DOI,IST REx ID,Research Group,Title of publication
10.1167/iovs.66.3.49,19566,SaSi,Optic nerve crush does not induce retinal ganglion cell loss in the contralateral eye
10.1016/j.isci.2023.107780,14363,SaSi,Mitochondrial network adaptations of microglia reveal sex-specific stress response after injury and UCP2 knockout
10.1186/s13024-023-00659-8,14401,SaSi,BAX activation in mouse retinal ganglion cells occurs in two temporally and mechanistically distinct steps
10.1016/j.omtm.2021.09.006,10655,"SaSi,SiHi",Optimizing AAV2/6 microglial targeting identified enhanced efficiency in the photoreceptor degenerative environment
10.1167/IOVS.62.10.14,10000,SaSi,Increased susceptibility and intrinsic apoptotic signaling in neurons by induced HDAC3 expression
10.1007/s10495-020-01654-w,9009,SaSi,Characteristics of intracellular propagation of mitochondrial BAX recruitment during apoptosis
10.3390/cells10071593,9761,SaSi,The influence of mitochondrial dynamics and function on retinal ganglion cell susceptibility in optic nerve disease
10.1016/j.celrep.2021.109313,9642,SaSi,Microglia enable mature perineuronal nets disassembly upon anesthetic ketamine exposure or 60-Hz light entrainment in the healthy brain
10.1007/s12035-019-01783-7,7033,SaSi,BAX-depleted retinal ganglion cells survive and become quiescent following optic nerve damage
10.1016/j.neulet.2019.134310,6521,SaSi,Targeting microglia with lentivirus and AAV: Recent advances and remaining challenges
10.1038/s41598-019-53049-w,7095,SaSi,Completion of BAX recruitment correlates with mitochondrial fission during apoptosis
10.1167/iovs.17-22634,557,SaSi,AAV2 mediated transduction of the mouse retina after optic nerve injury