---
_id: '7718'
abstract:
- lang: eng
text: Flores Island, Indonesia, was inhabited by the small-bodied hominin species
Homo floresiensis, which has an unknown evolutionary relationship to modern humans.
This island is also home to an extant human pygmy population. Here we describe
genome-scale single-nucleotide polymorphism data and whole-genome sequences from
a contemporary human pygmy population living on Flores near the cave where H.
floresiensis was found. The genomes of Flores pygmies reveal a complex history
of admixture with Denisovans and Neanderthals but no evidence for gene flow with
other archaic hominins. Modern individuals bear the signatures of recent positive
selection encompassing the FADS (fatty acid desaturase) gene cluster, likely related
to diet, and polygenic selection acting on standing variation that contributed
to their short-stature phenotype. Thus, multiple independent instances of hominin
insular dwarfism occurred on Flores.
article_processing_charge: No
article_type: original
author:
- first_name: Serena
full_name: Tucci, Serena
last_name: Tucci
- first_name: Samuel H.
full_name: Vohr, Samuel H.
last_name: Vohr
- first_name: Rajiv C.
full_name: McCoy, Rajiv C.
last_name: McCoy
- first_name: Benjamin
full_name: Vernot, Benjamin
last_name: Vernot
- first_name: Matthew Richard
full_name: Robinson, Matthew Richard
id: E5D42276-F5DA-11E9-8E24-6303E6697425
last_name: Robinson
orcid: 0000-0001-8982-8813
- first_name: Chiara
full_name: Barbieri, Chiara
last_name: Barbieri
- first_name: Brad J.
full_name: Nelson, Brad J.
last_name: Nelson
- first_name: Wenqing
full_name: Fu, Wenqing
last_name: Fu
- first_name: Gludhug A.
full_name: Purnomo, Gludhug A.
last_name: Purnomo
- first_name: Herawati
full_name: Sudoyo, Herawati
last_name: Sudoyo
- first_name: Evan E.
full_name: Eichler, Evan E.
last_name: Eichler
- first_name: Guido
full_name: Barbujani, Guido
last_name: Barbujani
- first_name: Peter M.
full_name: Visscher, Peter M.
last_name: Visscher
- first_name: Joshua M.
full_name: Akey, Joshua M.
last_name: Akey
- first_name: Richard E.
full_name: Green, Richard E.
last_name: Green
citation:
ama: Tucci S, Vohr SH, McCoy RC, et al. Evolutionary history and adaptation of a
human pygmy population of Flores Island, Indonesia. Science. 2018;361(6401):511-516.
doi:10.1126/science.aar8486
apa: Tucci, S., Vohr, S. H., McCoy, R. C., Vernot, B., Robinson, M. R., Barbieri,
C., … Green, R. E. (2018). Evolutionary history and adaptation of a human pygmy
population of Flores Island, Indonesia. Science. American Association for
the Advancement of Science. https://doi.org/10.1126/science.aar8486
chicago: Tucci, Serena, Samuel H. Vohr, Rajiv C. McCoy, Benjamin Vernot, Matthew
Richard Robinson, Chiara Barbieri, Brad J. Nelson, et al. “Evolutionary History
and Adaptation of a Human Pygmy Population of Flores Island, Indonesia.” Science.
American Association for the Advancement of Science, 2018. https://doi.org/10.1126/science.aar8486.
ieee: S. Tucci et al., “Evolutionary history and adaptation of a human pygmy
population of Flores Island, Indonesia,” Science, vol. 361, no. 6401. American
Association for the Advancement of Science, pp. 511–516, 2018.
ista: Tucci S, Vohr SH, McCoy RC, Vernot B, Robinson MR, Barbieri C, Nelson BJ,
Fu W, Purnomo GA, Sudoyo H, Eichler EE, Barbujani G, Visscher PM, Akey JM, Green
RE. 2018. Evolutionary history and adaptation of a human pygmy population of Flores
Island, Indonesia. Science. 361(6401), 511–516.
mla: Tucci, Serena, et al. “Evolutionary History and Adaptation of a Human Pygmy
Population of Flores Island, Indonesia.” Science, vol. 361, no. 6401, American
Association for the Advancement of Science, 2018, pp. 511–16, doi:10.1126/science.aar8486.
short: S. Tucci, S.H. Vohr, R.C. McCoy, B. Vernot, M.R. Robinson, C. Barbieri, B.J.
Nelson, W. Fu, G.A. Purnomo, H. Sudoyo, E.E. Eichler, G. Barbujani, P.M. Visscher,
J.M. Akey, R.E. Green, Science 361 (2018) 511–516.
date_created: 2020-04-30T10:43:24Z
date_published: 2018-08-03T00:00:00Z
date_updated: 2021-01-12T08:15:04Z
day: '03'
doi: 10.1126/science.aar8486
extern: '1'
external_id:
pmid:
- '30072539'
intvolume: ' 361'
issue: '6401'
language:
- iso: eng
month: '08'
oa_version: None
page: 511-516
pmid: 1
publication: Science
publication_identifier:
issn:
- 0036-8075
- 1095-9203
publication_status: published
publisher: American Association for the Advancement of Science
quality_controlled: '1'
status: public
title: Evolutionary history and adaptation of a human pygmy population of Flores Island,
Indonesia
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 361
year: '2018'
...
---
_id: '7'
abstract:
- lang: eng
text: Animal social networks are shaped by multiple selection pressures, including
the need to ensure efficient communication and functioning while simultaneously
limiting disease transmission. Social animals could potentially further reduce
epidemic risk by altering their social networks in the presence of pathogens,
yet there is currently no evidence for such pathogen-triggered responses. We tested
this hypothesis experimentally in the ant Lasius niger using a combination of
automated tracking, controlled pathogen exposure, transmission quantification,
and temporally explicit simulations. Pathogen exposure induced behavioral changes
in both exposed ants and their nestmates, which helped contain the disease by
reinforcing key transmission-inhibitory properties of the colony's contact network.
This suggests that social network plasticity in response to pathogens is an effective
strategy for mitigating the effects of disease in social groups.
acknowledgement: This project was funded by two European Research Council Advanced
Grants (Social Life, 249375, and resiliANT, 741491) and two Swiss National Science
Foundation grants (CR32I3_141063 and 310030_156732) to L.K. and a European Research
Council Starting Grant (SocialVaccines, 243071) to S.C.
article_processing_charge: No
article_type: original
author:
- first_name: Nathalie
full_name: Stroeymeyt, Nathalie
last_name: Stroeymeyt
- first_name: Anna V
full_name: Grasse, Anna V
id: 406F989C-F248-11E8-B48F-1D18A9856A87
last_name: Grasse
- first_name: Alessandro
full_name: Crespi, Alessandro
last_name: Crespi
- first_name: Danielle
full_name: Mersch, Danielle
last_name: Mersch
- first_name: Sylvia
full_name: Cremer, Sylvia
id: 2F64EC8C-F248-11E8-B48F-1D18A9856A87
last_name: Cremer
orcid: 0000-0002-2193-3868
- first_name: Laurent
full_name: Keller, Laurent
last_name: Keller
citation:
ama: Stroeymeyt N, Grasse AV, Crespi A, Mersch D, Cremer S, Keller L. Social network
plasticity decreases disease transmission in a eusocial insect. Science.
2018;362(6417):941-945. doi:10.1126/science.aat4793
apa: Stroeymeyt, N., Grasse, A. V., Crespi, A., Mersch, D., Cremer, S., & Keller,
L. (2018). Social network plasticity decreases disease transmission in a eusocial
insect. Science. AAAS. https://doi.org/10.1126/science.aat4793
chicago: Stroeymeyt, Nathalie, Anna V Grasse, Alessandro Crespi, Danielle Mersch,
Sylvia Cremer, and Laurent Keller. “Social Network Plasticity Decreases Disease
Transmission in a Eusocial Insect.” Science. AAAS, 2018. https://doi.org/10.1126/science.aat4793.
ieee: N. Stroeymeyt, A. V. Grasse, A. Crespi, D. Mersch, S. Cremer, and L. Keller,
“Social network plasticity decreases disease transmission in a eusocial insect,”
Science, vol. 362, no. 6417. AAAS, pp. 941–945, 2018.
ista: Stroeymeyt N, Grasse AV, Crespi A, Mersch D, Cremer S, Keller L. 2018. Social
network plasticity decreases disease transmission in a eusocial insect. Science.
362(6417), 941–945.
mla: Stroeymeyt, Nathalie, et al. “Social Network Plasticity Decreases Disease Transmission
in a Eusocial Insect.” Science, vol. 362, no. 6417, AAAS, 2018, pp. 941–45,
doi:10.1126/science.aat4793.
short: N. Stroeymeyt, A.V. Grasse, A. Crespi, D. Mersch, S. Cremer, L. Keller, Science
362 (2018) 941–945.
date_created: 2018-12-11T11:44:07Z
date_published: 2018-11-23T00:00:00Z
date_updated: 2026-06-18T19:15:22Z
day: '23'
ddc:
- '570'
department:
- _id: SyCr
doi: 10.1126/science.aat4793
ec_funded: 1
external_id:
isi:
- '000451124500041'
intvolume: ' 362'
isi: 1
issue: '6417'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://serval.unil.ch/resource/serval:BIB_E9228C205467.P001/REF.pdf
month: '11'
oa: 1
oa_version: Published Version
page: 941 - 945
project:
- _id: 25DC711C-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '243071'
name: 'Social Vaccination in Ant Colonies: from Individual Mechanisms to Society
Effects'
publication: Science
publication_identifier:
issn:
- 1095-9203
publication_status: published
publisher: AAAS
publist_id: '8049'
quality_controlled: '1'
related_material:
link:
- description: News on IST Homepage
relation: press_release
url: https://ist.ac.at/en/news/for-ants-unity-is-strength-and-health/
record:
- id: '13055'
relation: research_data
status: public
scopus_import: '1'
status: public
title: Social network plasticity decreases disease transmission in a eusocial insect
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 362
year: '2018'
...
---
_id: '7310'
abstract:
- lang: eng
text: The rechargeable nonaqueous lithium-air (Li-O2) battery is receiving a great
deal of interest because, theoretically, its specific energy far exceeds the best
that can be achieved with lithium-ion cells. Operation of the rechargeable Li-O2
battery depends critically on repeated and highly reversible formation/decomposition
of lithium peroxide (Li2O2) at the cathode upon cycling. Here, we show that this
process is possible with the use of a dimethyl sulfoxide electrolyte and a porous
gold electrode (95% capacity retention from cycles 1 to 100), whereas previously
only partial Li2O2 formation/decomposition and limited cycling could occur. Furthermore,
we present data indicating that the kinetics of Li2O2 oxidation on charge is approximately
10 times faster than on carbon electrodes.
article_processing_charge: No
article_type: original
author:
- first_name: Z.
full_name: Peng, Z.
last_name: Peng
- first_name: Stefan Alexander
full_name: Freunberger, Stefan Alexander
id: A8CA28E6-CE23-11E9-AD2D-EC27E6697425
last_name: Freunberger
orcid: 0000-0003-2902-5319
- first_name: Y.
full_name: Chen, Y.
last_name: Chen
- first_name: P. G.
full_name: Bruce, P. G.
last_name: Bruce
citation:
ama: Peng Z, Freunberger SA, Chen Y, Bruce PG. A reversible and higher-rate Li-O2
battery. Science. 2012;337(6094):563-566. doi:10.1126/science.1223985
apa: Peng, Z., Freunberger, S. A., Chen, Y., & Bruce, P. G. (2012). A reversible
and higher-rate Li-O2 battery. Science. AAAS. https://doi.org/10.1126/science.1223985
chicago: Peng, Z., Stefan Alexander Freunberger, Y. Chen, and P. G. Bruce. “A Reversible
and Higher-Rate Li-O2 Battery.” Science. AAAS, 2012. https://doi.org/10.1126/science.1223985.
ieee: Z. Peng, S. A. Freunberger, Y. Chen, and P. G. Bruce, “A reversible and higher-rate
Li-O2 battery,” Science, vol. 337, no. 6094. AAAS, pp. 563–566, 2012.
ista: Peng Z, Freunberger SA, Chen Y, Bruce PG. 2012. A reversible and higher-rate
Li-O2 battery. Science. 337(6094), 563–566.
mla: Peng, Z., et al. “A Reversible and Higher-Rate Li-O2 Battery.” Science,
vol. 337, no. 6094, AAAS, 2012, pp. 563–66, doi:10.1126/science.1223985.
short: Z. Peng, S.A. Freunberger, Y. Chen, P.G. Bruce, Science 337 (2012) 563–566.
date_created: 2020-01-15T12:19:23Z
date_published: 2012-08-03T00:00:00Z
date_updated: 2021-01-12T08:12:57Z
day: '03'
doi: 10.1126/science.1223985
extern: '1'
intvolume: ' 337'
issue: '6094'
language:
- iso: eng
month: '08'
oa_version: None
page: 563-566
publication: Science
publication_identifier:
issn:
- 0036-8075
- 1095-9203
publication_status: published
publisher: AAAS
quality_controlled: '1'
status: public
title: A reversible and higher-rate Li-O2 battery
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 337
year: '2012'
...
---
_id: '8074'
abstract:
- lang: eng
text: Cortical neurons receive balanced excitatory and inhibitory synaptic currents.
Such a balance could be established and maintained in an experience-dependent
manner by synaptic plasticity at inhibitory synapses. We show that this mechanism
provides an explanation for the sparse firing patterns observed in response to
natural stimuli and fits well with a recently observed interaction of excitatory
and inhibitory receptive field plasticity. The introduction of inhibitory plasticity
in suitable recurrent networks provides a homeostatic mechanism that leads to
asynchronous irregular network states. Further, it can accommodate synaptic memories
with activity patterns that become indiscernible from the background state but
can be reactivated by external stimuli. Our results suggest an essential role
of inhibitory plasticity in the formation and maintenance of functional cortical
circuitry.
article_processing_charge: No
article_type: original
author:
- first_name: Tim P
full_name: Vogels, Tim P
id: CB6FF8D2-008F-11EA-8E08-2637E6697425
last_name: Vogels
orcid: 0000-0003-3295-6181
- first_name: H.
full_name: Sprekeler, H.
last_name: Sprekeler
- first_name: F.
full_name: Zenke, F.
last_name: Zenke
- first_name: C.
full_name: Clopath, C.
last_name: Clopath
- first_name: W.
full_name: Gerstner, W.
last_name: Gerstner
citation:
ama: Vogels TP, Sprekeler H, Zenke F, Clopath C, Gerstner W. Inhibitory plasticity
balances excitation and inhibition in sensory pathways and memory networks. Science.
2011;334(6062):1569-1573. doi:10.1126/science.1211095
apa: Vogels, T. P., Sprekeler, H., Zenke, F., Clopath, C., & Gerstner, W. (2011).
Inhibitory plasticity balances excitation and inhibition in sensory pathways and
memory networks. Science. American Association for the Advancement of Science.
https://doi.org/10.1126/science.1211095
chicago: Vogels, Tim P, H. Sprekeler, F. Zenke, C. Clopath, and W. Gerstner. “Inhibitory
Plasticity Balances Excitation and Inhibition in Sensory Pathways and Memory Networks.”
Science. American Association for the Advancement of Science, 2011. https://doi.org/10.1126/science.1211095.
ieee: T. P. Vogels, H. Sprekeler, F. Zenke, C. Clopath, and W. Gerstner, “Inhibitory
plasticity balances excitation and inhibition in sensory pathways and memory networks,”
Science, vol. 334, no. 6062. American Association for the Advancement of
Science, pp. 1569–1573, 2011.
ista: Vogels TP, Sprekeler H, Zenke F, Clopath C, Gerstner W. 2011. Inhibitory plasticity
balances excitation and inhibition in sensory pathways and memory networks. Science.
334(6062), 1569–1573.
mla: Vogels, Tim P., et al. “Inhibitory Plasticity Balances Excitation and Inhibition
in Sensory Pathways and Memory Networks.” Science, vol. 334, no. 6062,
American Association for the Advancement of Science, 2011, pp. 1569–73, doi:10.1126/science.1211095.
short: T.P. Vogels, H. Sprekeler, F. Zenke, C. Clopath, W. Gerstner, Science 334
(2011) 1569–1573.
date_created: 2020-06-30T13:26:17Z
date_published: 2011-12-16T00:00:00Z
date_updated: 2021-06-02T14:57:22Z
day: '16'
doi: 10.1126/science.1211095
extern: '1'
external_id:
pmid:
- '22075724'
intvolume: ' 334'
issue: '6062'
language:
- iso: eng
month: '12'
oa_version: None
page: 1569-1573
pmid: 1
publication: Science
publication_identifier:
issn:
- 0036-8075
- 1095-9203
publication_status: published
publisher: American Association for the Advancement of Science
quality_controlled: '1'
related_material:
link:
- relation: erratum
url: https://doi.org/10.1126/science.336.6083.802-c
scopus_import: '1'
status: public
title: Inhibitory plasticity balances excitation and inhibition in sensory pathways
and memory networks
type: journal_article
user_id: D865714E-FA4E-11E9-B85B-F5C5E5697425
volume: 334
year: '2011'
...
---
_id: '11118'
abstract:
- lang: eng
text: Nuclear pore complexes are multiprotein channels that span the double lipid
bilayer of the nuclear envelope. How new pores are inserted into the intact nuclear
envelope of proliferating and differentiating eukaryotic cells is unknown. We
found that the Nup107-160 complex was incorporated into assembly sites in the
nuclear envelope from both the nucleoplasmic and the cytoplasmic sides. Nuclear
pore insertion required the generation of Ran guanosine triphosphate in the nuclear
and cytoplasmic compartments. Newly formed nuclear pore complexes did not contain
structural components of preexisting pores, suggesting that they can form de novo.
article_processing_charge: No
article_type: original
author:
- first_name: Maximiliano A.
full_name: D'Angelo, Maximiliano A.
last_name: D'Angelo
- first_name: Daniel J.
full_name: Anderson, Daniel J.
last_name: Anderson
- first_name: Erin
full_name: Richard, Erin
last_name: Richard
- first_name: Martin W
full_name: HETZER, Martin W
id: 86c0d31b-b4eb-11ec-ac5a-eae7b2e135ed
last_name: HETZER
orcid: 0000-0002-2111-992X
citation:
ama: D’Angelo MA, Anderson DJ, Richard E, Hetzer M. Nuclear pores form de novo from
both sides of the nuclear envelope. Science. 2006;312(5772):440-443. doi:10.1126/science.1124196
apa: D’Angelo, M. A., Anderson, D. J., Richard, E., & Hetzer, M. (2006). Nuclear
pores form de novo from both sides of the nuclear envelope. Science. American
Association for the Advancement of Science. https://doi.org/10.1126/science.1124196
chicago: D’Angelo, Maximiliano A., Daniel J. Anderson, Erin Richard, and Martin
Hetzer. “Nuclear Pores Form de Novo from Both Sides of the Nuclear Envelope.”
Science. American Association for the Advancement of Science, 2006. https://doi.org/10.1126/science.1124196.
ieee: M. A. D’Angelo, D. J. Anderson, E. Richard, and M. Hetzer, “Nuclear pores
form de novo from both sides of the nuclear envelope,” Science, vol. 312,
no. 5772. American Association for the Advancement of Science, pp. 440–443, 2006.
ista: D’Angelo MA, Anderson DJ, Richard E, Hetzer M. 2006. Nuclear pores form de
novo from both sides of the nuclear envelope. Science. 312(5772), 440–443.
mla: D’Angelo, Maximiliano A., et al. “Nuclear Pores Form de Novo from Both Sides
of the Nuclear Envelope.” Science, vol. 312, no. 5772, American Association
for the Advancement of Science, 2006, pp. 440–43, doi:10.1126/science.1124196.
short: M.A. D’Angelo, D.J. Anderson, E. Richard, M. Hetzer, Science 312 (2006) 440–443.
date_created: 2022-04-07T07:56:32Z
date_published: 2006-04-21T00:00:00Z
date_updated: 2024-10-14T11:30:50Z
day: '21'
doi: 10.1126/science.1124196
extern: '1'
external_id:
pmid:
- '16627745'
intvolume: ' 312'
issue: '5772'
keyword:
- Multidisciplinary
language:
- iso: eng
month: '04'
oa_version: None
page: 440-443
pmid: 1
publication: Science
publication_identifier:
issn:
- 0036-8075
- 1095-9203
publication_status: published
publisher: American Association for the Advancement of Science
quality_controlled: '1'
scopus_import: '1'
status: public
title: Nuclear pores form de novo from both sides of the nuclear envelope
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 312
year: '2006'
...
---
_id: '7706'
abstract:
- lang: eng
text: 'The Sir2 deacetylase modulates organismal life-span in various species. However,
the molecular mechanisms by which Sir2 increases longevity are largely unknown.
We show that in mammalian cells, the Sir2 homolog SIRT1 appears to control the
cellular response to stress by regulating the FOXO family of Forkhead transcription
factors, a family of proteins that function as sensors of the insulin signaling
pathway and as regulators of organismal longevity. SIRT1 and the FOXO transcription
factor FOXO3 formed a complex in cells in response to oxidative stress, and SIRT1
deacetylated FOXO3 in vitro and within cells. SIRT1 had a dual effect on FOXO3
function: SIRT1 increased FOXO3''s ability to induce cell cycle arrest and resistance
to oxidative stress but inhibited FOXO3''s ability to induce cell death. Thus,
one way in which members of the Sir2 family of proteins may increase organismal
longevity is by tipping FOXO-dependent responses away from apoptosis and toward
stress resistance.'
article_processing_charge: No
article_type: original
author:
- first_name: Anne
full_name: Brunet, Anne
last_name: Brunet
- first_name: Lora Beatrice Jaeger
full_name: Sweeney, Lora Beatrice Jaeger
id: 56BE8254-C4F0-11E9-8E45-0B23E6697425
last_name: Sweeney
orcid: 0000-0001-9242-5601
- first_name: 'J Fitzhugh '
full_name: 'Sturgill, J Fitzhugh '
last_name: Sturgill
- first_name: Katrin
full_name: Chua, Katrin
last_name: Chua
- first_name: Paul
full_name: Greer, Paul
last_name: Greer
- first_name: Yingxi
full_name: Lin, Yingxi
last_name: Lin
- first_name: Hien
full_name: Tran, Hien
last_name: Tran
- first_name: Sarah
full_name: Ross, Sarah
last_name: Ross
- first_name: Raul
full_name: Mostoslavsky, Raul
last_name: Mostoslavsky
- first_name: Haim
full_name: Cohen, Haim
last_name: Cohen
- first_name: Linda
full_name: Hu, Linda
last_name: Hu
- first_name: Hwei-Ling
full_name: Chen, Hwei-Ling
last_name: Chen
- first_name: Mark
full_name: Jedrychowski, Mark
last_name: Jedrychowski
- first_name: Steven
full_name: Gygi, Steven
last_name: Gygi
- first_name: David
full_name: Sinclair, David
last_name: Sinclair
- first_name: Frederick
full_name: Alt, Frederick
last_name: Alt
- first_name: Michael
full_name: Greenberg, Michael
last_name: Greenberg
citation:
ama: Brunet A, Sweeney LB, Sturgill JF, et al. Stress-dependent regulation of FOXO
transcription factors by the SIRT1 deacetylase. Science. 2004;303(5666):2011-2015.
doi:10.1126/science.1094637
apa: Brunet, A., Sweeney, L. B., Sturgill, J. F., Chua, K., Greer, P., Lin, Y.,
… Greenberg, M. (2004). Stress-dependent regulation of FOXO transcription factors
by the SIRT1 deacetylase. Science. American Association for the Advancement
of Science. https://doi.org/10.1126/science.1094637
chicago: Brunet, Anne, Lora B. Sweeney, J Fitzhugh Sturgill, Katrin Chua, Paul
Greer, Yingxi Lin, Hien Tran, et al. “Stress-Dependent Regulation of FOXO Transcription
Factors by the SIRT1 Deacetylase.” Science. American Association for the
Advancement of Science, 2004. https://doi.org/10.1126/science.1094637.
ieee: A. Brunet et al., “Stress-dependent regulation of FOXO transcription
factors by the SIRT1 deacetylase,” Science, vol. 303, no. 5666. American
Association for the Advancement of Science, pp. 2011–2015, 2004.
ista: Brunet A, Sweeney LB, Sturgill JF, Chua K, Greer P, Lin Y, Tran H, Ross S,
Mostoslavsky R, Cohen H, Hu L, Chen H-L, Jedrychowski M, Gygi S, Sinclair D, Alt
F, Greenberg M. 2004. Stress-dependent regulation of FOXO transcription factors
by the SIRT1 deacetylase. Science. 303(5666), 2011–2015.
mla: Brunet, Anne, et al. “Stress-Dependent Regulation of FOXO Transcription Factors
by the SIRT1 Deacetylase.” Science, vol. 303, no. 5666, American Association
for the Advancement of Science, 2004, pp. 2011–15, doi:10.1126/science.1094637.
short: A. Brunet, L.B. Sweeney, J.F. Sturgill, K. Chua, P. Greer, Y. Lin, H. Tran,
S. Ross, R. Mostoslavsky, H. Cohen, L. Hu, H.-L. Chen, M. Jedrychowski, S. Gygi,
D. Sinclair, F. Alt, M. Greenberg, Science 303 (2004) 2011–2015.
date_created: 2020-04-30T10:37:41Z
date_published: 2004-03-26T00:00:00Z
date_updated: 2024-01-31T10:14:17Z
day: '26'
doi: 10.1126/science.1094637
extern: '1'
intvolume: ' 303'
issue: '5666'
language:
- iso: eng
month: '03'
oa_version: None
page: 2011-2015
publication: Science
publication_identifier:
issn:
- 0036-8075
- 1095-9203
publication_status: published
publisher: American Association for the Advancement of Science
quality_controlled: '1'
status: public
title: Stress-dependent regulation of FOXO transcription factors by the SIRT1 deacetylase
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 303
year: '2004'
...