@article{2622,
  abstract     = {To understand the possible contribution of metabotropic γ-aminobutyric acid receptors (GABABR) in cortical development, we investigated the expression pattern and the cellular and subcellular localization of the GABABR1 and GABABR2 subtypes in the rat neocortex from embryonic day 14 (E14) to adulthood. At the light microscopic level, both GABABR1 and GABABR2 were detected as early as E14. During prenatal development, both subtypes were expressed highly in the cortical plate. Using double immunofluorescence, GABABR1 colocalized with GABABR2 in neurons of the marginal zone and subplate, indicating that these proteins are coexpressed and could be forming functional GABABRs during prenatal development in vivo. In contrast, only GABABR1 but not GABABR2 was detected in the tangentially migratory cells in the lower intermediate zone. During postnatal development, immunoreactivity for GABABR1 and GABABR2 was distributed mainly in pyramidal cells. Discrete GABABR1-immunopositive cell bodies of interneurons were present throughout the neocortex. In addition, GABABR1 but not GABABR2 was found in identified Cajal-Retzius cells in layer I. At the electron microscopic level, immunoreactivity for GABABR1 and GABABR2 was found in dendritic spines and dendritic shafts at extrasynaptic and perisynaptic sites throughout postnatal development. We further demonstrated the presynaptic localization of GABABR1 and GABABR2, as well as the association of the receptors with asymmetrical synaptic junctions. These results indicate potentially important roles for the GABABRs in the regulation of migratory processes during corticogenesis and in the modulation of synaptic transmission during early development of cortical circuitry.},
  author       = {López Bendito, Guillermina and Shigemoto, Ryuichi and Kulik, Ákos and Paulsen, Ole and Fairén, Alfonso and Luján, Rafael},
  issn         = {0953-816X},
  journal      = {European Journal of Neuroscience},
  number       = {11},
  pages        = {1766 -- 1778},
  publisher    = {Wiley-Blackwell},
  title        = {{Expression and distribution of metabotropic GABA receptor subtypes GABABR1 and GABABR2 during rat neocortical development}},
  doi          = {10.1046/j.1460-9568.2002.02032.x},
  volume       = {15},
  year         = {2002},
}

@article{2624,
  abstract     = {Metabotropic γ-aminobutyric acid receptors (GABABRs) are involved in modulation of synaptic transmission and activity of cerebellar and thalamic neurons. We used subtype-specific antibodies in pre- and postembedding immunohistochemistry combined with three-dimensional reconstruction of labelled profiles and quantification of immunoparticles to reveal the subcellular distribution of pre- and postsynaptic GABABR1a/b and GABABR2 in the rat cerebellum and ventrobasal thalamus. GABABR1a/b and R2 were extensively colocalized in most brain regions including the cerebellum and thalamus. In the cerebellum, immunoreactivity for both subtypes was prevalent in the molecular layer. The most intense immunoreactivity was found in Purkinje cell spines with a high density of immunoparticles at extrasynaptic sites peaking at around 240 nm from glutamatergic synapses between spines and parallel fibre varicosities. This is in contrast to dendrites at sites around GABAergic synapses where sparse and random distribution was found for both subtypes. In addition, more than one-tenth of the synaptic membrane specialization of spine-parallel fibre synapses were labelled at pre- or postsynaptic sites. Weak immunolabelling for both subtypes was also seen in parallel fibres but only rarely in GABAergic axons. In the ventrobasal thalamus, immunolabelling for both receptor subtypes was intense over the dendritic field of thalamocortical cells. Electron microscopy demonstrated an extrasynaptic localization of GABABR1a/b and R2 exclusively in postsynaptic elements. Quantitative analysis further revealed the density of GABABR1a/b around GABAergic synapses was higher than glutamatergic synapses on thalamocortical cell dendrites. The distinct localization of GABABRs relative to synaptic sites in the cerebellum and ventrobasal thalamus suggests that GABABRs differentially regulate activity of different neuronal populations.},
  author       = {Kulik, Ákos and Nakadate, Kazuhiko and Nyíri, Gábor and Notomi, Takuya and Malitschek, Barbara and Bettler, Bernhard and Shigemoto, Ryuichi},
  issn         = {1460-9568},
  journal      = {European Journal of Neuroscience},
  number       = {2},
  pages        = {291 -- 307},
  publisher    = {Wiley},
  title        = {{Distinct localization of GABAB receptors relative to synaptic sites in the rat cerebellum and ventrobasal thalamus}},
  doi          = {10.1046/j.0953-816x.2001.01855.x},
  volume       = {15},
  year         = {2002},
}

@inbook{2694,
  abstract     = {We outline the status of rigorous derivations of certain classical evolution equations as limits of Schrödinger dynamics. We explain two recent results jointly with H.T. Yau in more details. The first one is the derivation of the linear Boltzmann equation as the long time limit of the one-body Schrödinger equation with a random potential. The second one is the mean field limit of high density bosons with Coulomb interaction that leads to the nonlinear Hartree equation.},
  author       = {Erdös, László},
  booktitle    = {Dynamics of Dissipation},
  isbn         = {9783540441113},
  pages        = {487 -- 506},
  publisher    = {Springer},
  title        = {{Scaling limits of Schrödinger quantum mechanics}},
  doi          = {10.1007/3-540-46122-1_19},
  year         = {2002},
}

@inproceedings{2708,
  author       = {László Erdös},
  pages        = {129 -- 133},
  publisher    = {World Scientific Publishing},
  title        = {{Two dimensional Pauli operator via scalar potential}},
  doi          = {10.1090/conm/307},
  volume       = {307},
  year         = {2002},
}

@article{2737,
  abstract     = {We derive the time-dependent Schrödinger–Poisson equation as the weak coupling limit of the N-body linear Schrödinger equation with Coulomb potential.},
  author       = {Bardos, Claude and Erdös, László and Golse, François and Mauser, Norbert and Yau, Horng},
  issn         = {1631-073X},
  journal      = {Comptes Rendus Mathematique},
  number       = {6},
  pages        = {515 -- 520},
  publisher    = {Elsevier},
  title        = {{Derivation of the Schrödinger-Poisson equation from the quantum N-body problem}},
  doi          = {10.1016/S1631-073X(02)02253-7},
  volume       = {334},
  year         = {2002},
}

@article{2738,
  abstract     = {We consider the long time evolution of a quantum particle weakly interacting with a phonon field. We show that in the weak coupling limit the Wigner distribution of the electron density matrix converges to the solution of the linear Boltzmann equation globally in time. The collision kernel is identified as the sum of an emission and an absorption term that depend on the equilibrium distribution of the free phonon modes.},
  author       = {Erdös, László},
  issn         = {0022-4715},
  journal      = {Journal of Statistical Physics},
  number       = {5-6},
  pages        = {1043 -- 1127},
  publisher    = {Springer},
  title        = {{Linear Boltzmann equation as the long time dynamics of an electron weakly coupled to a phonon field}},
  doi          = {10.1023/A:1015157624384},
  volume       = {107},
  year         = {2002},
}

@article{2739,
  abstract     = {We define the two dimensional Pauli operator and identify its core for magnetic fields that are regular Borel measures. The magnetic field is generated by a scalar potential hence we bypass the usual A L 2loc condition on the vector potential, which does not allow to consider such singular fields. We extend the Aharonov-Casher theorem for magnetic fields that are measures with finite total variation and we present a counterexample in case of infinite total variation. One of the key technical tools is a weighted L 2 estimate on a singular integral operator.},
  author       = {Erdös, László and Vougalter, Vitali},
  issn         = {0010-3616},
  journal      = {Communications in Mathematical Physics},
  number       = {2},
  pages        = {399 -- 421},
  publisher    = {Springer},
  title        = {{Pauli operator and Aharonov–Casher theorem¶ for measure valued magnetic fields}},
  doi          = {10.1007/s002200100585},
  volume       = {225},
  year         = {2002},
}

@article{2740,
  abstract     = {We show that the lowest eigenvalue of the magnetic Schrödinger operator on a line bundle over a compact Riemann surface M is bounded by the L1-norm of the magnetic field B. This implies a similar bound on the multiplicity of the ground state. An example shows that this degeneracy can indeed be comparable with ∫M |B| even in case of the trivial bundle.},
  author       = {Erdös, László},
  issn         = {0373-0956},
  journal      = {Annales de l'Institut Fourier},
  number       = {6},
  pages        = {1833--1874},
  publisher    = {Association des Annales de l'Institut Fourier},
  title        = {{Spectral shift and multiplicity of the first eigenvalue of the magnetic Schrödinger operator in two dimensions}},
  doi          = {10.5802/aif.1936},
  volume       = {52},
  year         = {2002},
}

@article{2866,
  abstract     = {Developmental responses to the plant hormone auxin are thought to be mediated by interacting pairs from two protein families: short-lived inhibitory IAA proteins and ARF transcription factors binding to auxin-response elements. Monopteros mutants lacking activating ARF5 and the auxin-insensitive mutant bodenlos fail to initiate the root meristem during early embryogenesis. Here we show that the bodenlos phenotype results from an amino-acid exchange in the conserved degradation domain of IAA12. BODENLOS and MONOPTEROS interact in the yeast two-hybrid assay and the two genes are coexpressed in early embryogenesis, suggesting that BODENLOS inhibits MONOPTEROS action in root meristem initiation.},
  author       = {Hamann, Thorsten and Benková, Eva and Bäurle, Isabel and Kientz, Marika and Jürgens, Gerd},
  issn         = {0890-9369},
  journal      = {Genes and Development},
  number       = {13},
  pages        = {1610 -- 1615},
  publisher    = {Cold Spring Harbor Laboratory Press},
  title        = {{The Arabidopsis BODENLOS gene encodes an auxin response protein inhibiting MONOPTEROS-mediated embryo patterning}},
  doi          = {10.1101/gad.229402},
  volume       = {16},
  year         = {2002},
}

@inproceedings{2927,
  abstract     = {In the last few years, several new algorithms based on graph cuts have been developed to solve energy minimization problems in computer vision. Each of these techniques constructs a graph such that the minimum cut on the graph also minimizes the energy. Yet because these graph constructions are complex and highly specific to a particular energy function, graph cuts have seen limited application to date. In this paper we characterize the energy functions that can be minimized by graph cuts. Our results are restricted to energy functions with binary variables. However, our work generalizes many previous constructions, and is easily applicable to vision problems that involve large numbers of labels, such as stereo, motion, image restoration and scene reconstruction. We present three main results: a necessary condition for any energy function that can be minimized by graph cuts; a sufficient condition for energy functions that can be written as a sum of functions of up to three variables at a time; and a general-purpose construction to minimize such an energy function. Researchers who are considering the use of graph cuts to optimize a particular energy function can use our results to determine if this is possible, and then follow our construction to create the appropriate graph.},
  author       = {Kolmogorov, Vladimir and Zabih, Ramin},
  booktitle    = {Proceedings of the 7th European Conference on Computer Vision},
  isbn         = {9783540437468},
  location     = {Copenhagen, Denmark},
  pages        = {65 -- 81},
  publisher    = {Springer},
  title        = {{Multi-camera scene reconstruction via graph cuts}},
  doi          = {10.1007/3-540-47977-5_5},
  year         = {2002},
}

@article{2986,
  abstract     = {Long-standing models propose that plant growth responses to light or gravity are mediated by asymmetric distribution of the phytohormone auxin. Physiological studies implicated a specific transport system that relocates auxin laterally, thereby effecting differential growth; however, neither the molecular components of this system nor the cellular mechanism of auxin redistribution on light or gravity perception have been identified. Here, we show that auxin accumulates asymmetrically during differential growth in an efflux-dependent manner. Mutations in the Arabidopsis gene PIN3, a regulator of auxin efflux, alter differential growth. PIN3 is expressed in gravity-sensing tissues, with PIN3 protein accumulating predominantly at the lateral cell surface. PIN3 localizes to the plasma membrane and to vesicles that cycle in an actin-dependent manner. In the root columella, PIN3 is positioned symmetrically at the plasma membrane but rapidly relocalizes laterally on gravity stimulation. Our data indicate that PIN3 is a component of the lateral auxin transport system regulating tropic growth. In addition, actin-dependent relocalization of PIN3 in response to gravity provides a mechanism for redirecting auxin flux to trigger asymmetric growth.},
  author       = {Friml, Jirí and Wiśniewska, Justyna and Benková, Eva and Mendgen, Kurt and Palme, Klaus},
  issn         = {0028-0836},
  journal      = {Nature},
  number       = {6873},
  pages        = {806 -- 809},
  publisher    = {Nature Publishing Group},
  title        = {{Lateral relocation of auxin efflux regulator PIN3 mediates tropism in Arabidopsis}},
  doi          = {10.1038/415806a},
  volume       = {415},
  year         = {2002},
}

@article{2987,
  abstract     = {The hydra mutants of Arabidopsis are characterized by a pleiotropic phenotype that shows defective embryonic and seedling cell patterning, morphogenesis, and root growth. We demonstrate that the HYDRA1 gene encodes a Δ8-Δ7 sterol isomerase, whereas HYDRA2 encodes a sterol C14 reductase, previously identified as the FACKEL gene product. Seedlings mutant for each gene are similarly defective in the concentrations of the three major Arabidopsis sterols. Promoter::reporter gene analysis showed misexpression of the auxin-regulated DR5 and ACS1 promoters and of the epidermal cell file-specific GL2 promoter in the mutants. The mutants exhibit enhanced responses to auxin. The phenotypes can be rescued partially by inhibition of auxin and ethylene signaling but not by exogenous sterols or brassinosteroids. We propose a model in which correct sterol profiles are required for regulated auxin and ethylene signaling through effects on membrane function.},
  author       = {Souter, Martin and Topping, Jennifer and Pullen, Margaret and Friml, Jirí and Palme, Klaus and Hackett, Rachel and Grierson, Don and Lindsey, Keith},
  issn         = {1040-4651},
  journal      = {Plant Cell},
  number       = {5},
  pages        = {1017 -- 1031},
  publisher    = {American Society of Plant Biologists},
  title        = {{Hydra mutants of Arabidopsis are defective in sterol profiles and auxin and ethylene signaling}},
  doi          = {10.1105/tpc.001248},
  volume       = {14},
  year         = {2002},
}

@article{2988,
  abstract     = {Coordination of cell and tissue polarity commonly involves directional signaling [1]. In the Arabidopsis root epidermis, cell polarity is revealed by basal, root tip-oriented, hair outgrowth from hair-forming cells (trichoblasts) [2]. The plant hormone auxin displays polar movements [1, 3] and accumulates at maximum concentration in the root tip [4, 5]. The application of polar auxin transport inhibitors [3] evokes changes in trichoblast polarity only at high concentrations and after long-term application [2, 4]. Thus, it remains open whether components of the auxin transport machinery mediate establishment of trichoblast polarity. Here we report that the presumptive auxin influx carrier AUX1 [6, 7] contributes to apical-basal hair cell polarity. AUX1 function is required for polarity changes induced by exogenous application of the auxin 2,4-D, a preferential influx carrier substrate. Similar to aux1 mutants, the vesicle trafficking inhibitor brefeldin A (BFA) interferes with polar hair initiation, and AUX1 function is required for BFA-mediated polarity changes. Consistently, BFA inhibits membrane trafficking of AUX1, trichoblast hyperpolarization induced by 2,4-D, and alters the distal auxin maximum. Our results identify AUX1 as one component of a novel BFA-sensitive auxin transport pathway polarizing cells toward a hormone maximum.},
  author       = {Grebe, Markus and Friml, Jirí and Swarup, Ranjan and Ljung, Karin and Sandberg, Göran and Terlou, Maarten and Palme, Klaus and Bennett, Malcolm and Scheres, Ben},
  issn         = {0960-9822},
  journal      = {Current Biology},
  number       = {4},
  pages        = {329 -- 334},
  publisher    = {Cell Press},
  title        = {{Cell polarity signaling in Arabidopsis involves a BFA sensitive auxin influx pathway}},
  doi          = {10.1016/S0960-9822(02)00654-1},
  volume       = {12},
  year         = {2002},
}

@article{2989,
  abstract     = {In contrast to animals, little is known about pattern formation in plants. Physiological and genetic data suggest the involvement of the phytohormone auxin in this process. Here, we characterize a novel member of the PIN family of putative auxin efflux carriers, Arabidopsis PIN4, that is localized in developing and mature root meristems. Atpin4 mutants are defective in establishment and maintenance of endogenous auxin gradients, fail to canalize externally applied auxin, and display various patterning defects in both embryonic and seedling roots. We propose a role for AtPIN4 in generating a sink for auxin below the quiescent center of the root meristem that is essential for auxin distribution and patterning.},
  author       = {Friml, Jirí and Benková, Eva and Blilou, Ikram and Wiśniewska, Justyna and Hamann, Thorsten and Ljung, Karin and Woody, Scott and Sandberg, Göran and Scheres, Ben and Jürgens, Gerd and Palme, Klaus},
  issn         = {0092-8674},
  journal      = {Cell},
  number       = {5},
  pages        = {661 -- 673},
  publisher    = {Cell Press},
  title        = {{AtPIN4 mediates sink-driven auxin gradients and root patterning in Arabidopsis}},
  doi          = {10.1016/S0092-8674(02)00656-6},
  volume       = {108},
  year         = {2002},
}

@article{2991,
  abstract     = {Polar auxin transport controls multiple aspects of plant development including differential growth, embryo and root patterning and vascular tissue differentiation. Identification of proteins involved in this process and availability of new tools enabling `visualization' of auxin and auxin routes in planta largely contributed to the significant progress that has recently been made. New data support classical concepts, but several recent findings are likely to challenge our view on the mechanism of auxin transport. The aim of this review is to provide a comprehensive overview of the polar auxin transport field. It starts with classical models resulting from physiological studies, describes the genetic contributions and discusses the molecular basis of auxin influx and efflux. Finally, selected questions are presented in the context of developmental biology, integrating available data from different fields.},
  author       = {Friml, Jirí and Palme, Klaus},
  journal      = {Plant Molecular Biology},
  number       = {3-4},
  pages        = {273 -- 284},
  publisher    = {Springer},
  title        = {{Polar auxin transport - Old questions and new concepts?}},
  doi          = {10.1023/A:1015248926412},
  volume       = {49},
  year         = {2002},
}

@article{12659,
  abstract     = {For many years considerable efforts have been put into investigating and modelling hydrological processes of mountainous catchments. On the one hand, the complexity and intrinsically high variability of the involved processes as well as insufficient knowledge of the underlying physical mechanisms still induce large uncertainties in understanding observed phenomena and predicting the behaviour of the system. On the other hand, the demand for models that are able to simulate mountainous water resource systems is increasing because of the needs related to both water exploitation and water conservation, which clearly call for an integrated vision and modelling of these systems.
Accordingly, this paper moves from a brief survey of the most significant achievements in mountain hydrology to discuss what could be future challenging issues related to the broader spectrum of questions, which hydrologic modelling of mountainous river systems may face in the next decades. Firstly, reference is made to existing methodologies for modelling alpine water systems, focussing on some specific aspects that provide a basis for the discussion of the weaknesses and perspectives of present simulation tools. The future is thus discussed, delineating some of the research challenges that may foster a comprehensive and integrated vision of water related issues in mountainous regions.},
  author       = {Burlando, Paolo and Pellicciotti, Francesca and Strasser, Ulrich},
  issn         = {2224-7955},
  journal      = {Hydrology Research},
  number       = {1},
  pages        = {47--74},
  publisher    = {IWA Publishing},
  title        = {{Modelling mountainous water systems between learning and speculating looking for challenges}},
  doi          = {10.2166/nh.2002.0004},
  volume       = {33},
  year         = {2002},
}

@article{21646,
  abstract     = {Specimen preparation using abrasives results in surface and subsurface mechanical (stresses, strains), geometrical (roughness), chemical (contaminants, reaction products) and physical modifications (structure, texture, lattice defects). The mechanisms involved in polishing with abrasives are presented to illustrate the effects of surface topography, surface and subsurface composition and induced lattice defects on the accuracy of quantitative x-ray microanalysis of mineral materials with the electron probe microanalyzer (EPMA).},
  author       = {Remond, G. and Nockolds, C. and Phillips, M. and Roques-Carmes, Charles},
  issn         = {1044-677X},
  journal      = {Journal of Research of the National Institute of Standards and Technology},
  keywords     = {abrasive wear, bound abrasives, chemical-mechanical polishing, loose abrasives, polishing, surface and subsurface damage, x-ray microanalysis.},
  number       = {6},
  pages        = {639--662},
  publisher    = {National Institute of Standards and Technology},
  title        = {{Implications of polishing techniques in quantitative X-ray microanalysis}},
  doi          = {10.6028/jres.107.052},
  volume       = {107},
  year         = {2002},
}

@article{13438,
  abstract     = {ICln is an ion channel identified by expression cloning using a cDNA library from Madin-Darby canine kidney cells. In all organisms tested so far, only one transcript for the ICln protein could be identified. Here we show that two splice variants of the ICln ion channel can be found in Caenorhabditis elegans. Moreover, we show that these two splice variants of the ICln channel protein, which we termed IClnN1 and IClnN2, can be functionally reconstituted and tested in an artificial lipid bilayer. In these experiments, the IClnN1-induced currents showed no voltage-dependent inactivation, whereas the IClnN2-induced currents fully inactivated at positive potentials. The molecular entity responsible for the voltage-dependent inactivation of IClnN2 is a cluster of positively charged amino acids encoded by exon 2a, which is absent in IClnN1. Our experiments suggest a mechanism of channel inactivation that is similar to the “ball and chain” model proposed for the Shaker potassium channel,i.e. a cluster of positively charged amino acids hinders ion permeation through the channel by a molecular and voltage-dependent interaction at the inner vestibulum of the pore. This hypothesis is supported by the finding that synthetic peptides with the same amino acid sequence as the positive cluster can transform the IClnN1-induced current to the current observed after reconstitution of IClnN2. Furthermore, we show that the nematode ICln gene is embedded in an operon harboring two additional genes, which we termed Nx and Ny. Co-reconstitution of Nx and IClnN2 and functional analysis of the related currents revealed a functional interaction between the two proteins, as evidenced by the fact that the IClnN2-induced current in the presence of Nx was no longer voltage-sensitive. The experiments described indicate that the genome organization in nematodes allows an effective approach for the identification of functional partner proteins of ion channels.},
  author       = {Fürst, Johannes and Ritter, Markus and Rudzki, Jakob and Danzl, Johann G and Gschwentner, Martin and Scandella, Elke and Jakab, Martin and König, Matthias and Oehl, Bernhard and Lang, Florian and Deetjen, Peter and Paulmichl, Markus},
  issn         = {0021-9258},
  journal      = {Journal of Biological Chemistry},
  keywords     = {Cell Biology, Molecular Biology, Biochemistry},
  number       = {6},
  pages        = {4435--4445},
  publisher    = {Elsevier},
  title        = {{ICln Ion channel splice variants in Caenorhabditis elegans}},
  doi          = {10.1074/jbc.m107372200},
  volume       = {277},
  year         = {2002},
}

@article{1451,
  abstract     = {Extending work of Bielawski-Dancer 3 and Konno 14, we develop a theory of toric hyperkähler varieties, which involves toric geometry, matroid theory and convex polyhedra. The framework is a detailed study of semi-projective toric varieties, meaning GIT quotients of affine spaces by torus actions, and specifically, of Lawrence toric varieties, meaning GIT quotients of even-dimensional affine spaces by symplectic torus actions. A toric hyperkähler variety is a complete intersection in a Lawrence toric variety. Both varieties are non-compact, and they share the same cohomology ring, namely, the Stanley-Reisner ring of a matroid modulo a linear system of parameters. Familiar applications of toric geometry to combinatorics, including the Hard Lefschetz Theorem and the volume polynomials of Khovanskii-Pukhlikov 11, are extended to the hyperkähler setting. When the matroid is graphic, our construction gives the toric quiver varieties, in the sense of Nakajima 17.},
  author       = {Hausel, Tamas and Sturmfels, Bernd},
  issn         = {1431-0635},
  journal      = {Documenta Mathematica},
  number       = {1},
  pages        = {495 -- 534},
  publisher    = {Deutsche Mathematiker Vereinigung},
  title        = {{Toric hyperkähler varieties}},
  doi          = {10.4171/DM/130},
  volume       = {7},
  year         = {2002},
}

@article{1458,
  abstract     = {The moduli space of stable bundles of rank $2$ and degree $1$ on a Riemann surface has rational cohomology generated by the so-called universal classes. The work of Baranovsky, King-Newstead, Siebert-Tian and Zagier provided a complete set of relations between these classes, expressed in terms of a recursion in the genus. This paper accomplishes the same thing for the noncompact moduli spaces of Higgs bundles, in the sense of Hitchin and Simpson. There are many more independent relations than for stable bundles, but in a sense the answer is simpler, since the formulas are completely explicit, not recursive. The results of Kirwan on equivariant cohomology for holomorphic circle actions are of key importance.},
  author       = {Hausel, Tamas and Thaddeus, Michael},
  issn         = {1088-6834},
  journal      = {Journal of the American Mathematical Society},
  pages        = {303 -- 329},
  publisher    = {American Mathematical Society},
  title        = {{Relations in the cohomology ring of the moduli space of rank 2 Higgs bundles}},
  doi          = {10.1090/S0894-0347-02-00417-4},
  volume       = {16},
  year         = {2002},
}