, 1526, Springer Nature,
2022, doi:10.1038/s41598-022-05333-5.
short: J. Svoboda, J. Tkadlec, A. Pavlogiannis, K. Chatterjee, M.A. Nowak, Scientific
Reports 12 (2022).
date_created: 2022-02-06T23:01:30Z
date_published: 2022-01-27T00:00:00Z
date_updated: 2025-04-14T07:52:45Z
day: '27'
ddc:
- '570'
department:
- _id: KrCh
doi: 10.1038/s41598-022-05333-5
ec_funded: 1
external_id:
arxiv:
- '2012.15155'
isi:
- '000749198000039'
file:
- access_level: open_access
checksum: 247afd30c173390940f099ead35a28ed
content_type: application/pdf
creator: alisjak
date_created: 2022-02-07T14:57:59Z
date_updated: 2022-02-07T14:57:59Z
file_id: '10744'
file_name: 2022_ScientificReports_Svoboda.pdf
file_size: 2971922
relation: main_file
success: 1
file_date_updated: 2022-02-07T14:57:59Z
has_accepted_license: '1'
intvolume: ' 12'
isi: 1
issue: '1'
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
project:
- _id: 0599E47C-7A3F-11EA-A408-12923DDC885E
call_identifier: H2020
grant_number: '863818'
name: 'Formal Methods for Stochastic Models: Algorithms and Applications'
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Infection dynamics of COVID-19 virus under lockdown and reopening
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 12
year: '2022'
...
---
_id: '10069'
abstract:
- lang: eng
text: 'The extent to which women differ in the course of blood cell counts throughout
pregnancy, and the importance of these changes to pregnancy outcomes has not been
well defined. Here, we develop a series of statistical analyses of repeated measures
data to reveal the degree to which women differ in the course of pregnancy, predict
the changes that occur, and determine the importance of these changes for post-partum
hemorrhage (PPH) which is one of the leading causes of maternal mortality. We
present a prospective cohort of 4082 births recorded at the University Hospital,
Lausanne, Switzerland between 2009 and 2014 where full labour records could be
obtained, along with complete blood count data taken at hospital admission. We
find significant differences, at a [Formula: see text] level, among women in how
blood count values change through pregnancy for mean corpuscular hemoglobin, mean
corpuscular volume, mean platelet volume, platelet count and red cell distribution
width. We find evidence that almost all complete blood count values show trimester-specific
associations with PPH. For example, high platelet count (OR 1.20, 95% CI 1.01-1.53),
high mean platelet volume (OR 1.58, 95% CI 1.04-2.08), and high erythrocyte levels
(OR 1.36, 95% CI 1.01-1.57) in trimester 1 increased PPH, but high values in trimester
3 decreased PPH risk (OR 0.85, 0.79, 0.67 respectively). We show that differences
among women in the course of blood cell counts throughout pregnancy have an important
role in shaping pregnancy outcome and tracking blood count value changes through
pregnancy improves identification of women at increased risk of postpartum hemorrhage.
This study provides greater understanding of the complex changes in blood count
values that occur through pregnancy and provides indicators to guide the stratification
of patients into risk groups.'
acknowledgement: This project was funded by an SNSF Eccellenza Grant to MRR (PCEGP3-181181),
and by core funding from the Institute of Science and Technology Austria. We would
like to thank the participants of the study and all the midwives and doctors for
the computerized obstetrical data.
article_number: '19238'
article_processing_charge: Yes
article_type: original
author:
- first_name: Matthew Richard
full_name: Robinson, Matthew Richard
id: E5D42276-F5DA-11E9-8E24-6303E6697425
last_name: Robinson
orcid: 0000-0001-8982-8813
- first_name: Marion
full_name: Patxot, Marion
last_name: Patxot
- first_name: Miloš
full_name: Stojanov, Miloš
last_name: Stojanov
- first_name: Sabine
full_name: Blum, Sabine
last_name: Blum
- first_name: David
full_name: Baud, David
last_name: Baud
citation:
ama: Robinson MR, Patxot M, Stojanov M, Blum S, Baud D. Postpartum hemorrhage risk
is driven by changes in blood composition through pregnancy. Scientific Reports.
2021;11. doi:10.1038/s41598-021-98411-z
apa: Robinson, M. R., Patxot, M., Stojanov, M., Blum, S., & Baud, D. (2021).
Postpartum hemorrhage risk is driven by changes in blood composition through pregnancy.
Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-021-98411-z
chicago: Robinson, Matthew Richard, Marion Patxot, Miloš Stojanov, Sabine Blum,
and David Baud. “Postpartum Hemorrhage Risk Is Driven by Changes in Blood Composition
through Pregnancy.” Scientific Reports. Springer Nature, 2021. https://doi.org/10.1038/s41598-021-98411-z.
ieee: M. R. Robinson, M. Patxot, M. Stojanov, S. Blum, and D. Baud, “Postpartum
hemorrhage risk is driven by changes in blood composition through pregnancy,”
Scientific Reports, vol. 11. Springer Nature, 2021.
ista: Robinson MR, Patxot M, Stojanov M, Blum S, Baud D. 2021. Postpartum hemorrhage
risk is driven by changes in blood composition through pregnancy. Scientific Reports.
11, 19238.
mla: Robinson, Matthew Richard, et al. “Postpartum Hemorrhage Risk Is Driven by
Changes in Blood Composition through Pregnancy.” Scientific Reports, vol.
11, 19238, Springer Nature, 2021, doi:10.1038/s41598-021-98411-z.
short: M.R. Robinson, M. Patxot, M. Stojanov, S. Blum, D. Baud, Scientific Reports
11 (2021).
corr_author: '1'
date_created: 2021-10-03T22:01:21Z
date_published: 2021-09-28T00:00:00Z
date_updated: 2024-10-09T21:00:57Z
day: '28'
ddc:
- '618'
department:
- _id: MaRo
doi: 10.1038/s41598-021-98411-z
external_id:
isi:
- '000701575500083'
pmid:
- '34584125'
file:
- access_level: open_access
checksum: f002ec22f609f58e1263b79e7f79601e
content_type: application/pdf
creator: cchlebak
date_created: 2021-10-05T14:56:48Z
date_updated: 2021-10-05T14:56:48Z
file_id: '10091'
file_name: 2021_ScientificReports_Robinson.pdf
file_size: 6970368
relation: main_file
success: 1
file_date_updated: 2021-10-05T14:56:48Z
has_accepted_license: '1'
intvolume: ' 11'
isi: 1
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Postpartum hemorrhage risk is driven by changes in blood composition through
pregnancy
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 11
year: '2021'
...
---
DOAJ_listed: '1'
OA_place: publisher
OA_type: gold
_id: '9097'
abstract:
- lang: eng
text: Psoriasis is a chronic inflammatory skin disease clinically characterized
by the appearance of red colored, well-demarcated plaques with thickened skin
and with silvery scales. Recent studies have established the involvement of a
complex signalling network of interactions between cytokines, immune cells and
skin cells called keratinocytes. Keratinocytes form the cells of the outermost
layer of the skin (epidermis). Visible plaques in psoriasis are developed due
to the fast proliferation and unusual differentiation of keratinocyte cells. Despite
that, the exact mechanism of the appearance of these plaques in the cytokine-immune
cell network is not clear. A mathematical model embodying interactions between
key immune cells believed to be involved in psoriasis, keratinocytes and relevant
cytokines has been developed. The complex network formed of these interactions
poses several challenges. Here, we choose to study subnetworks of this complex
network and initially focus on interactions involving TNFα, IL-23/IL-17, and IL-15.
These are chosen based on known evidence of their therapeutic efficacy. In addition,
we explore the role of IL-15 in the pathogenesis of psoriasis and its potential
as a future drug target for a novel treatment option. We perform steady state
analyses for these subnetworks and demonstrate that the interactions between cells,
driven by cytokines could cause the emergence of a psoriasis state (hyper-proliferation
of keratinocytes) when levels of TNFα, IL-23/IL-17 or IL-15 are increased. The
model results explain and support the clinical potentiality of anti-cytokine treatments.
Interestingly, our results suggest different dynamic scenarios underpin the pathogenesis
of psoriasis, depending upon the dominant cytokines of subnetworks. We observed
that the increase in the level of IL-23/IL-17 and IL-15 could lead to psoriasis
via a bistable route, whereas an increase in the level of TNFα would lead to a
monotonic and gradual disease progression. Further, we demonstrate how this insight,
bistability, could be exploited to improve the current therapies and develop novel
treatment strategies for psoriasis.
acknowledgement: RP acknowledges the Department of Science and Technology, India for
the support through the DST-INSPIRE Faculty Award (DST/INSPIRE/04/2015/001939).
This work was supported by the Engineering and Physical Sciences Research Council
(EPSRC), United Kingdom (Grant numbers EP/J018295/1, EP/J018392/1, EP/N014391/1).
The contribution of RP was also supported by the later Grant. This work was generously
supported by the Welcome Trust Institutional Strategic Support Award (204909/Z/16/Z)
too. The contribution of MG was supported by the EPSRC via EP/N014391/1 and a Wellcome
Trust Institutional Strategic Support Award (WT105618MA). The contribution of YA
was generously supported by the Wellcome Trust Institutional Strategic Support Award
(WT105618MA).
article_number: '2204'
article_processing_charge: No
article_type: original
author:
- first_name: Rakesh
full_name: Pandey, Rakesh
last_name: Pandey
- first_name: Yusur
full_name: Al-Nuaimi, Yusur
last_name: Al-Nuaimi
- first_name: Rajiv Kumar
full_name: Mishra, Rajiv Kumar
id: 46CB58F2-F248-11E8-B48F-1D18A9856A87
last_name: Mishra
- first_name: Sarah K.
full_name: Spurgeon, Sarah K.
last_name: Spurgeon
- first_name: Marc
full_name: Goodfellow, Marc
last_name: Goodfellow
citation:
ama: Pandey R, Al-Nuaimi Y, Mishra RK, Spurgeon SK, Goodfellow M. Role of subnetworks
mediated by TNF α, IL-23/IL-17 and IL-15 in a network involved in the pathogenesis
of psoriasis. Scientific Reports. 2021;11. doi:10.1038/s41598-020-80507-7
apa: Pandey, R., Al-Nuaimi, Y., Mishra, R. K., Spurgeon, S. K., & Goodfellow,
M. (2021). Role of subnetworks mediated by TNF α, IL-23/IL-17 and IL-15 in a network
involved in the pathogenesis of psoriasis. Scientific Reports. Springer
Nature. https://doi.org/10.1038/s41598-020-80507-7
chicago: Pandey, Rakesh, Yusur Al-Nuaimi, Rajiv Kumar Mishra, Sarah K. Spurgeon,
and Marc Goodfellow. “Role of Subnetworks Mediated by TNF α, IL-23/IL-17 and IL-15
in a Network Involved in the Pathogenesis of Psoriasis.” Scientific Reports.
Springer Nature, 2021. https://doi.org/10.1038/s41598-020-80507-7.
ieee: R. Pandey, Y. Al-Nuaimi, R. K. Mishra, S. K. Spurgeon, and M. Goodfellow,
“Role of subnetworks mediated by TNF α, IL-23/IL-17 and IL-15 in a network involved
in the pathogenesis of psoriasis,” Scientific Reports, vol. 11. Springer
Nature, 2021.
ista: Pandey R, Al-Nuaimi Y, Mishra RK, Spurgeon SK, Goodfellow M. 2021. Role of
subnetworks mediated by TNF α, IL-23/IL-17 and IL-15 in a network involved in
the pathogenesis of psoriasis. Scientific Reports. 11, 2204.
mla: Pandey, Rakesh, et al. “Role of Subnetworks Mediated by TNF α, IL-23/IL-17
and IL-15 in a Network Involved in the Pathogenesis of Psoriasis.” Scientific
Reports, vol. 11, 2204, Springer Nature, 2021, doi:10.1038/s41598-020-80507-7.
short: R. Pandey, Y. Al-Nuaimi, R.K. Mishra, S.K. Spurgeon, M. Goodfellow, Scientific
Reports 11 (2021).
date_created: 2021-02-07T23:01:12Z
date_published: 2021-01-26T00:00:00Z
date_updated: 2026-04-02T14:16:22Z
day: '26'
ddc:
- '570'
doi: 10.1038/s41598-020-80507-7
external_id:
isi:
- '000667506800004'
pmid:
- '33500449'
file:
- access_level: open_access
checksum: e8a68df48750712671f5c47b0228e531
content_type: application/pdf
creator: dernst
date_created: 2021-02-09T07:33:23Z
date_updated: 2021-02-09T07:33:23Z
file_id: '9106'
file_name: 2021_ScientificReports_Pandey.pdf
file_size: 2885056
relation: main_file
success: 1
file_date_updated: 2021-02-09T07:33:23Z
has_accepted_license: '1'
intvolume: ' 11'
isi: 1
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Role of subnetworks mediated by TNF α, IL-23/IL-17 and IL-15 in a network involved
in the pathogenesis of psoriasis
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 11
year: '2021'
...
---
_id: '9905'
abstract:
- lang: eng
text: Vaccines are thought to be the best available solution for controlling the
ongoing SARS-CoV-2 pandemic. However, the emergence of vaccine-resistant strains
may come too rapidly for current vaccine developments to alleviate the health,
economic and social consequences of the pandemic. To quantify and characterize
the risk of such a scenario, we created a SIR-derived model with initial stochastic
dynamics of the vaccine-resistant strain to study the probability of its emergence
and establishment. Using parameters realistically resembling SARS-CoV-2 transmission,
we model a wave-like pattern of the pandemic and consider the impact of the rate
of vaccination and the strength of non-pharmaceutical intervention measures on
the probability of emergence of a resistant strain. As expected, we found that
a fast rate of vaccination decreases the probability of emergence of a resistant
strain. Counterintuitively, when a relaxation of non-pharmaceutical interventions
happened at a time when most individuals of the population have already been vaccinated
the probability of emergence of a resistant strain was greatly increased. Consequently,
we show that a period of transmission reduction close to the end of the vaccination
campaign can substantially reduce the probability of resistant strain establishment.
Our results suggest that policymakers and individuals should consider maintaining
non-pharmaceutical interventions and transmission-reducing behaviours throughout
the entire vaccination period.
acknowledgement: We thank Alexey Kondrashov, Nick Machnik, Raimundo Julian Saona Urmeneta,
Gasper Tkacik and Nick Barton for fruitful discussions. We also thank participants
of EvoLunch seminar at IST Austria and the internal seminar at the Banco de España
for useful comments. The opinions expressed in this document are exclusively of
the authors and, therefore, do not necessarily coincide with those of the Banco
de España or the Eurosystem. ETD is supported by the Swiss National Science and
Louis Jeantet Foundation. The work of FAK was in part supported by the ERC Consolidator
Grant (771209-CharFL).
article_number: '15729'
article_processing_charge: Yes
article_type: original
author:
- first_name: Simon
full_name: Rella, Simon
id: B4765ACA-AA38-11E9-AC9A-0930E6697425
last_name: Rella
- first_name: Yuliya A.
full_name: Kulikova, Yuliya A.
last_name: Kulikova
- first_name: Emmanouil T.
full_name: Dermitzakis, Emmanouil T.
last_name: Dermitzakis
- first_name: Fyodor
full_name: Kondrashov, Fyodor
id: 44FDEF62-F248-11E8-B48F-1D18A9856A87
last_name: Kondrashov
orcid: 0000-0001-8243-4694
citation:
ama: Rella S, Kulikova YA, Dermitzakis ET, Kondrashov F. Rates of SARS-CoV-2 transmission
and vaccination impact the fate of vaccine-resistant strains. Scientific Reports.
2021;11(1). doi:10.1038/s41598-021-95025-3
apa: Rella, S., Kulikova, Y. A., Dermitzakis, E. T., & Kondrashov, F. (2021).
Rates of SARS-CoV-2 transmission and vaccination impact the fate of vaccine-resistant
strains. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-021-95025-3
chicago: Rella, Simon, Yuliya A. Kulikova, Emmanouil T. Dermitzakis, and Fyodor
Kondrashov. “Rates of SARS-CoV-2 Transmission and Vaccination Impact the Fate
of Vaccine-Resistant Strains.” Scientific Reports. Springer Nature, 2021.
https://doi.org/10.1038/s41598-021-95025-3.
ieee: S. Rella, Y. A. Kulikova, E. T. Dermitzakis, and F. Kondrashov, “Rates of
SARS-CoV-2 transmission and vaccination impact the fate of vaccine-resistant strains,”
Scientific Reports, vol. 11, no. 1. Springer Nature, 2021.
ista: Rella S, Kulikova YA, Dermitzakis ET, Kondrashov F. 2021. Rates of SARS-CoV-2
transmission and vaccination impact the fate of vaccine-resistant strains. Scientific
Reports. 11(1), 15729.
mla: Rella, Simon, et al. “Rates of SARS-CoV-2 Transmission and Vaccination Impact
the Fate of Vaccine-Resistant Strains.” Scientific Reports, vol. 11, no.
1, 15729, Springer Nature, 2021, doi:10.1038/s41598-021-95025-3.
short: S. Rella, Y.A. Kulikova, E.T. Dermitzakis, F. Kondrashov, Scientific Reports
11 (2021).
date_created: 2021-08-15T22:01:26Z
date_published: 2021-07-30T00:00:00Z
date_updated: 2026-07-29T12:57:49Z
day: '30'
ddc:
- '570'
- '610'
department:
- _id: FyKo
doi: 10.1038/s41598-021-95025-3
ec_funded: 1
external_id:
isi:
- '000683329100001'
pmid:
- '34330988'
file:
- access_level: open_access
checksum: ac86892ed17e6724c7251844da5cef5c
content_type: application/pdf
creator: asandaue
date_created: 2021-08-16T11:36:49Z
date_updated: 2021-08-16T11:36:49Z
file_id: '9927'
file_name: 2021_ScientificReports_Rella.pdf
file_size: 3432001
relation: main_file
success: 1
file_date_updated: 2021-08-16T11:36:49Z
has_accepted_license: '1'
intvolume: ' 11'
isi: 1
issue: '1'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Published Version
pmid: 1
project:
- _id: 26580278-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '771209'
name: Characterizing the fitness landscape on population and global scales
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
related_material:
link:
- description: News on IST Website
relation: press_release
url: https://ist.ac.at/en/news/counterintuitive-dynamics-threaten-the-end-of-the-pandemic/
record:
- id: '20811'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Rates of SARS-CoV-2 transmission and vaccination impact the fate of vaccine-resistant
strains
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 11
year: '2021'
...
---
_id: '9997'
abstract:
- lang: eng
text: Indirect reciprocity is a mechanism for the evolution of cooperation based
on social norms. This mechanism requires that individuals in a population observe
and judge each other’s behaviors. Individuals with a good reputation are more
likely to receive help from others. Previous work suggests that indirect reciprocity
is only effective when all relevant information is reliable and publicly available.
Otherwise, individuals may disagree on how to assess others, even if they all
apply the same social norm. Such disagreements can lead to a breakdown of cooperation.
Here we explore whether the predominantly studied ‘leading eight’ social norms
of indirect reciprocity can be made more robust by equipping them with an element
of generosity. To this end, we distinguish between two kinds of generosity. According
to assessment generosity, individuals occasionally assign a good reputation to
group members who would usually be regarded as bad. According to action generosity,
individuals occasionally cooperate with group members with whom they would usually
defect. Using individual-based simulations, we show that the two kinds of generosity
have a very different effect on the resulting reputation dynamics. Assessment
generosity tends to add to the overall noise and allows defectors to invade. In
contrast, a limited amount of action generosity can be beneficial in a few cases.
However, even when action generosity is beneficial, the respective simulations
do not result in full cooperation. Our results suggest that while generosity can
favor cooperation when individuals use the most simple strategies of reciprocity,
it is disadvantageous when individuals use more complex social norms.
acknowledgement: 'This work was supported by the European Research Council CoG 863818
(ForM-SMArt) (to K.C.) and the European Research Council Starting Grant 850529:
E-DIRECT (to C.H.). L.S. received additional partial support by the Austrian Science
Fund (FWF) under Grant Z211-N23 (Wittgenstein Award).'
article_number: '17443'
article_processing_charge: Yes
article_type: original
author:
- first_name: Laura
full_name: Schmid, Laura
id: 38B437DE-F248-11E8-B48F-1D18A9856A87
last_name: Schmid
orcid: 0000-0002-6978-7329
- first_name: Pouya
full_name: Shati, Pouya
last_name: Shati
- first_name: Christian
full_name: Hilbe, Christian
last_name: Hilbe
- first_name: Krishnendu
full_name: Chatterjee, Krishnendu
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
citation:
ama: Schmid L, Shati P, Hilbe C, Chatterjee K. The evolution of indirect reciprocity
under action and assessment generosity. Scientific Reports. 2021;11(1).
doi:10.1038/s41598-021-96932-1
apa: Schmid, L., Shati, P., Hilbe, C., & Chatterjee, K. (2021). The evolution
of indirect reciprocity under action and assessment generosity. Scientific
Reports. Springer Nature. https://doi.org/10.1038/s41598-021-96932-1
chicago: Schmid, Laura, Pouya Shati, Christian Hilbe, and Krishnendu Chatterjee.
“The Evolution of Indirect Reciprocity under Action and Assessment Generosity.”
Scientific Reports. Springer Nature, 2021. https://doi.org/10.1038/s41598-021-96932-1.
ieee: L. Schmid, P. Shati, C. Hilbe, and K. Chatterjee, “The evolution of indirect
reciprocity under action and assessment generosity,” Scientific Reports,
vol. 11, no. 1. Springer Nature, 2021.
ista: Schmid L, Shati P, Hilbe C, Chatterjee K. 2021. The evolution of indirect
reciprocity under action and assessment generosity. Scientific Reports. 11(1),
17443.
mla: Schmid, Laura, et al. “The Evolution of Indirect Reciprocity under Action and
Assessment Generosity.” Scientific Reports, vol. 11, no. 1, 17443, Springer
Nature, 2021, doi:10.1038/s41598-021-96932-1.
short: L. Schmid, P. Shati, C. Hilbe, K. Chatterjee, Scientific Reports 11 (2021).
corr_author: '1'
date_created: 2021-09-11T16:22:02Z
date_published: 2021-08-31T00:00:00Z
date_updated: 2026-08-02T22:30:49Z
day: '31'
ddc:
- '003'
department:
- _id: GradSch
- _id: KrCh
doi: 10.1038/s41598-021-96932-1
ec_funded: 1
external_id:
isi:
- '000692406400018'
pmid:
- '34465830'
file:
- access_level: open_access
checksum: 19df8816cf958b272b85841565c73182
content_type: application/pdf
creator: cchlebak
date_created: 2021-09-13T10:31:21Z
date_updated: 2021-09-13T10:31:21Z
file_id: '10006'
file_name: 2021_ScientificReports_Schmid.pdf
file_size: 2424943
relation: main_file
success: 1
file_date_updated: 2021-09-13T10:31:21Z
has_accepted_license: '1'
intvolume: ' 11'
isi: 1
issue: '1'
keyword:
- Multidisciplinary
language:
- iso: eng
month: '08'
oa: 1
oa_version: Published Version
pmid: 1
project:
- _id: 0599E47C-7A3F-11EA-A408-12923DDC885E
call_identifier: H2020
grant_number: '863818'
name: 'Formal Methods for Stochastic Models: Algorithms and Applications'
- _id: 25F42A32-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: Z211
name: Formal methods for the design and analysis of complex systems
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
related_material:
record:
- id: '10293'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: The evolution of indirect reciprocity under action and assessment generosity
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 11
year: '2021'
...
---
_id: '7632'
abstract:
- lang: eng
text: The posterior parietal cortex (PPC) and frontal motor areas comprise a cortical
network supporting goal-directed behaviour, with functions including sensorimotor
transformations and decision making. In primates, this network links performed
and observed actions via mirror neurons, which fire both when individuals perform
an action and when they observe the same action performed by a conspecific. Mirror
neurons are believed to be important for social learning, but it is not known
whether mirror-like neurons occur in similar networks in other social species,
such as rodents, or if they can be measured in such models using paradigms where
observers passively view a demonstrator. Therefore, we imaged Ca2+ responses in
PPC and secondary motor cortex (M2) while mice performed and observed pellet-reaching
and wheel-running tasks, and found that cell populations in both areas robustly
encoded several naturalistic behaviours. However, neural responses to the same
set of observed actions were absent, although we verified that observer mice were
attentive to performers and that PPC neurons responded reliably to visual cues.
Statistical modelling also indicated that executed actions outperformed observed
actions in predicting neural responses. These results raise the possibility that
sensorimotor action recognition in rodents could take place outside of the parieto-frontal
circuit, and underscore that detecting socially-driven neural coding depends critically
on the species and behavioural paradigm used.
article_number: '5559'
article_processing_charge: No
article_type: original
author:
- first_name: Tuce
full_name: Tombaz, Tuce
last_name: Tombaz
- first_name: Benjamin A.
full_name: Dunn, Benjamin A.
last_name: Dunn
- first_name: Karoline
full_name: Hovde, Karoline
last_name: Hovde
- first_name: Ryan J
full_name: Cubero, Ryan J
id: 850B2E12-9CD4-11E9-837F-E719E6697425
last_name: Cubero
orcid: 0000-0003-0002-1867
- first_name: Bartul
full_name: Mimica, Bartul
last_name: Mimica
- first_name: Pranav
full_name: Mamidanna, Pranav
last_name: Mamidanna
- first_name: Yasser
full_name: Roudi, Yasser
last_name: Roudi
- first_name: Jonathan R.
full_name: Whitlock, Jonathan R.
last_name: Whitlock
citation:
ama: Tombaz T, Dunn BA, Hovde K, et al. Action representation in the mouse parieto-frontal
network. Scientific reports. 2020;10(1). doi:10.1038/s41598-020-62089-6
apa: Tombaz, T., Dunn, B. A., Hovde, K., Cubero, R. J., Mimica, B., Mamidanna, P.,
… Whitlock, J. R. (2020). Action representation in the mouse parieto-frontal network.
Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-020-62089-6
chicago: Tombaz, Tuce, Benjamin A. Dunn, Karoline Hovde, Ryan J Cubero, Bartul Mimica,
Pranav Mamidanna, Yasser Roudi, and Jonathan R. Whitlock. “Action Representation
in the Mouse Parieto-Frontal Network.” Scientific Reports. Springer Nature,
2020. https://doi.org/10.1038/s41598-020-62089-6.
ieee: T. Tombaz et al., “Action representation in the mouse parieto-frontal
network,” Scientific reports, vol. 10, no. 1. Springer Nature, 2020.
ista: Tombaz T, Dunn BA, Hovde K, Cubero RJ, Mimica B, Mamidanna P, Roudi Y, Whitlock
JR. 2020. Action representation in the mouse parieto-frontal network. Scientific
reports. 10(1), 5559.
mla: Tombaz, Tuce, et al. “Action Representation in the Mouse Parieto-Frontal Network.”
Scientific Reports, vol. 10, no. 1, 5559, Springer Nature, 2020, doi:10.1038/s41598-020-62089-6.
short: T. Tombaz, B.A. Dunn, K. Hovde, R.J. Cubero, B. Mimica, P. Mamidanna, Y.
Roudi, J.R. Whitlock, Scientific Reports 10 (2020).
date_created: 2020-04-05T22:00:47Z
date_published: 2020-03-27T00:00:00Z
date_updated: 2026-04-02T14:23:52Z
day: '27'
ddc:
- '570'
department:
- _id: SaSi
doi: 10.1038/s41598-020-62089-6
external_id:
isi:
- '000560406800007'
file:
- access_level: open_access
checksum: e6cfaaaf7986532132934400038b824a
content_type: application/pdf
creator: dernst
date_created: 2020-04-06T10:44:23Z
date_updated: 2020-07-14T12:48:01Z
file_id: '7644'
file_name: 2020_ScientificReports_Tombaz.pdf
file_size: 2621249
relation: main_file
file_date_updated: 2020-07-14T12:48:01Z
has_accepted_license: '1'
intvolume: ' 10'
isi: 1
issue: '1'
language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
publication: Scientific reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Action representation in the mouse parieto-frontal network
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 10
year: '2020'
...
---
_id: '7931'
abstract:
- lang: eng
text: In the course of sample preparation for Next Generation Sequencing (NGS),
DNA is fragmented by various methods. Fragmentation shows a persistent bias with
regard to the cleavage rates of various dinucleotides. With the exception of CpG
dinucleotides the previously described biases were consistent with results of
the DNA cleavage in solution. Here we computed cleavage rates of all dinucleotides
including the methylated CpG and unmethylated CpG dinucleotides using data of
the Whole Genome Sequencing datasets of the 1000 Genomes project. We found that
the cleavage rate of CpG is significantly higher for the methylated CpG dinucleotides.
Using this information, we developed a classifier for distinguishing cancer and
healthy tissues based on their CpG islands statuses of the fragmentation. A simple
Support Vector Machine classifier based on this algorithm shows an accuracy of
84%. The proposed method allows the detection of epigenetic markers purely based
on mechanochemical DNA fragmentation, which can be detected by a simple analysis
of the NGS sequencing data.
article_number: '8635'
article_processing_charge: No
article_type: original
author:
- first_name: Leonid A.
full_name: Uroshlev, Leonid A.
last_name: Uroshlev
- first_name: Eldar T.
full_name: Abdullaev, Eldar T.
last_name: Abdullaev
- first_name: Iren R.
full_name: Umarova, Iren R.
last_name: Umarova
- first_name: Irina A.
full_name: Il’Icheva, Irina A.
last_name: Il’Icheva
- first_name: Larisa A.
full_name: Panchenko, Larisa A.
last_name: Panchenko
- first_name: Robert V.
full_name: Polozov, Robert V.
last_name: Polozov
- first_name: Fyodor
full_name: Kondrashov, Fyodor
id: 44FDEF62-F248-11E8-B48F-1D18A9856A87
last_name: Kondrashov
orcid: 0000-0001-8243-4694
- first_name: Yury D.
full_name: Nechipurenko, Yury D.
last_name: Nechipurenko
- first_name: Sergei L.
full_name: Grokhovsky, Sergei L.
last_name: Grokhovsky
citation:
ama: Uroshlev LA, Abdullaev ET, Umarova IR, et al. A method for identification of
the methylation level of CpG islands from NGS data. Scientific Reports.
2020;10. doi:10.1038/s41598-020-65406-1
apa: Uroshlev, L. A., Abdullaev, E. T., Umarova, I. R., Il’Icheva, I. A., Panchenko,
L. A., Polozov, R. V., … Grokhovsky, S. L. (2020). A method for identification
of the methylation level of CpG islands from NGS data. Scientific Reports.
Springer Nature. https://doi.org/10.1038/s41598-020-65406-1
chicago: Uroshlev, Leonid A., Eldar T. Abdullaev, Iren R. Umarova, Irina A. Il’Icheva,
Larisa A. Panchenko, Robert V. Polozov, Fyodor Kondrashov, Yury D. Nechipurenko,
and Sergei L. Grokhovsky. “A Method for Identification of the Methylation Level
of CpG Islands from NGS Data.” Scientific Reports. Springer Nature, 2020.
https://doi.org/10.1038/s41598-020-65406-1.
ieee: L. A. Uroshlev et al., “A method for identification of the methylation
level of CpG islands from NGS data,” Scientific Reports, vol. 10. Springer
Nature, 2020.
ista: Uroshlev LA, Abdullaev ET, Umarova IR, Il’Icheva IA, Panchenko LA, Polozov
RV, Kondrashov F, Nechipurenko YD, Grokhovsky SL. 2020. A method for identification
of the methylation level of CpG islands from NGS data. Scientific Reports. 10,
8635.
mla: Uroshlev, Leonid A., et al. “A Method for Identification of the Methylation
Level of CpG Islands from NGS Data.” Scientific Reports, vol. 10, 8635,
Springer Nature, 2020, doi:10.1038/s41598-020-65406-1.
short: L.A. Uroshlev, E.T. Abdullaev, I.R. Umarova, I.A. Il’Icheva, L.A. Panchenko,
R.V. Polozov, F. Kondrashov, Y.D. Nechipurenko, S.L. Grokhovsky, Scientific Reports
10 (2020).
date_created: 2020-06-07T22:00:51Z
date_published: 2020-05-25T00:00:00Z
date_updated: 2026-04-03T09:26:06Z
day: '25'
ddc:
- '570'
department:
- _id: FyKo
doi: 10.1038/s41598-020-65406-1
external_id:
isi:
- '000560774200007'
pmid:
- '32451390'
file:
- access_level: open_access
checksum: 099e51611a5b7ca04244d03b2faddf33
content_type: application/pdf
creator: dernst
date_created: 2020-06-08T06:27:32Z
date_updated: 2020-07-14T12:48:05Z
file_id: '7947'
file_name: 2020_ScientificReports_Uroshlev.pdf
file_size: 1001724
relation: main_file
file_date_updated: 2020-07-14T12:48:05Z
has_accepted_license: '1'
intvolume: ' 10'
isi: 1
language:
- iso: eng
month: '05'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: A method for identification of the methylation level of CpG islands from NGS
data
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 10
year: '2020'
...
---
_id: '8643'
abstract:
- lang: eng
text: The parabigeminal nucleus (PBG) is the mammalian homologue to the isthmic
complex of other vertebrates. Optogenetic stimulation of the PBG induces freezing
and escape in mice, a result thought to be caused by a PBG projection to the central
nucleus of the amygdala. However, the isthmic complex, including the PBG, has
been classically considered satellite nuclei of the Superior Colliculus (SC),
which upon stimulation of its medial part also triggers fear and avoidance reactions.
As the PBG-SC connectivity is not well characterized, we investigated whether
the topology of the PBG projection to the SC could be related to the behavioral
consequences of PBG stimulation. To that end, we performed immunohistochemistry,
in situ hybridization and neural tracer injections in the SC and PBG in a diurnal
rodent, the Octodon degus. We found that all PBG neurons expressed both glutamatergic
and cholinergic markers and were distributed in clearly defined anterior (aPBG)
and posterior (pPBG) subdivisions. The pPBG is connected reciprocally and topographically
to the ipsilateral SC, whereas the aPBG receives afferent axons from the ipsilateral
SC and projected exclusively to the contralateral SC. This contralateral projection
forms a dense field of terminals that is restricted to the medial SC, in correspondence
with the SC representation of the aerial binocular field which, we also found,
in O. degus prompted escape reactions upon looming stimulation. Therefore, this
specialized topography allows binocular interactions in the SC region controlling
responses to aerial predators, suggesting a link between the mechanisms by which
the SC and PBG produce defensive behaviors.
acknowledgement: 'We thank Elisa Sentis and Solano Henriquez for their expert technical
assistance. Dr. David Sterratt for his helpful advice in using the Retistruct package.
Dr. Joao Botelho for his valuable assistance in scanning the retinas. To Mrs. Diane
Greenstein for kindly reading and correcting our manuscript. Macarena Ruiz for her
helpful comments during figures elaboration. Dr. Alexia Nunez-Parra for kindly providing
us with the transgenic mouse line. Dr. Harald Luksch for granting us access to the
confocal microscope at his lab. This study was supported by: FONDECYT 1151432 (to
G.M.), FONDECYT 1170027 (to J.M.) and Doctoral fellowship CONICYT 21161599 (to A.D.).'
article_number: '16220'
article_processing_charge: No
article_type: original
author:
- first_name: Alfonso
full_name: Deichler, Alfonso
last_name: Deichler
- first_name: Denisse
full_name: Carrasco, Denisse
last_name: Carrasco
- first_name: Luciana
full_name: Lopez-Jury, Luciana
last_name: Lopez-Jury
- first_name: Tomas A
full_name: Vega Zuniga, Tomas A
id: 2E7C4E78-F248-11E8-B48F-1D18A9856A87
last_name: Vega Zuniga
- first_name: Natalia
full_name: Marquez, Natalia
last_name: Marquez
- first_name: Jorge
full_name: Mpodozis, Jorge
last_name: Mpodozis
- first_name: Gonzalo
full_name: Marin, Gonzalo
last_name: Marin
citation:
ama: Deichler A, Carrasco D, Lopez-Jury L, et al. A specialized reciprocal connectivity
suggests a link between the mechanisms by which the superior colliculus and parabigeminal
nucleus produce defensive behaviors in rodents. Scientific Reports. 2020;10.
doi:10.1038/s41598-020-72848-0
apa: Deichler, A., Carrasco, D., Lopez-Jury, L., Vega Zuniga, T. A., Marquez, N.,
Mpodozis, J., & Marin, G. (2020). A specialized reciprocal connectivity suggests
a link between the mechanisms by which the superior colliculus and parabigeminal
nucleus produce defensive behaviors in rodents. Scientific Reports. Springer
Nature. https://doi.org/10.1038/s41598-020-72848-0
chicago: Deichler, Alfonso, Denisse Carrasco, Luciana Lopez-Jury, Tomas A Vega Zuniga,
Natalia Marquez, Jorge Mpodozis, and Gonzalo Marin. “A Specialized Reciprocal
Connectivity Suggests a Link between the Mechanisms by Which the Superior Colliculus
and Parabigeminal Nucleus Produce Defensive Behaviors in Rodents.” Scientific
Reports. Springer Nature, 2020. https://doi.org/10.1038/s41598-020-72848-0.
ieee: A. Deichler et al., “A specialized reciprocal connectivity suggests
a link between the mechanisms by which the superior colliculus and parabigeminal
nucleus produce defensive behaviors in rodents,” Scientific Reports, vol.
10. Springer Nature, 2020.
ista: Deichler A, Carrasco D, Lopez-Jury L, Vega Zuniga TA, Marquez N, Mpodozis
J, Marin G. 2020. A specialized reciprocal connectivity suggests a link between
the mechanisms by which the superior colliculus and parabigeminal nucleus produce
defensive behaviors in rodents. Scientific Reports. 10, 16220.
mla: Deichler, Alfonso, et al. “A Specialized Reciprocal Connectivity Suggests a
Link between the Mechanisms by Which the Superior Colliculus and Parabigeminal
Nucleus Produce Defensive Behaviors in Rodents.” Scientific Reports, vol.
10, 16220, Springer Nature, 2020, doi:10.1038/s41598-020-72848-0.
short: A. Deichler, D. Carrasco, L. Lopez-Jury, T.A. Vega Zuniga, N. Marquez, J.
Mpodozis, G. Marin, Scientific Reports 10 (2020).
date_created: 2020-10-11T22:01:14Z
date_published: 2020-10-01T00:00:00Z
date_updated: 2026-04-03T09:26:41Z
day: '01'
ddc:
- '570'
department:
- _id: MaJö
doi: 10.1038/s41598-020-72848-0
external_id:
isi:
- '000577142600032'
pmid:
- '33004866'
file:
- access_level: open_access
checksum: f6dd99954f1c0ffb4da5a1d2d739bf31
content_type: application/pdf
creator: dernst
date_created: 2020-10-12T12:39:10Z
date_updated: 2020-10-12T12:39:10Z
file_id: '8651'
file_name: 2020_ScientificReport_Deichler.pdf
file_size: 3906744
relation: main_file
success: 1
file_date_updated: 2020-10-12T12:39:10Z
has_accepted_license: '1'
intvolume: ' 10'
isi: 1
language:
- iso: eng
month: '10'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: A specialized reciprocal connectivity suggests a link between the mechanisms
by which the superior colliculus and parabigeminal nucleus produce defensive behaviors
in rodents
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 10
year: '2020'
...
---
_id: '7487'
abstract:
- lang: eng
text: 'Glutaminase (GA) catalyzes the first step in mitochondrial glutaminolysis
playing a key role in cancer metabolic reprogramming. Humans express two types
of GA isoforms: GLS and GLS2. GLS isozymes have been consistently related to cell
proliferation, but the role of GLS2 in cancer remains poorly understood. GLS2
is repressed in many tumor cells and a better understanding of its function in
tumorigenesis may further the development of new therapeutic approaches. We analyzed
GLS2 expression in HCC, GBM and neuroblastoma cells, as well as in monkey COS-7
cells. We studied GLS2 expression after induction of differentiation with phorbol
ester (PMA) and transduction with the full-length cDNA of GLS2. In parallel, we
investigated cell cycle progression and levels of p53, p21 and c-Myc proteins.
Using the baculovirus system, human GLS2 protein was overexpressed, purified and
analyzed for posttranslational modifications employing a proteomics LC-MS/MS platform.
We have demonstrated a dual targeting of GLS2 in human cancer cells. Immunocytochemistry
and subcellular fractionation gave consistent results demonstrating nuclear and
mitochondrial locations, with the latter being predominant. Nuclear targeting
was confirmed in cancer cells overexpressing c-Myc- and GFP-tagged GLS2 proteins.
We assessed the subnuclear location finding a widespread distribution of GLS2
in the nucleoplasm without clear overlapping with specific nuclear substructures.
GLS2 expression and nuclear accrual notably increased by treatment of SH-SY5Y
cells with PMA and it correlated with cell cycle arrest at G2/M, upregulation
of tumor suppressor p53 and p21 protein. A similar response was obtained by overexpression
of GLS2 in T98G glioma cells, including downregulation of oncogene c-Myc. Furthermore,
human GLS2 was identified as being hypusinated by MS analysis, a posttranslational
modification which may be relevant for its nuclear targeting and/or function.
Our studies provide evidence for a tumor suppressor role of GLS2 in certain types
of cancer. The data imply that GLS2 can be regarded as a highly mobile and multilocalizing
protein translocated to both mitochondria and nuclei. Upregulation of GLS2 in
cancer cells induced an antiproliferative response with cell cycle arrest at the
G2/M phase.'
article_number: '2259'
article_processing_charge: No
article_type: original
author:
- first_name: Amada R.
full_name: López De La Oliva, Amada R.
last_name: López De La Oliva
- first_name: José A.
full_name: Campos-Sandoval, José A.
last_name: Campos-Sandoval
- first_name: María C.
full_name: Gómez-García, María C.
last_name: Gómez-García
- first_name: Carolina
full_name: Cardona, Carolina
last_name: Cardona
- first_name: Mercedes
full_name: Martín-Rufián, Mercedes
last_name: Martín-Rufián
- first_name: Fernando J.
full_name: Sialana, Fernando J.
last_name: Sialana
- first_name: Laura
full_name: Castilla, Laura
last_name: Castilla
- first_name: Narkhyun
full_name: Bae, Narkhyun
id: 3A5F7CD8-F248-11E8-B48F-1D18A9856A87
last_name: Bae
- first_name: Carolina
full_name: Lobo, Carolina
last_name: Lobo
- first_name: Ana
full_name: Peñalver, Ana
last_name: Peñalver
- first_name: Marina
full_name: García-Frutos, Marina
last_name: García-Frutos
- first_name: David
full_name: Carro, David
last_name: Carro
- first_name: Victoria
full_name: Enrique, Victoria
last_name: Enrique
- first_name: José C.
full_name: Paz, José C.
last_name: Paz
- first_name: Raghavendra G.
full_name: Mirmira, Raghavendra G.
last_name: Mirmira
- first_name: Antonia
full_name: Gutiérrez, Antonia
last_name: Gutiérrez
- first_name: Francisco J.
full_name: Alonso, Francisco J.
last_name: Alonso
- first_name: Juan A.
full_name: Segura, Juan A.
last_name: Segura
- first_name: José M.
full_name: Matés, José M.
last_name: Matés
- first_name: Gert
full_name: Lubec, Gert
last_name: Lubec
- first_name: Javier
full_name: Márquez, Javier
last_name: Márquez
citation:
ama: López De La Oliva AR, Campos-Sandoval JA, Gómez-García MC, et al. Nuclear translocation
of glutaminase GLS2 in human cancer cells associates with proliferation arrest
and differentiation. Scientific reports. 2020;10(1). doi:10.1038/s41598-020-58264-4
apa: López De La Oliva, A. R., Campos-Sandoval, J. A., Gómez-García, M. C., Cardona,
C., Martín-Rufián, M., Sialana, F. J., … Márquez, J. (2020). Nuclear translocation
of glutaminase GLS2 in human cancer cells associates with proliferation arrest
and differentiation. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-020-58264-4
chicago: López De La Oliva, Amada R., José A. Campos-Sandoval, María C. Gómez-García,
Carolina Cardona, Mercedes Martín-Rufián, Fernando J. Sialana, Laura Castilla,
et al. “Nuclear Translocation of Glutaminase GLS2 in Human Cancer Cells Associates
with Proliferation Arrest and Differentiation.” Scientific Reports. Springer
Nature, 2020. https://doi.org/10.1038/s41598-020-58264-4.
ieee: A. R. López De La Oliva et al., “Nuclear translocation of glutaminase
GLS2 in human cancer cells associates with proliferation arrest and differentiation,”
Scientific reports, vol. 10, no. 1. Springer Nature, 2020.
ista: López De La Oliva AR, Campos-Sandoval JA, Gómez-García MC, Cardona C, Martín-Rufián
M, Sialana FJ, Castilla L, Bae N, Lobo C, Peñalver A, García-Frutos M, Carro D,
Enrique V, Paz JC, Mirmira RG, Gutiérrez A, Alonso FJ, Segura JA, Matés JM, Lubec
G, Márquez J. 2020. Nuclear translocation of glutaminase GLS2 in human cancer
cells associates with proliferation arrest and differentiation. Scientific reports.
10(1), 2259.
mla: López De La Oliva, Amada R., et al. “Nuclear Translocation of Glutaminase GLS2
in Human Cancer Cells Associates with Proliferation Arrest and Differentiation.”
Scientific Reports, vol. 10, no. 1, 2259, Springer Nature, 2020, doi:10.1038/s41598-020-58264-4.
short: A.R. López De La Oliva, J.A. Campos-Sandoval, M.C. Gómez-García, C. Cardona,
M. Martín-Rufián, F.J. Sialana, L. Castilla, N. Bae, C. Lobo, A. Peñalver, M.
García-Frutos, D. Carro, V. Enrique, J.C. Paz, R.G. Mirmira, A. Gutiérrez, F.J.
Alonso, J.A. Segura, J.M. Matés, G. Lubec, J. Márquez, Scientific Reports 10 (2020).
date_created: 2020-02-16T23:00:49Z
date_published: 2020-02-10T00:00:00Z
date_updated: 2026-04-02T11:51:06Z
day: '10'
ddc:
- '570'
department:
- _id: CaBe
doi: 10.1038/s41598-020-58264-4
external_id:
isi:
- '000560694800012'
pmid:
- '32042057'
file:
- access_level: open_access
checksum: c780bd87476a9c9e12668ff66de3dc96
content_type: application/pdf
creator: dernst
date_created: 2020-02-18T07:43:21Z
date_updated: 2020-07-14T12:47:59Z
file_id: '7495'
file_name: 2020_ScientificReport_Lopez.pdf
file_size: 4703751
relation: main_file
file_date_updated: 2020-07-14T12:47:59Z
has_accepted_license: '1'
intvolume: ' 10'
isi: 1
issue: '1'
language:
- iso: eng
month: '02'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
related_material:
link:
- relation: erratum
url: https://doi.org/10.1038/s41598-020-80651-0
scopus_import: '1'
status: public
title: Nuclear translocation of glutaminase GLS2 in human cancer cells associates
with proliferation arrest and differentiation
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 10
year: '2020'
...
---
_id: '6867'
abstract:
- lang: eng
text: A novel magnetic scratch method achieves repeatability, reproducibility and
geometric control greater than pipette scratch assays and closely approximating
the precision of cell exclusion assays while inducing the cell injury inherently
necessary for wound healing assays. The magnetic scratch is affordable, easily
implemented and standardisable and thus may contribute toward better comparability
of data generated in different studies and laboratories.
article_number: '12625'
article_processing_charge: No
author:
- first_name: M.
full_name: Fenu, M.
last_name: Fenu
- first_name: T.
full_name: Bettermann, T.
last_name: Bettermann
- first_name: C.
full_name: Vogl, C.
last_name: Vogl
- first_name: Nasser
full_name: Darwish-Miranda, Nasser
id: 39CD9926-F248-11E8-B48F-1D18A9856A87
last_name: Darwish-Miranda
orcid: 0000-0002-8821-8236
- first_name: J.
full_name: Schramel, J.
last_name: Schramel
- first_name: F.
full_name: Jenner, F.
last_name: Jenner
- first_name: I.
full_name: Ribitsch, I.
last_name: Ribitsch
citation:
ama: Fenu M, Bettermann T, Vogl C, et al. A novel magnet-based scratch method for
standardisation of wound-healing assays. Scientific Reports. 2019;9(1).
doi:10.1038/s41598-019-48930-7
apa: Fenu, M., Bettermann, T., Vogl, C., Darwish-Miranda, N., Schramel, J., Jenner,
F., & Ribitsch, I. (2019). A novel magnet-based scratch method for standardisation
of wound-healing assays. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-019-48930-7
chicago: Fenu, M., T. Bettermann, C. Vogl, Nasser Darwish-Miranda, J. Schramel,
F. Jenner, and I. Ribitsch. “A Novel Magnet-Based Scratch Method for Standardisation
of Wound-Healing Assays.” Scientific Reports. Springer Nature, 2019. https://doi.org/10.1038/s41598-019-48930-7.
ieee: M. Fenu et al., “A novel magnet-based scratch method for standardisation
of wound-healing assays,” Scientific Reports, vol. 9, no. 1. Springer Nature,
2019.
ista: Fenu M, Bettermann T, Vogl C, Darwish-Miranda N, Schramel J, Jenner F, Ribitsch
I. 2019. A novel magnet-based scratch method for standardisation of wound-healing
assays. Scientific Reports. 9(1), 12625.
mla: Fenu, M., et al. “A Novel Magnet-Based Scratch Method for Standardisation of
Wound-Healing Assays.” Scientific Reports, vol. 9, no. 1, 12625, Springer
Nature, 2019, doi:10.1038/s41598-019-48930-7.
short: M. Fenu, T. Bettermann, C. Vogl, N. Darwish-Miranda, J. Schramel, F. Jenner,
I. Ribitsch, Scientific Reports 9 (2019).
date_created: 2019-09-15T22:00:42Z
date_published: 2019-09-02T00:00:00Z
date_updated: 2026-04-03T09:39:11Z
day: '02'
ddc:
- '570'
department:
- _id: Bio
doi: 10.1038/s41598-019-48930-7
external_id:
isi:
- '000483697800007'
pmid:
- '31477739'
file:
- access_level: open_access
checksum: 9cfd986d4108e288cc72276ef047ab0c
content_type: application/pdf
creator: dernst
date_created: 2019-09-16T12:42:40Z
date_updated: 2020-07-14T12:47:42Z
file_id: '6879'
file_name: 2019_ScientificReports_Fenu.pdf
file_size: 3523795
relation: main_file
file_date_updated: 2020-07-14T12:47:42Z
has_accepted_license: '1'
intvolume: ' 9'
isi: 1
issue: '1'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: A novel magnet-based scratch method for standardisation of wound-healing assays
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 9
year: '2019'
...
---
_id: '7095'
abstract:
- lang: eng
text: BAX, a member of the BCL2 gene family, controls the committed step of the
intrinsic apoptotic program. Mitochondrial fragmentation is a commonly observed
feature of apoptosis, which occurs through the process of mitochondrial fission.
BAX has consistently been associated with mitochondrial fission, yet how BAX participates
in the process of mitochondrial fragmentation during apoptosis remains to be tested.
Time-lapse imaging of BAX recruitment and mitochondrial fragmentation demonstrates
that rapid mitochondrial fragmentation during apoptosis occurs after the complete
recruitment of BAX to the mitochondrial outer membrane (MOM). The requirement
of a fully functioning BAX protein for the fission process was demonstrated further
in BAX/BAK-deficient HCT116 cells expressing a P168A mutant of BAX. The mutant
performed fusion to restore the mitochondrial network. but was not demonstrably
recruited to the MOM after apoptosis induction. Under these conditions, mitochondrial
fragmentation was blocked. Additionally, we show that loss of the fission protein,
dynamin-like protein 1 (DRP1), does not temporally affect the initiation time
or rate of BAX recruitment, but does reduce the final level of BAX recruited to
the MOM during the late phase of BAX recruitment. These correlative observations
suggest a model where late-stage BAX oligomers play a functional part of the mitochondrial
fragmentation machinery in apoptotic cells.
article_number: '16565'
article_processing_charge: No
article_type: original
author:
- first_name: Margaret E
full_name: Maes, Margaret E
id: 3838F452-F248-11E8-B48F-1D18A9856A87
last_name: Maes
orcid: 0000-0001-9642-1085
- first_name: J. A.
full_name: Grosser, J. A.
last_name: Grosser
- first_name: R. L.
full_name: Fehrman, R. L.
last_name: Fehrman
- first_name: C. L.
full_name: Schlamp, C. L.
last_name: Schlamp
- first_name: R. W.
full_name: Nickells, R. W.
last_name: Nickells
citation:
ama: Maes ME, Grosser JA, Fehrman RL, Schlamp CL, Nickells RW. Completion of BAX
recruitment correlates with mitochondrial fission during apoptosis. Scientific
Reports. 2019;9. doi:10.1038/s41598-019-53049-w
apa: Maes, M. E., Grosser, J. A., Fehrman, R. L., Schlamp, C. L., & Nickells,
R. W. (2019). Completion of BAX recruitment correlates with mitochondrial fission
during apoptosis. Scientific Reports. Springer Nature. https://doi.org/10.1038/s41598-019-53049-w
chicago: Maes, Margaret E, J. A. Grosser, R. L. Fehrman, C. L. Schlamp, and R. W.
Nickells. “Completion of BAX Recruitment Correlates with Mitochondrial Fission
during Apoptosis.” Scientific Reports. Springer Nature, 2019. https://doi.org/10.1038/s41598-019-53049-w.
ieee: M. E. Maes, J. A. Grosser, R. L. Fehrman, C. L. Schlamp, and R. W. Nickells,
“Completion of BAX recruitment correlates with mitochondrial fission during apoptosis,”
Scientific Reports, vol. 9. Springer Nature, 2019.
ista: Maes ME, Grosser JA, Fehrman RL, Schlamp CL, Nickells RW. 2019. Completion
of BAX recruitment correlates with mitochondrial fission during apoptosis. Scientific
Reports. 9, 16565.
mla: Maes, Margaret E., et al. “Completion of BAX Recruitment Correlates with Mitochondrial
Fission during Apoptosis.” Scientific Reports, vol. 9, 16565, Springer
Nature, 2019, doi:10.1038/s41598-019-53049-w.
short: M.E. Maes, J.A. Grosser, R.L. Fehrman, C.L. Schlamp, R.W. Nickells, Scientific
Reports 9 (2019).
date_created: 2019-11-25T07:45:17Z
date_published: 2019-11-12T00:00:00Z
date_updated: 2023-08-30T07:26:54Z
day: '12'
ddc:
- '570'
department:
- _id: SaSi
doi: 10.1038/s41598-019-53049-w
external_id:
isi:
- '000495857600019'
pmid:
- '31719602'
file:
- access_level: open_access
checksum: 9ab397ed9c1c454b34bffb8cc863d734
content_type: application/pdf
creator: dernst
date_created: 2019-11-25T07:49:52Z
date_updated: 2020-07-14T12:47:49Z
file_id: '7096'
file_name: 2019_ScientificReports_Maes.pdf
file_size: 6467393
relation: main_file
file_date_updated: 2020-07-14T12:47:49Z
has_accepted_license: '1'
intvolume: ' 9'
isi: 1
language:
- iso: eng
month: '11'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Completion of BAX recruitment correlates with mitochondrial fission during
apoptosis
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 9
year: '2019'
...
---
DOAJ_listed: '1'
OA_place: publisher
OA_type: gold
_id: '390'
abstract:
- lang: eng
text: "In the underdoped copper-oxides, high-temperature superconductivity condenses
from a\r\nnonconventional metallic ”pseudogap” phase that exhibits a variety of
non-Fermi liquid properties.\r\nRecently, it has become clear that a charge density
wave (CDW) phase exists within the pseudogap\r\nregime. This CDW coexists and
competes with superconductivity (SC) below the transition temperature\r\nTc, suggesting
that these two orders are intimately related. Here we show that the condensation
of\r\nthe superfluid from this unconventional precursor is reflected in deviations
from the predictions of\r\nBSC theory regarding the recombination rate of quasiparticles.
We report a detailed investigation of\r\nthe quasiparticle (QP) recombination
lifetime, τqp, as a function of temperature and magnetic field in\r\nunderdoped
HgBa2CuO4+δ (Hg-1201) and YBa2Cu3O6+x (YBCO) single crystals by ultrafast time-resolved\r\nreflectivity.
We find that τqp(T) exhibits a local maximum in a small temperature window near
Tc that is\r\nprominent in underdoped samples with coexisting charge order and
vanishes with application of a small\r\nmagnetic field. We explain this unusual,
non-BCS behavior by positing that Tc marks a transition from\r\nphase-fluctuating
SC/CDW composite order above to a SC/CDW condensate below. Our results suggest\r\nthat
the superfluid in underdoped cuprates is a condensate of coherently-mixed particle-particle
and\r\nparticle-hole pairs."
article_number: '23610'
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: James
full_name: Hinton, James
last_name: Hinton
- first_name: E
full_name: Thewalt, E
last_name: Thewalt
- first_name: Zhanybek
full_name: Alpichshev, Zhanybek
id: 45E67A2A-F248-11E8-B48F-1D18A9856A87
last_name: Alpichshev
orcid: 0000-0002-7183-5203
- first_name: Fahad
full_name: Mahmood, Fahad
last_name: Mahmood
- first_name: Jake
full_name: Koralek, Jake
last_name: Koralek
- first_name: Mun
full_name: Chan, Mun
last_name: Chan
- first_name: Michael
full_name: Veit, Michael
last_name: Veit
- first_name: Chelsey
full_name: Dorow, Chelsey
last_name: Dorow
- first_name: Neven
full_name: Barišić, Neven
last_name: Barišić
- first_name: Alexander
full_name: Kemper, Alexander
last_name: Kemper
- first_name: Doug
full_name: Bonn, Doug
last_name: Bonn
- first_name: Walter
full_name: Hardy, Walter
last_name: Hardy
- first_name: Ruixing
full_name: Liang, Ruixing
last_name: Liang
- first_name: Nuh
full_name: Gedik, Nuh
last_name: Gedik
- first_name: Martin
full_name: Greven, Martin
last_name: Greven
- first_name: Alessandra
full_name: Lanzara, Alessandra
last_name: Lanzara
- first_name: Joseph
full_name: Orenstein, Joseph
last_name: Orenstein
citation:
ama: Hinton J, Thewalt E, Alpichshev Z, et al. The rate of quasiparticle recombination
probes the onset of coherence in cuprate superconductors. Scientific Reports.
2016;6. doi:10.1038/srep23610
apa: Hinton, J., Thewalt, E., Alpichshev, Z., Mahmood, F., Koralek, J., Chan, M.,
… Orenstein, J. (2016). The rate of quasiparticle recombination probes the onset
of coherence in cuprate superconductors. Scientific Reports. Nature Publishing
Group. https://doi.org/10.1038/srep23610
chicago: Hinton, James, E Thewalt, Zhanybek Alpichshev, Fahad Mahmood, Jake Koralek,
Mun Chan, Michael Veit, et al. “The Rate of Quasiparticle Recombination Probes
the Onset of Coherence in Cuprate Superconductors.” Scientific Reports.
Nature Publishing Group, 2016. https://doi.org/10.1038/srep23610.
ieee: J. Hinton et al., “The rate of quasiparticle recombination probes the
onset of coherence in cuprate superconductors,” Scientific Reports, vol.
6. Nature Publishing Group, 2016.
ista: Hinton J, Thewalt E, Alpichshev Z, Mahmood F, Koralek J, Chan M, Veit M, Dorow
C, Barišić N, Kemper A, Bonn D, Hardy W, Liang R, Gedik N, Greven M, Lanzara A,
Orenstein J. 2016. The rate of quasiparticle recombination probes the onset of
coherence in cuprate superconductors. Scientific Reports. 6, 23610.
mla: Hinton, James, et al. “The Rate of Quasiparticle Recombination Probes the Onset
of Coherence in Cuprate Superconductors.” Scientific Reports, vol. 6, 23610,
Nature Publishing Group, 2016, doi:10.1038/srep23610.
short: J. Hinton, E. Thewalt, Z. Alpichshev, F. Mahmood, J. Koralek, M. Chan, M.
Veit, C. Dorow, N. Barišić, A. Kemper, D. Bonn, W. Hardy, R. Liang, N. Gedik,
M. Greven, A. Lanzara, J. Orenstein, Scientific Reports 6 (2016).
date_created: 2018-12-11T11:46:12Z
date_published: 2016-04-13T00:00:00Z
date_updated: 2026-05-12T12:41:04Z
day: '13'
doi: 10.1038/srep23610
extern: '1'
external_id:
arxiv:
- '1601.05224'
pmid:
- '27071712'
intvolume: ' 6'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://doi.org/10.1038/srep23610
month: '04'
oa: 1
oa_version: Published Version
pmid: 1
publication: Scientific Reports
publication_identifier:
eissn:
- 2045-2322
publication_status: published
publisher: Nature Publishing Group
publist_id: '7439'
quality_controlled: '1'
scopus_import: '1'
status: public
title: The rate of quasiparticle recombination probes the onset of coherence in cuprate
superconductors
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: ba8df636-2132-11f1-aed0-ed93e2281fdd
volume: 6
year: '2016'
...