[{"year":"2018","external_id":{"isi":["000443832300005"]},"quality_controlled":"1","isi":1,"page":"489-501","language":[{"iso":"eng"}],"oa_version":"Published Version","day":"01","publist_id":"7281","title":"Communication-efficient randomized consensus","date_updated":"2026-04-16T09:53:54Z","abstract":[{"text":"We consider the problem of consensus in the challenging classic model. In this model, the adversary is adaptive; it can choose which processors crash at any point during the course of the algorithm. Further, communication is via asynchronous message passing: there is no known upper bound on the time to send a message from one processor to another, and all messages and coin flips are seen by the adversary. We describe a new randomized consensus protocol with expected message complexity O(n2log2n) when fewer than n / 2 processes may fail by crashing. This is an almost-linear improvement over the best previously known protocol, and within logarithmic factors of a known Ω(n2) message lower bound. The protocol further ensures that no process sends more than O(nlog3n) messages in expectation, which is again within logarithmic factors of optimal. We also present a generalization of the algorithm to an arbitrary number of failures t, which uses expected O(nt+t2log2t) total messages. Our approach is to build a message-efficient, resilient mechanism for aggregating individual processor votes, implementing the message-passing equivalent of a weak shared coin. Roughly, in our protocol, a processor first announces its votes to small groups, then propagates them to increasingly larger groups as it generates more and more votes. To bound the number of messages that an individual process might have to send or receive, the protocol progressively increases the weight of generated votes. The main technical challenge is bounding the impact of votes that are still “in flight” (generated, but not fully propagated) on the final outcome of the shared coin, especially since such votes might have different weights. We achieve this by leveraging the structure of the algorithm, and a technical argument based on martingale concentration bounds. Overall, we show that it is possible to build an efficient message-passing implementation of a shared coin, and in the process (almost-optimally) solve the classic consensus problem in the asynchronous message-passing model.","lang":"eng"}],"file":[{"relation":"main_file","file_size":595707,"file_id":"5867","content_type":"application/pdf","checksum":"69b46e537acdcac745237ddb853fcbb5","creator":"dernst","date_created":"2019-01-22T07:25:51Z","file_name":"2017_DistribComp_Alistarh.pdf","date_updated":"2020-07-14T12:46:38Z","access_level":"open_access"}],"intvolume":"        31","doi":"10.1007/s00446-017-0315-1","status":"public","file_date_updated":"2020-07-14T12:46:38Z","type":"journal_article","corr_author":"1","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"author":[{"full_name":"Alistarh, Dan-Adrian","id":"4A899BFC-F248-11E8-B48F-1D18A9856A87","first_name":"Dan-Adrian","last_name":"Alistarh","orcid":"0000-0003-3650-940X"},{"last_name":"Aspnes","first_name":"James","full_name":"Aspnes, James"},{"last_name":"King","first_name":"Valerie","full_name":"King, Valerie"},{"full_name":"Saia, Jared","first_name":"Jared","last_name":"Saia"}],"ddc":["000"],"issue":"6","date_created":"2018-12-11T11:47:01Z","month":"11","publication_status":"published","date_published":"2018-11-01T00:00:00Z","user_id":"ba8df636-2132-11f1-aed0-ed93e2281fdd","department":[{"_id":"DaAl"}],"article_processing_charge":"Yes (via OA deal)","citation":{"ieee":"D.-A. Alistarh, J. Aspnes, V. King, and J. Saia, “Communication-efficient randomized consensus,” <i>Distributed Computing</i>, vol. 31, no. 6. Springer, pp. 489–501, 2018.","mla":"Alistarh, Dan-Adrian, et al. “Communication-Efficient Randomized Consensus.” <i>Distributed Computing</i>, vol. 31, no. 6, Springer, 2018, pp. 489–501, doi:<a href=\"https://doi.org/10.1007/s00446-017-0315-1\">10.1007/s00446-017-0315-1</a>.","apa":"Alistarh, D.-A., Aspnes, J., King, V., &#38; Saia, J. (2018). Communication-efficient randomized consensus. <i>Distributed Computing</i>. Springer. <a href=\"https://doi.org/10.1007/s00446-017-0315-1\">https://doi.org/10.1007/s00446-017-0315-1</a>","ista":"Alistarh D-A, Aspnes J, King V, Saia J. 2018. Communication-efficient randomized consensus. Distributed Computing. 31(6), 489–501.","short":"D.-A. Alistarh, J. Aspnes, V. King, J. Saia, Distributed Computing 31 (2018) 489–501.","ama":"Alistarh D-A, Aspnes J, King V, Saia J. Communication-efficient randomized consensus. <i>Distributed Computing</i>. 2018;31(6):489-501. doi:<a href=\"https://doi.org/10.1007/s00446-017-0315-1\">10.1007/s00446-017-0315-1</a>","chicago":"Alistarh, Dan-Adrian, James Aspnes, Valerie King, and Jared Saia. “Communication-Efficient Randomized Consensus.” <i>Distributed Computing</i>. Springer, 2018. <a href=\"https://doi.org/10.1007/s00446-017-0315-1\">https://doi.org/10.1007/s00446-017-0315-1</a>."},"volume":31,"project":[{"name":"IST Austria Open Access Fund","_id":"B67AFEDC-15C9-11EA-A837-991A96BB2854"}],"has_accepted_license":"1","oa":1,"publisher":"Springer","scopus_import":"1","fulldoi":"https://doi.org/10.1007/s00446-017-0315-1","publication_identifier":{"issn":["0178-2770"]},"_id":"536","publication":"Distributed Computing"},{"_id":"54","publication":"Developmental Cell","publisher":"Cell Press","scopus_import":"1","fulldoi":"https://doi.org/10.1016/j.devcel.2018.09.014","oa":1,"citation":{"chicago":"Nunes Pinheiro, Diana C, and Yohanns Bellaïche. “Mechanical Force-Driven Adherents Junction Remodeling and Epithelial Dynamics.” <i>Developmental Cell</i>. Cell Press, 2018. <a href=\"https://doi.org/10.1016/j.devcel.2018.09.014\">https://doi.org/10.1016/j.devcel.2018.09.014</a>.","ama":"Nunes Pinheiro DC, Bellaïche Y. Mechanical force-driven adherents junction remodeling and epithelial dynamics. <i>Developmental Cell</i>. 2018;47(1):3-19. doi:<a href=\"https://doi.org/10.1016/j.devcel.2018.09.014\">10.1016/j.devcel.2018.09.014</a>","short":"D.C. Nunes Pinheiro, Y. Bellaïche, Developmental Cell 47 (2018) 3–19.","mla":"Nunes Pinheiro, Diana C., and Yohanns Bellaïche. “Mechanical Force-Driven Adherents Junction Remodeling and Epithelial Dynamics.” <i>Developmental Cell</i>, vol. 47, no. 1, Cell Press, 2018, pp. 3–19, doi:<a href=\"https://doi.org/10.1016/j.devcel.2018.09.014\">10.1016/j.devcel.2018.09.014</a>.","apa":"Nunes Pinheiro, D. C., &#38; Bellaïche, Y. (2018). Mechanical force-driven adherents junction remodeling and epithelial dynamics. <i>Developmental Cell</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.devcel.2018.09.014\">https://doi.org/10.1016/j.devcel.2018.09.014</a>","ista":"Nunes Pinheiro DC, Bellaïche Y. 2018. Mechanical force-driven adherents junction remodeling and epithelial dynamics. Developmental Cell. 47(1), 3–19.","ieee":"D. C. Nunes Pinheiro and Y. Bellaïche, “Mechanical force-driven adherents junction remodeling and epithelial dynamics,” <i>Developmental Cell</i>, vol. 47, no. 1. Cell Press, pp. 3–19, 2018."},"volume":47,"article_processing_charge":"No","department":[{"_id":"CaHe"}],"article_type":"review","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","month":"10","publication_status":"published","date_published":"2018-10-08T00:00:00Z","issue":"1","acknowledgement":"Research in the Bellaïche laboratory is supported by the European Research Council (ERC Advanced, TiMoprh, 340784), the Fondation ARC pour la Recherche sur le Cancer (SL220130607097), the Agence Nationale de la Recherche (ANR lLabex DEEP; 11-LBX-0044, ANR-10-IDEX-0001-02), the Centre National de la Recherche Scientifique, the Institut National de la Santé et de la Recherche Médicale, and Institut Curie and PSL Research University funding or grants.","main_file_link":[{"url":"https://doi.org/10.1016/j.devcel.2018.09.014","open_access":"1"}],"date_created":"2018-12-11T11:44:23Z","ddc":["570"],"author":[{"id":"2E839F16-F248-11E8-B48F-1D18A9856A87","first_name":"Diana C","last_name":"Nunes Pinheiro","orcid":"0000-0003-4333-7503","full_name":"Nunes Pinheiro, Diana C"},{"last_name":"Bellaïche","first_name":"Yohanns","full_name":"Bellaïche, Yohanns"}],"status":"public","doi":"10.1016/j.devcel.2018.09.014","OA_type":"free access","type":"journal_article","abstract":[{"text":"During epithelial tissue development, repair, and homeostasis, adherens junctions (AJs) ensure intercellular adhesion and tissue integrity while allowing for cell and tissue dynamics. Mechanical forces play critical roles in AJs’ composition and dynamics. Recent findings highlight that beyond a well-established role in reinforcing cell-cell adhesion, AJ mechanosensitivity promotes junctional remodeling and polarization, thereby regulating critical processes such as cell intercalation, division, and collective migration. Here, we provide an integrated view of mechanosensing mechanisms that regulate cell-cell contact composition, geometry, and integrity under tension and highlight pivotal roles for mechanosensitive AJ remodeling in preserving epithelial integrity and sustaining tissue dynamics.","lang":"eng"}],"intvolume":"        47","language":[{"iso":"eng"}],"oa_version":"Published Version","publist_id":"8000","title":"Mechanical force-driven adherents junction remodeling and epithelial dynamics","date_updated":"2026-06-18T18:52:56Z","day":"08","quality_controlled":"1","page":"3 - 19","isi":1,"year":"2018","external_id":{"isi":["000446579900002"]}},{"quality_controlled":"1","page":"186 - 191","isi":1,"year":"2018","external_id":{"isi":["000419128700049"]},"abstract":[{"text":"A central goal in theoretical neuroscience is to predict the response properties of sensory neurons from first principles. To this end, “efficient coding” posits that sensory neurons encode maximal information about their inputs given internal constraints. There exist, however, many variants of efficient coding (e.g., redundancy reduction, different formulations of predictive coding, robust coding, sparse coding, etc.), differing in their regimes of applicability, in the relevance of signals to be encoded, and in the choice of constraints. It is unclear how these types of efficient coding relate or what is expected when different coding objectives are combined. Here we present a unified framework that encompasses previously proposed efficient coding models and extends to unique regimes. We show that optimizing neural responses to encode predictive information can lead them to either correlate or decorrelate their inputs, depending on the stimulus statistics; in contrast, at low noise, efficiently encoding the past always predicts decorrelation. Later, we investigate coding of naturalistic movies and show that qualitatively different types of visual motion tuning and levels of response sparsity are predicted, depending on whether the objective is to recover the past or predict the future. Our approach promises a way to explain the observed diversity of sensory neural responses, as due to multiple functional goals and constraints fulfilled by different cell types and/or circuits.","lang":"eng"}],"intvolume":"       115","language":[{"iso":"eng"}],"oa_version":"Submitted Version","publist_id":"7273","title":"Toward a unified theory of efficient, predictive, and sparse coding","date_updated":"2025-05-14T10:55:59Z","day":"02","status":"public","doi":"10.1073/pnas.1711114115","type":"journal_article","issue":"1","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1101/152660 "}],"date_created":"2018-12-11T11:47:04Z","corr_author":"1","author":[{"full_name":"Chalk, Matthew J","orcid":"0000-0001-7782-4436","last_name":"Chalk","id":"2BAAC544-F248-11E8-B48F-1D18A9856A87","first_name":"Matthew J"},{"first_name":"Olivier","last_name":"Marre","full_name":"Marre, Olivier"},{"id":"3D494DCA-F248-11E8-B48F-1D18A9856A87","first_name":"Gasper","last_name":"Tkacik","orcid":"0000-0002-6699-1455","full_name":"Tkacik, Gasper"}],"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","month":"01","publication_status":"published","date_published":"2018-01-02T00:00:00Z","citation":{"ieee":"M. J. Chalk, O. Marre, and G. Tkačik, “Toward a unified theory of efficient, predictive, and sparse coding,” <i>Proceedings of the National Academy of Sciences of the United States of America</i>, vol. 115, no. 1. National Academy of Sciences, pp. 186–191, 2018.","mla":"Chalk, Matthew J., et al. “Toward a Unified Theory of Efficient, Predictive, and Sparse Coding.” <i>Proceedings of the National Academy of Sciences of the United States of America</i>, vol. 115, no. 1, National Academy of Sciences, 2018, pp. 186–91, doi:<a href=\"https://doi.org/10.1073/pnas.1711114115\">10.1073/pnas.1711114115</a>.","apa":"Chalk, M. J., Marre, O., &#38; Tkačik, G. (2018). Toward a unified theory of efficient, predictive, and sparse coding. <i>Proceedings of the National Academy of Sciences of the United States of America</i>. National Academy of Sciences. <a href=\"https://doi.org/10.1073/pnas.1711114115\">https://doi.org/10.1073/pnas.1711114115</a>","ista":"Chalk MJ, Marre O, Tkačik G. 2018. Toward a unified theory of efficient, predictive, and sparse coding. Proceedings of the National Academy of Sciences of the United States of America. 115(1), 186–191.","short":"M.J. Chalk, O. Marre, G. Tkačik, Proceedings of the National Academy of Sciences of the United States of America 115 (2018) 186–191.","ama":"Chalk MJ, Marre O, Tkačik G. Toward a unified theory of efficient, predictive, and sparse coding. <i>Proceedings of the National Academy of Sciences of the United States of America</i>. 2018;115(1):186-191. doi:<a href=\"https://doi.org/10.1073/pnas.1711114115\">10.1073/pnas.1711114115</a>","chicago":"Chalk, Matthew J, Olivier Marre, and Gašper Tkačik. “Toward a Unified Theory of Efficient, Predictive, and Sparse Coding.” <i>Proceedings of the National Academy of Sciences of the United States of America</i>. National Academy of Sciences, 2018. <a href=\"https://doi.org/10.1073/pnas.1711114115\">https://doi.org/10.1073/pnas.1711114115</a>."},"volume":115,"article_processing_charge":"No","department":[{"_id":"GaTk"}],"oa":1,"project":[{"_id":"254D1A94-B435-11E9-9278-68D0E5697425","call_identifier":"FWF","grant_number":"P 25651-N26","name":"Sensitivity to higher-order statistics in natural scenes"}],"_id":"543","publication":"Proceedings of the National Academy of Sciences of the United States of America","scopus_import":"1","publisher":"National Academy of Sciences","fulldoi":"https://doi.org/10.1073/pnas.1711114115"},{"oa":1,"has_accepted_license":"1","type":"technical_report","status":"public","file_date_updated":"2020-07-14T12:47:00Z","date_created":"2018-12-12T11:39:26Z","publication_identifier":{"issn":["2664-1690"]},"_id":"5457","author":[{"first_name":"1","last_name":"Anonymous","full_name":"Anonymous, 1"},{"first_name":"2","last_name":"Anonymous","full_name":"Anonymous, 2"},{"first_name":"3","last_name":"Anonymous","full_name":"Anonymous, 3"},{"first_name":"4","last_name":"Anonymous","full_name":"Anonymous, 4"},{"full_name":"Anonymous, 5","last_name":"Anonymous","first_name":"5"},{"last_name":"Anonymous","first_name":"6","full_name":"Anonymous, 6"}],"ddc":["000"],"corr_author":"1","pubrep_id":"1066","alternative_title":["IST Austria Technical Report"],"publisher":"IST Austria","scopus_import":1,"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","page":"27","related_material":{"record":[{"id":"6175","status":"public","relation":"later_version"}]},"publication_status":"published","date_published":"2018-11-11T00:00:00Z","month":"11","year":"2018","abstract":[{"text":"We consider the problem of expected cost analysis over nondeterministic probabilistic programs, which aims at automated methods for analyzing the resource-usage of such programs. Previous approaches for this problem could only handle nonnegative bounded costs. However, in many scenarios, such as queuing networks or analysis of cryptocurrency protocols, both positive and negative costs are necessary and the costs are unbounded as well.\r\n\r\nIn this work, we present a sound and efficient approach to obtain polynomial bounds on the expected accumulated cost of nondeterministic probabilistic programs. Our approach can handle (a) general positive and negative costs with bounded updates in variables; and (b) nonnegative costs with general updates to variables. We show that several natural examples which could not be handled by previous approaches are captured in our framework.\r\n\r\nMoreover, our approach leads to an efficient polynomial-time algorithm, while no previous approach for cost analysis of probabilistic programs could guarantee polynomial runtime. Finally, we show the effectiveness of our approach by presenting experimental results on a variety of programs, motivated by real-world applications, for which we efficiently synthesize tight resource-usage bounds.","lang":"eng"}],"citation":{"chicago":"Anonymous, 1, 2 Anonymous, 3 Anonymous, 4 Anonymous, 5 Anonymous, and 6 Anonymous. <i>Cost Analysis of Nondeterministic Probabilistic Programs</i>. IST Austria, 2018.","ama":"Anonymous 1, Anonymous 2, Anonymous 3, Anonymous 4, Anonymous 5, Anonymous 6. <i>Cost Analysis of Nondeterministic Probabilistic Programs</i>. IST Austria; 2018.","short":"1 Anonymous, 2 Anonymous, 3 Anonymous, 4 Anonymous, 5 Anonymous, 6 Anonymous, Cost Analysis of Nondeterministic Probabilistic Programs, IST Austria, 2018.","ista":"Anonymous 1, Anonymous 2, Anonymous 3, Anonymous 4, Anonymous 5, Anonymous 6. 2018. Cost analysis of nondeterministic probabilistic programs, IST Austria, 27p.","mla":"Anonymous, 1, et al. <i>Cost Analysis of Nondeterministic Probabilistic Programs</i>. IST Austria, 2018.","apa":"Anonymous, 1, Anonymous, 2, Anonymous, 3, Anonymous, 4, Anonymous, 5, &#38; Anonymous, 6. (2018). <i>Cost analysis of nondeterministic probabilistic programs</i>. IST Austria.","ieee":"1 Anonymous, 2 Anonymous, 3 Anonymous, 4 Anonymous, 5 Anonymous, and 6 Anonymous, <i>Cost analysis of nondeterministic probabilistic programs</i>. IST Austria, 2018."},"file":[{"file_size":4202966,"relation":"main_file","access_level":"open_access","file_name":"IST-2018-1066-v1+1_techreport.pdf","date_updated":"2020-07-14T12:47:00Z","creator":"system","checksum":"ba3adafd36fe200385ccda583063b9eb","content_type":"application/pdf","file_id":"5493","date_created":"2018-12-12T11:53:32Z"},{"date_updated":"2020-07-14T12:47:00Z","file_name":"authors-names.txt","access_level":"closed","file_id":"6402","creator":"dernst","checksum":"6cf3a19164bb8e5048a9c8c84dfd9fa3","content_type":"text/plain","date_created":"2019-05-10T13:22:12Z","file_size":322,"relation":"main_file"}],"day":"11","title":"Cost analysis of nondeterministic probabilistic programs","date_updated":"2025-04-15T08:11:42Z","language":[{"iso":"eng"}],"oa_version":"Published Version"},{"fulldoi":"https://doi.org/10.1016/j.conb.2017.12.005","publisher":"Elsevier","scopus_import":"1","publication":"Current Opinion in Neurobiology","_id":"546","publication_status":"published","date_published":"2018-02-01T00:00:00Z","month":"02","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","department":[{"_id":"GaNo"}],"article_processing_charge":"No","volume":48,"citation":{"ama":"Sacco R, Cacci E, Novarino G. Neural stem cells in neuropsychiatric disorders. <i>Current Opinion in Neurobiology</i>. 2018;48(2):131-138. doi:<a href=\"https://doi.org/10.1016/j.conb.2017.12.005\">10.1016/j.conb.2017.12.005</a>","chicago":"Sacco, Roberto, Emanuele Cacci, and Gaia Novarino. “Neural Stem Cells in Neuropsychiatric Disorders.” <i>Current Opinion in Neurobiology</i>. Elsevier, 2018. <a href=\"https://doi.org/10.1016/j.conb.2017.12.005\">https://doi.org/10.1016/j.conb.2017.12.005</a>.","short":"R. Sacco, E. Cacci, G. Novarino, Current Opinion in Neurobiology 48 (2018) 131–138.","mla":"Sacco, Roberto, et al. “Neural Stem Cells in Neuropsychiatric Disorders.” <i>Current Opinion in Neurobiology</i>, vol. 48, no. 2, Elsevier, 2018, pp. 131–38, doi:<a href=\"https://doi.org/10.1016/j.conb.2017.12.005\">10.1016/j.conb.2017.12.005</a>.","apa":"Sacco, R., Cacci, E., &#38; Novarino, G. (2018). Neural stem cells in neuropsychiatric disorders. <i>Current Opinion in Neurobiology</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.conb.2017.12.005\">https://doi.org/10.1016/j.conb.2017.12.005</a>","ista":"Sacco R, Cacci E, Novarino G. 2018. Neural stem cells in neuropsychiatric disorders. Current Opinion in Neurobiology. 48(2), 131–138.","ieee":"R. Sacco, E. Cacci, and G. Novarino, “Neural stem cells in neuropsychiatric disorders,” <i>Current Opinion in Neurobiology</i>, vol. 48, no. 2. Elsevier, pp. 131–138, 2018."},"type":"journal_article","doi":"10.1016/j.conb.2017.12.005","status":"public","author":[{"full_name":"Sacco, Roberto","first_name":"Roberto","id":"42C9F57E-F248-11E8-B48F-1D18A9856A87","last_name":"Sacco"},{"first_name":"Emanuele","last_name":"Cacci","full_name":"Cacci, Emanuele"},{"full_name":"Novarino, Gaia","first_name":"Gaia","id":"3E57A680-F248-11E8-B48F-1D18A9856A87","last_name":"Novarino","orcid":"0000-0002-7673-7178"}],"corr_author":"1","date_created":"2018-12-11T11:47:06Z","issue":"2","external_id":{"isi":["000427101600018"]},"year":"2018","isi":1,"page":"131 - 138","quality_controlled":"1","day":"01","publist_id":"7268","title":"Neural stem cells in neuropsychiatric disorders","date_updated":"2024-10-09T20:58:31Z","language":[{"iso":"eng"}],"oa_version":"None","intvolume":"        48","abstract":[{"text":"The precise control of neural stem cell (NSC) proliferation and differentiation is crucial for the development and function of the human brain. Here, we review the emerging links between the alteration of embryonic and adult neurogenesis and the etiology of neuropsychiatric disorders (NPDs) such as autism spectrum disorders (ASDs) and schizophrenia (SCZ), as well as the advances in stem cell-based modeling and the novel therapeutic targets derived from these studies.","lang":"eng"}]},{"publication":"Nature Neuroscience","_id":"547","fulldoi":"https://doi.org/10.1038/s41593-017-0053-5","publisher":"Nature Publishing Group","extern":"1","volume":21,"citation":{"ama":"Gstrein T, Edwards A, Přistoupilová A, et al. Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans. <i>Nature Neuroscience</i>. 2018;21(2):207-217. doi:<a href=\"https://doi.org/10.1038/s41593-017-0053-5\">10.1038/s41593-017-0053-5</a>","chicago":"Gstrein, Thomas, Andrew Edwards, Anna Přistoupilová, Ines Leca, Martin Breuss, Sandra Pilat Carotta, Andi H Hansen, et al. “Mutations in Vps15 Perturb Neuronal Migration in Mice and Are Associated with Neurodevelopmental Disease in Humans.” <i>Nature Neuroscience</i>. Nature Publishing Group, 2018. <a href=\"https://doi.org/10.1038/s41593-017-0053-5\">https://doi.org/10.1038/s41593-017-0053-5</a>.","apa":"Gstrein, T., Edwards, A., Přistoupilová, A., Leca, I., Breuss, M., Pilat Carotta, S., … Keays, D. (2018). Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans. <i>Nature Neuroscience</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/s41593-017-0053-5\">https://doi.org/10.1038/s41593-017-0053-5</a>","ista":"Gstrein T, Edwards A, Přistoupilová A, Leca I, Breuss M, Pilat Carotta S, Hansen AH, Tripathy R, Traunbauer A, Hochstoeger T, Rosoklija G, Repic M, Landler L, Stránecký V, Dürnberger G, Keane T, Zuber J, Adams D, Flint J, Honzik T, Gut M, Beltran S, Mechtler K, Sherr E, Kmoch S, Gut I, Keays D. 2018. Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans. Nature Neuroscience. 21(2), 207–217.","mla":"Gstrein, Thomas, et al. “Mutations in Vps15 Perturb Neuronal Migration in Mice and Are Associated with Neurodevelopmental Disease in Humans.” <i>Nature Neuroscience</i>, vol. 21, no. 2, Nature Publishing Group, 2018, pp. 207–17, doi:<a href=\"https://doi.org/10.1038/s41593-017-0053-5\">10.1038/s41593-017-0053-5</a>.","short":"T. Gstrein, A. Edwards, A. Přistoupilová, I. Leca, M. Breuss, S. Pilat Carotta, A.H. Hansen, R. Tripathy, A. Traunbauer, T. Hochstoeger, G. Rosoklija, M. Repic, L. Landler, V. Stránecký, G. Dürnberger, T. Keane, J. Zuber, D. Adams, J. Flint, T. Honzik, M. Gut, S. Beltran, K. Mechtler, E. Sherr, S. Kmoch, I. Gut, D. Keays, Nature Neuroscience 21 (2018) 207–217.","ieee":"T. Gstrein <i>et al.</i>, “Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans,” <i>Nature Neuroscience</i>, vol. 21, no. 2. Nature Publishing Group, pp. 207–217, 2018."},"article_processing_charge":"No","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publication_status":"published","date_published":"2018-06-06T00:00:00Z","month":"06","date_created":"2018-12-11T11:47:06Z","acknowledgement":"We also acknowledge the input of P. Potter and S. Wells from the mutagenesis program at MRC Harwell and the MRC funding that underpinned it (MC U142684172). We are indebted to R. Williams for modeling the VPS15 human mutation. We also thank the transgenic, bio-optics, proteomic and graphics services groups at the IMP/IMBA. We thank The National Center for Medical Genomics (LM2015091) for providing allelic frequencies in ethnically matched populations (project CZ.02.1.01/0.0/0.0/16_013/0001634). We thank Boehringer Ingelheim and the FWF for funding this research (D.A.K., I914, P24267). The human studies were funded by the European Community’s 7th Framework Program (FP7/2007-2013). S.K., A.P. and V.S. were supported by institutional programs of Charles University in Prague (UNCE 204011, PROGRES-Q26/LF1 and SVV 260367/2017). We acknowledge grants 15-28208A and RVO-VFN 64165 from the Ministry of Health of the Czech Republic and the project LQ1604 NPU II from the Ministry of Education.","issue":"2","author":[{"full_name":"Gstrein, Thomas","last_name":"Gstrein","first_name":"Thomas"},{"full_name":"Edwards, Andrew","first_name":"Andrew","last_name":"Edwards"},{"last_name":"Přistoupilová","first_name":"Anna","full_name":"Přistoupilová, Anna"},{"full_name":"Leca, Ines","first_name":"Ines","last_name":"Leca"},{"full_name":"Breuss, Martin","last_name":"Breuss","first_name":"Martin"},{"last_name":"Pilat Carotta","first_name":"Sandra","full_name":"Pilat Carotta, Sandra"},{"first_name":"Andi H","id":"38853E16-F248-11E8-B48F-1D18A9856A87","last_name":"Hansen","full_name":"Hansen, Andi H"},{"first_name":"Ratna","last_name":"Tripathy","full_name":"Tripathy, Ratna"},{"full_name":"Traunbauer, Anna","first_name":"Anna","last_name":"Traunbauer"},{"first_name":"Tobias","last_name":"Hochstoeger","full_name":"Hochstoeger, Tobias"},{"full_name":"Rosoklija, Gavril","first_name":"Gavril","last_name":"Rosoklija"},{"full_name":"Repic, Marco","first_name":"Marco","last_name":"Repic"},{"last_name":"Landler","first_name":"Lukas","full_name":"Landler, Lukas"},{"last_name":"Stránecký","first_name":"Viktor","full_name":"Stránecký, Viktor"},{"full_name":"Dürnberger, Gerhard","first_name":"Gerhard","last_name":"Dürnberger"},{"full_name":"Keane, Thomas","first_name":"Thomas","last_name":"Keane"},{"first_name":"Johannes","last_name":"Zuber","full_name":"Zuber, Johannes"},{"full_name":"Adams, David","last_name":"Adams","first_name":"David"},{"first_name":"Jonathan","last_name":"Flint","full_name":"Flint, Jonathan"},{"full_name":"Honzik, Tomas","last_name":"Honzik","first_name":"Tomas"},{"last_name":"Gut","first_name":"Marta","full_name":"Gut, Marta"},{"last_name":"Beltran","first_name":"Sergi","full_name":"Beltran, Sergi"},{"last_name":"Mechtler","first_name":"Karl","full_name":"Mechtler, Karl"},{"last_name":"Sherr","first_name":"Elliott","full_name":"Sherr, Elliott"},{"full_name":"Kmoch, Stanislav","last_name":"Kmoch","first_name":"Stanislav"},{"last_name":"Gut","first_name":"Ivo","full_name":"Gut, Ivo"},{"first_name":"David","last_name":"Keays","full_name":"Keays, David"}],"type":"journal_article","status":"public","doi":"10.1038/s41593-017-0053-5","intvolume":"        21","abstract":[{"text":"The formation of the vertebrate brain requires the generation, migration, differentiation and survival of neurons. Genetic mutations that perturb these critical cellular events can result in malformations of the telencephalon, providing a molecular window into brain development. Here we report the identification of an N-ethyl-N-nitrosourea-induced mouse mutant characterized by a fractured hippocampal pyramidal cell layer, attributable to defects in neuronal migration. We show that this is caused by a hypomorphic mutation in Vps15 that perturbs endosomal-lysosomal trafficking and autophagy, resulting in an upregulation of Nischarin, which inhibits Pak1 signaling. The complete ablation of Vps15 results in the accumulation of autophagic substrates, the induction of apoptosis and severe cortical atrophy. Finally, we report that mutations in VPS15 are associated with cortical atrophy and epilepsy in humans. These data highlight the importance of the Vps15-Vps34 complex and the Nischarin-Pak1 signaling hub in the development of the telencephalon.","lang":"eng"}],"title":"Mutations in Vps15 perturb neuronal migration in mice and are associated with neurodevelopmental disease in humans","date_updated":"2023-09-13T08:59:52Z","publist_id":"7267","day":"06","oa_version":"None","language":[{"iso":"eng"}],"page":"207 - 217","isi":1,"external_id":{"isi":["000424269900012"]},"year":"2018"},{"quality_controlled":"1","page":"R1139 - R1140","isi":1,"year":"2018","external_id":{"isi":["000446693400008"]},"abstract":[{"lang":"eng","text":"Many animals use antimicrobials to prevent or cure disease [1,2]. For example, some animals will ingest plants with medicinal properties, both prophylactically to prevent infection and therapeutically to self-medicate when sick. Antimicrobial substances are also used as topical disinfectants, to prevent infection, protect offspring and to sanitise their surroundings [1,2]. Social insects (ants, bees, wasps and termites) build nests in environments with a high abundance and diversity of pathogenic microorganisms — such as soil and rotting wood — and colonies are often densely crowded, creating conditions that favour disease outbreaks. Consequently, social insects have evolved collective disease defences to protect their colonies from epidemics. These traits can be seen as functionally analogous to the immune system of individual organisms [3,4]. This ‘social immunity’ utilises antimicrobials to prevent and eradicate infections, and to keep the brood and nest clean. However, these antimicrobial compounds can be harmful to the insects themselves, and it is unknown how colonies prevent collateral damage when using them. Here, we demonstrate that antimicrobial acids, produced by workers to disinfect the colony, are harmful to the delicate pupal brood stage, but that the pupae are protected from the acids by the presence of a silk cocoon. Garden ants spray their nests with an antimicrobial poison to sanitize contaminated nestmates and brood. Here, Pull et al show that they also prophylactically sanitise their colonies, and that the silk cocoon serves as a barrier to protect developing pupae, thus preventing collateral damage during nest sanitation."}],"intvolume":"        28","oa_version":"Published Version","language":[{"iso":"eng"}],"title":"Protection against the lethal side effects of social immunity in ants","publist_id":"7999","date_updated":"2026-06-18T18:53:43Z","day":"08","doi":"10.1016/j.cub.2018.08.063","status":"public","type":"journal_article","issue":"19","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1016/j.cub.2018.08.063"}],"date_created":"2018-12-11T11:44:23Z","ddc":["570"],"author":[{"full_name":"Pull, Christopher","orcid":"0000-0003-1122-3982","last_name":"Pull","id":"3C7F4840-F248-11E8-B48F-1D18A9856A87","first_name":"Christopher"},{"first_name":"Sina","id":"48204546-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0002-9547-2494","last_name":"Metzler","full_name":"Metzler, Sina"},{"first_name":"Elisabeth","id":"31757262-F248-11E8-B48F-1D18A9856A87","last_name":"Naderlinger","full_name":"Naderlinger, Elisabeth"},{"first_name":"Sylvia","id":"2F64EC8C-F248-11E8-B48F-1D18A9856A87","last_name":"Cremer","orcid":"0000-0002-2193-3868","full_name":"Cremer, Sylvia"}],"article_type":"original","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","month":"10","date_published":"2018-10-08T00:00:00Z","publication_status":"published","citation":{"ieee":"C. Pull, S. Metzler, E. Naderlinger, and S. Cremer, “Protection against the lethal side effects of social immunity in ants,” <i>Current Biology</i>, vol. 28, no. 19. Cell Press, pp. R1139–R1140, 2018.","mla":"Pull, Christopher, et al. “Protection against the Lethal Side Effects of Social Immunity in Ants.” <i>Current Biology</i>, vol. 28, no. 19, Cell Press, 2018, pp. R1139–40, doi:<a href=\"https://doi.org/10.1016/j.cub.2018.08.063\">10.1016/j.cub.2018.08.063</a>.","ista":"Pull C, Metzler S, Naderlinger E, Cremer S. 2018. Protection against the lethal side effects of social immunity in ants. Current Biology. 28(19), R1139–R1140.","apa":"Pull, C., Metzler, S., Naderlinger, E., &#38; Cremer, S. (2018). Protection against the lethal side effects of social immunity in ants. <i>Current Biology</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.cub.2018.08.063\">https://doi.org/10.1016/j.cub.2018.08.063</a>","short":"C. Pull, S. Metzler, E. Naderlinger, S. Cremer, Current Biology 28 (2018) R1139–R1140.","chicago":"Pull, Christopher, Sina Metzler, Elisabeth Naderlinger, and Sylvia Cremer. “Protection against the Lethal Side Effects of Social Immunity in Ants.” <i>Current Biology</i>. Cell Press, 2018. <a href=\"https://doi.org/10.1016/j.cub.2018.08.063\">https://doi.org/10.1016/j.cub.2018.08.063</a>.","ama":"Pull C, Metzler S, Naderlinger E, Cremer S. Protection against the lethal side effects of social immunity in ants. <i>Current Biology</i>. 2018;28(19):R1139-R1140. doi:<a href=\"https://doi.org/10.1016/j.cub.2018.08.063\">10.1016/j.cub.2018.08.063</a>"},"volume":28,"article_processing_charge":"No","department":[{"_id":"SyCr"}],"oa":1,"_id":"55","publication":"Current Biology","publisher":"Cell Press","scopus_import":"1","fulldoi":"https://doi.org/10.1016/j.cub.2018.08.063"},{"oa":1,"project":[{"call_identifier":"FWF","grant_number":"P27533_N27","name":"Structure of the Excitation Spectrum for Many-Body Quantum Systems","_id":"25C878CE-B435-11E9-9278-68D0E5697425"}],"arxiv":1,"publication":"Communications in Mathematical Physics","publication_identifier":{"issn":["0010-3616"]},"_id":"554","fulldoi":"https://doi.org/10.1007/s00220-017-3064-x","publisher":"Springer","scopus_import":"1","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","date_published":"2018-05-01T00:00:00Z","publication_status":"published","month":"05","volume":360,"citation":{"short":"M.M. Napiórkowski, R. Reuvers, J. Solovej, Communications in Mathematical Physics 360 (2018) 347–403.","ista":"Napiórkowski MM, Reuvers R, Solovej J. 2018. The Bogoliubov free energy functional II: The dilute Limit. Communications in Mathematical Physics. 360(1), 347–403.","mla":"Napiórkowski, Marcin M., et al. “The Bogoliubov Free Energy Functional II: The Dilute Limit.” <i>Communications in Mathematical Physics</i>, vol. 360, no. 1, Springer, 2018, pp. 347–403, doi:<a href=\"https://doi.org/10.1007/s00220-017-3064-x\">10.1007/s00220-017-3064-x</a>.","apa":"Napiórkowski, M. M., Reuvers, R., &#38; Solovej, J. (2018). The Bogoliubov free energy functional II: The dilute Limit. <i>Communications in Mathematical Physics</i>. Springer. <a href=\"https://doi.org/10.1007/s00220-017-3064-x\">https://doi.org/10.1007/s00220-017-3064-x</a>","ieee":"M. M. Napiórkowski, R. Reuvers, and J. Solovej, “The Bogoliubov free energy functional II: The dilute Limit,” <i>Communications in Mathematical Physics</i>, vol. 360, no. 1. Springer, pp. 347–403, 2018.","ama":"Napiórkowski MM, Reuvers R, Solovej J. The Bogoliubov free energy functional II: The dilute Limit. <i>Communications in Mathematical Physics</i>. 2018;360(1):347-403. doi:<a href=\"https://doi.org/10.1007/s00220-017-3064-x\">10.1007/s00220-017-3064-x</a>","chicago":"Napiórkowski, Marcin M, Robin Reuvers, and Jan Solovej. “The Bogoliubov Free Energy Functional II: The Dilute Limit.” <i>Communications in Mathematical Physics</i>. Springer, 2018. <a href=\"https://doi.org/10.1007/s00220-017-3064-x\">https://doi.org/10.1007/s00220-017-3064-x</a>."},"article_processing_charge":"No","department":[{"_id":"RoSe"}],"type":"journal_article","doi":"10.1007/s00220-017-3064-x","status":"public","date_created":"2018-12-11T11:47:09Z","main_file_link":[{"url":"https://arxiv.org/abs/1511.05953","open_access":"1"}],"issue":"1","author":[{"full_name":"Napiórkowski, Marcin M","last_name":"Napiórkowski","id":"4197AD04-F248-11E8-B48F-1D18A9856A87","first_name":"Marcin M"},{"full_name":"Reuvers, Robin","last_name":"Reuvers","first_name":"Robin"},{"full_name":"Solovej, Jan","last_name":"Solovej","first_name":"Jan"}],"page":"347-403","quality_controlled":"1","external_id":{"arxiv":["1511.05953"]},"year":"2018","intvolume":"       360","abstract":[{"lang":"eng","text":"We analyse the canonical Bogoliubov free energy functional in three dimensions at low temperatures in the dilute limit. We prove existence of a first-order phase transition and, in the limit (Formula presented.), we determine the critical temperature to be (Formula presented.) to leading order. Here, (Formula presented.) is the critical temperature of the free Bose gas, ρ is the density of the gas and a is the scattering length of the pair-interaction potential V. We also prove asymptotic expansions for the free energy. In particular, we recover the Lee–Huang–Yang formula in the limit (Formula presented.)."}],"day":"01","publist_id":"7260","title":"The Bogoliubov free energy functional II: The dilute Limit","date_updated":"2025-07-10T11:52:52Z","oa_version":"Submitted Version","language":[{"iso":"eng"}]},{"day":"01","publist_id":"7259","title":"Glycosaminoglycans in extracellular matrix organisation: Are concepts from soft matter physics key to understanding the formation of perineuronal nets?","date_updated":"2023-09-11T14:07:03Z","oa_version":"Submitted Version","language":[{"iso":"eng"}],"intvolume":"        50","abstract":[{"text":"Conventional wisdom has it that proteins fold and assemble into definite structures, and that this defines their function. Glycosaminoglycans (GAGs) are different. In most cases the structures they form have a low degree of order, even when interacting with proteins. Here, we discuss how physical features common to all GAGs — hydrophilicity, charge, linearity and semi-flexibility — underpin the overall properties of GAG-rich matrices. By integrating soft matter physics concepts (e.g. polymer brushes and phase separation) with our molecular understanding of GAG–protein interactions, we can better comprehend how GAG-rich matrices assemble, what their properties are, and how they function. Taking perineuronal nets (PNNs) — a GAG-rich matrix enveloping neurons — as a relevant example, we propose that microphase separation determines the holey PNN anatomy that is pivotal to PNN functions.","lang":"eng"}],"external_id":{"isi":["000443661300011"]},"year":"2018","isi":1,"page":"65 - 74","quality_controlled":"1","author":[{"last_name":"Richter","first_name":"Ralf","full_name":"Richter, Ralf"},{"full_name":"Baranova, Natalia","first_name":"Natalia","id":"38661662-F248-11E8-B48F-1D18A9856A87","last_name":"Baranova","orcid":"0000-0002-3086-9124"},{"full_name":"Day, Anthony","first_name":"Anthony","last_name":"Day"},{"full_name":"Kwok, Jessica","first_name":"Jessica","last_name":"Kwok"}],"date_created":"2018-12-11T11:47:09Z","main_file_link":[{"url":"http://eprints.whiterose.ac.uk/125524/","open_access":"1"}],"acknowledgement":"This work was supported by the European Research Council [Starting Grant 306435 ‘JELLY’; to RPR], the Spanish Ministry of Competitiveness and Innovation [MAT2014-54867-R, to RPR], the EPSRC Centre for Doctoral Training in Tissue Engineering and Regenerative Medicine — Innovation in Medical and Biological Engineering [EP/L014823/1, to JCFK], the Royal Society [RG160410, to JCFK], Wings for Life [WFL-UK-008/15, to JCFK] and the European Union, the Operational Programme Research, Development and Education in the framework of the project ‘Centre of Reconstructive Neuroscience’ [CZ.02.1.01/0.0./0.0/15_003/0000419, to JCFK]. AJD would like to thank Arthritis Research UK [16539, 19489] and the MRC [76445, G0900538] for funding his work on GAG–protein interactions.\r\n","type":"journal_article","status":"public","doi":"10.1016/j.sbi.2017.12.002","department":[{"_id":"MaLo"}],"article_processing_charge":"No","volume":50,"citation":{"chicago":"Richter, Ralf, Natalia S. Baranova, Anthony Day, and Jessica Kwok. “Glycosaminoglycans in Extracellular Matrix Organisation: Are Concepts from Soft Matter Physics Key to Understanding the Formation of Perineuronal Nets?” <i>Current Opinion in Structural Biology</i>. Elsevier, 2018. <a href=\"https://doi.org/10.1016/j.sbi.2017.12.002\">https://doi.org/10.1016/j.sbi.2017.12.002</a>.","ama":"Richter R, Baranova NS, Day A, Kwok J. Glycosaminoglycans in extracellular matrix organisation: Are concepts from soft matter physics key to understanding the formation of perineuronal nets? <i>Current Opinion in Structural Biology</i>. 2018;50:65-74. doi:<a href=\"https://doi.org/10.1016/j.sbi.2017.12.002\">10.1016/j.sbi.2017.12.002</a>","ieee":"R. Richter, N. S. Baranova, A. Day, and J. Kwok, “Glycosaminoglycans in extracellular matrix organisation: Are concepts from soft matter physics key to understanding the formation of perineuronal nets?,” <i>Current Opinion in Structural Biology</i>, vol. 50. Elsevier, pp. 65–74, 2018.","apa":"Richter, R., Baranova, N. S., Day, A., &#38; Kwok, J. (2018). Glycosaminoglycans in extracellular matrix organisation: Are concepts from soft matter physics key to understanding the formation of perineuronal nets? <i>Current Opinion in Structural Biology</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.sbi.2017.12.002\">https://doi.org/10.1016/j.sbi.2017.12.002</a>","ista":"Richter R, Baranova NS, Day A, Kwok J. 2018. Glycosaminoglycans in extracellular matrix organisation: Are concepts from soft matter physics key to understanding the formation of perineuronal nets? Current Opinion in Structural Biology. 50, 65–74.","mla":"Richter, Ralf, et al. “Glycosaminoglycans in Extracellular Matrix Organisation: Are Concepts from Soft Matter Physics Key to Understanding the Formation of Perineuronal Nets?” <i>Current Opinion in Structural Biology</i>, vol. 50, Elsevier, 2018, pp. 65–74, doi:<a href=\"https://doi.org/10.1016/j.sbi.2017.12.002\">10.1016/j.sbi.2017.12.002</a>.","short":"R. Richter, N.S. Baranova, A. Day, J. Kwok, Current Opinion in Structural Biology 50 (2018) 65–74."},"date_published":"2018-06-01T00:00:00Z","publication_status":"published","month":"06","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","article_type":"original","fulldoi":"https://doi.org/10.1016/j.sbi.2017.12.002","publisher":"Elsevier","scopus_import":"1","publication":"Current Opinion in Structural Biology","_id":"555","oa":1},{"ddc":["500"],"author":[{"full_name":"Betea, Dan","last_name":"Betea","first_name":"Dan"},{"full_name":"Bouttier, Jeremie","first_name":"Jeremie","last_name":"Bouttier"},{"full_name":"Nejjar, Peter","last_name":"Nejjar","first_name":"Peter","id":"4BF426E2-F248-11E8-B48F-1D18A9856A87"},{"last_name":"Vuletic","first_name":"Mirjana","full_name":"Vuletic, Mirjana"}],"tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"date_created":"2018-12-11T11:47:09Z","issue":"12","type":"journal_article","file_date_updated":"2020-07-14T12:47:03Z","status":"public","doi":"10.1007/s00023-018-0723-1","date_updated":"2025-09-18T07:34:29Z","publist_id":"7258","title":"The free boundary Schur process and applications I","day":"13","oa_version":"Published Version","language":[{"iso":"eng"}],"intvolume":"        19","file":[{"file_size":3084674,"relation":"main_file","access_level":"open_access","file_name":"2018_Annales_Betea.pdf","date_updated":"2020-07-14T12:47:03Z","date_created":"2019-01-21T15:18:55Z","checksum":"0c38abe73569b7166b7487ad5d23cc68","content_type":"application/pdf","creator":"dernst","file_id":"5866"}],"abstract":[{"text":"We investigate the free boundary Schur process, a variant of the Schur process introduced by Okounkov and Reshetikhin, where we allow the first and the last partitions to be arbitrary (instead of empty in the original setting). The pfaffian Schur process, previously studied by several authors, is recovered when just one of the boundary partitions is left free. We compute the correlation functions of the process in all generality via the free fermion formalism, which we extend with the thorough treatment of “free boundary states.” For the case of one free boundary, our approach yields a new proof that the process is pfaffian. For the case of two free boundaries, we find that the process is not pfaffian, but a closely related process is. We also study three different applications of the Schur process with one free boundary: fluctuations of symmetrized last passage percolation models, limit shapes and processes for symmetric plane partitions and for plane overpartitions.","lang":"eng"}],"external_id":{"isi":["000450487900003"],"arxiv":["1704.05809"]},"year":"2018","page":"3663-3742","isi":1,"quality_controlled":"1","fulldoi":"https://doi.org/10.1007/s00023-018-0723-1","scopus_import":"1","publisher":"Springer Nature","publication":"Annales Henri Poincare","publication_identifier":{"issn":["1424-0637"]},"_id":"556","project":[{"_id":"258DCDE6-B435-11E9-9278-68D0E5697425","grant_number":"338804","name":"Random matrices, universality and disordered quantum systems","call_identifier":"FP7"},{"call_identifier":"H2020","grant_number":"716117","name":"Optimal Transport and Stochastic Dynamics","_id":"256E75B8-B435-11E9-9278-68D0E5697425"}],"arxiv":1,"oa":1,"has_accepted_license":"1","department":[{"_id":"LaEr"},{"_id":"JaMa"}],"article_processing_charge":"Yes (via OA deal)","volume":19,"citation":{"ieee":"D. Betea, J. Bouttier, P. Nejjar, and M. Vuletic, “The free boundary Schur process and applications I,” <i>Annales Henri Poincare</i>, vol. 19, no. 12. Springer Nature, pp. 3663–3742, 2018.","short":"D. Betea, J. Bouttier, P. Nejjar, M. Vuletic, Annales Henri Poincare 19 (2018) 3663–3742.","ista":"Betea D, Bouttier J, Nejjar P, Vuletic M. 2018. The free boundary Schur process and applications I. Annales Henri Poincare. 19(12), 3663–3742.","apa":"Betea, D., Bouttier, J., Nejjar, P., &#38; Vuletic, M. (2018). The free boundary Schur process and applications I. <i>Annales Henri Poincare</i>. Springer Nature. <a href=\"https://doi.org/10.1007/s00023-018-0723-1\">https://doi.org/10.1007/s00023-018-0723-1</a>","mla":"Betea, Dan, et al. “The Free Boundary Schur Process and Applications I.” <i>Annales Henri Poincare</i>, vol. 19, no. 12, Springer Nature, 2018, pp. 3663–742, doi:<a href=\"https://doi.org/10.1007/s00023-018-0723-1\">10.1007/s00023-018-0723-1</a>.","ama":"Betea D, Bouttier J, Nejjar P, Vuletic M. The free boundary Schur process and applications I. <i>Annales Henri Poincare</i>. 2018;19(12):3663-3742. doi:<a href=\"https://doi.org/10.1007/s00023-018-0723-1\">10.1007/s00023-018-0723-1</a>","chicago":"Betea, Dan, Jeremie Bouttier, Peter Nejjar, and Mirjana Vuletic. “The Free Boundary Schur Process and Applications I.” <i>Annales Henri Poincare</i>. 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