---
_id: '6952'
abstract:
- lang: eng
  text: 'We present a unified framework tackling two problems: class-specific 3D reconstruction
    from a single image, and generation of new 3D shape samples. These tasks have
    received considerable attention recently; however, most existing approaches rely
    on 3D supervision, annotation of 2D images with keypoints or poses, and/or training
    with multiple views of each object instance. Our framework is very general: it
    can be trained in similar settings to existing approaches, while also supporting
    weaker supervision. Importantly, it can be trained purely from 2D images, without
    pose annotations, and with only a single view per instance. We employ meshes as
    an output representation, instead of voxels used in most prior work. This allows
    us to reason over lighting parameters and exploit shading information during training,
    which previous 2D-supervised methods cannot. Thus, our method can learn to generate
    and reconstruct concave object classes. We evaluate our approach in various settings,
    showing that: (i) it learns to disentangle shape from pose and lighting; (ii)
    using shading in the loss improves performance compared to just silhouettes; (iii)
    when using a standard single white light, our model outperforms state-of-the-art
    2D-supervised methods, both with and without pose supervision, thanks to exploiting
    shading cues; (iv) performance improves further when using multiple coloured lights,
    even approaching that of state-of-the-art 3D-supervised methods; (v) shapes produced
    by our model capture smooth surfaces and fine details better than voxel-based
    approaches; and (vi) our approach supports concave classes such as bathtubs and
    sofas, which methods based on silhouettes cannot learn.'
acknowledgement: Open access funding provided by Institute of Science and Technology
  (IST Austria).
article_processing_charge: Yes (via OA deal)
article_type: original
arxiv: 1
author:
- first_name: Paul M
  full_name: Henderson, Paul M
  id: 13C09E74-18D9-11E9-8878-32CFE5697425
  last_name: Henderson
  orcid: 0000-0002-5198-7445
- first_name: Vittorio
  full_name: Ferrari, Vittorio
  last_name: Ferrari
citation:
  ama: Henderson PM, Ferrari V. Learning single-image 3D reconstruction by generative
    modelling of shape, pose and shading. <i>International Journal of Computer Vision</i>.
    2020;128:835-854. doi:<a href="https://doi.org/10.1007/s11263-019-01219-8">10.1007/s11263-019-01219-8</a>
  apa: Henderson, P. M., &#38; Ferrari, V. (2020). Learning single-image 3D reconstruction
    by generative modelling of shape, pose and shading. <i>International Journal of
    Computer Vision</i>. Springer Nature. <a href="https://doi.org/10.1007/s11263-019-01219-8">https://doi.org/10.1007/s11263-019-01219-8</a>
  chicago: Henderson, Paul M, and Vittorio Ferrari. “Learning Single-Image 3D Reconstruction
    by Generative Modelling of Shape, Pose and Shading.” <i>International Journal
    of Computer Vision</i>. Springer Nature, 2020. <a href="https://doi.org/10.1007/s11263-019-01219-8">https://doi.org/10.1007/s11263-019-01219-8</a>.
  ieee: P. M. Henderson and V. Ferrari, “Learning single-image 3D reconstruction by
    generative modelling of shape, pose and shading,” <i>International Journal of
    Computer Vision</i>, vol. 128. Springer Nature, pp. 835–854, 2020.
  ista: Henderson PM, Ferrari V. 2020. Learning single-image 3D reconstruction by
    generative modelling of shape, pose and shading. International Journal of Computer
    Vision. 128, 835–854.
  mla: Henderson, Paul M., and Vittorio Ferrari. “Learning Single-Image 3D Reconstruction
    by Generative Modelling of Shape, Pose and Shading.” <i>International Journal
    of Computer Vision</i>, vol. 128, Springer Nature, 2020, pp. 835–54, doi:<a href="https://doi.org/10.1007/s11263-019-01219-8">10.1007/s11263-019-01219-8</a>.
  short: P.M. Henderson, V. Ferrari, International Journal of Computer Vision 128
    (2020) 835–854.
corr_author: '1'
date_created: 2019-10-17T13:38:20Z
date_published: 2020-04-01T00:00:00Z
date_updated: 2025-04-15T06:53:15Z
day: '01'
ddc:
- '004'
department:
- _id: ChLa
doi: 10.1007/s11263-019-01219-8
external_id:
  arxiv:
  - '1901.06447'
  isi:
  - '000491042100002'
file:
- access_level: open_access
  checksum: a0f05dd4f5f64e4f713d8d9d4b5b1e3f
  content_type: application/pdf
  creator: dernst
  date_created: 2019-10-25T10:28:29Z
  date_updated: 2020-07-14T12:47:46Z
  file_id: '6973'
  file_name: 2019_CompVision_Henderson.pdf
  file_size: 2243134
  relation: main_file
file_date_updated: 2020-07-14T12:47:46Z
has_accepted_license: '1'
intvolume: '       128'
isi: 1
language:
- iso: eng
license: https://creativecommons.org/licenses/by/4.0/
month: '04'
oa: 1
oa_version: Published Version
page: 835-854
project:
- _id: B67AFEDC-15C9-11EA-A837-991A96BB2854
  name: IST Austria Open Access Fund
publication: International Journal of Computer Vision
publication_identifier:
  eissn:
  - 1573-1405
  issn:
  - 0920-5691
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Learning single-image 3D reconstruction by generative modelling of shape, pose
  and shading
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 128
year: '2020'
...
---
_id: '6976'
abstract:
- lang: eng
  text: Origami is rapidly transforming the design of robots1,2, deployable structures3,4,5,6
    and metamaterials7,8,9,10,11,12,13,14. However, as foldability requires a large
    number of complex compatibility conditions that are difficult to satisfy, the
    design of crease patterns is limited to heuristics and computer optimization.
    Here we introduce a systematic strategy that enables intuitive and effective design
    of complex crease patterns that are guaranteed to fold. First, we exploit symmetries
    to construct 140 distinct foldable motifs, and represent these as jigsaw puzzle
    pieces. We then show that when these pieces are fitted together they encode foldable
    crease patterns. This maps origami design to solving combinatorial problems, which
    allows us to systematically create, count and classify a vast number of crease
    patterns. We show that all of these crease patterns are pluripotent—capable of
    folding into multiple shapes—and solve exactly for the number of possible shapes
    for each pattern. Finally, we employ our framework to rationally design a crease
    pattern that folds into two independently defined target shapes, and fabricate
    such pluripotent origami. Our results provide physicists, mathematicians and engineers
    with a powerful new design strategy.
article_processing_charge: No
article_type: letter_note
author:
- first_name: Peter
  full_name: Dieleman, Peter
  last_name: Dieleman
- first_name: Niek
  full_name: Vasmel, Niek
  last_name: Vasmel
- first_name: Scott R
  full_name: Waitukaitis, Scott R
  id: 3A1FFC16-F248-11E8-B48F-1D18A9856A87
  last_name: Waitukaitis
  orcid: 0000-0002-2299-3176
- first_name: Martin
  full_name: van Hecke, Martin
  last_name: van Hecke
citation:
  ama: Dieleman P, Vasmel N, Waitukaitis SR, van Hecke M. Jigsaw puzzle design of
    pluripotent origami. <i>Nature Physics</i>. 2020;16(1):63–68. doi:<a href="https://doi.org/10.1038/s41567-019-0677-3">10.1038/s41567-019-0677-3</a>
  apa: Dieleman, P., Vasmel, N., Waitukaitis, S. R., &#38; van Hecke, M. (2020). Jigsaw
    puzzle design of pluripotent origami. <i>Nature Physics</i>. Springer Nature.
    <a href="https://doi.org/10.1038/s41567-019-0677-3">https://doi.org/10.1038/s41567-019-0677-3</a>
  chicago: Dieleman, Peter, Niek Vasmel, Scott R Waitukaitis, and Martin van Hecke.
    “Jigsaw Puzzle Design of Pluripotent Origami.” <i>Nature Physics</i>. Springer
    Nature, 2020. <a href="https://doi.org/10.1038/s41567-019-0677-3">https://doi.org/10.1038/s41567-019-0677-3</a>.
  ieee: P. Dieleman, N. Vasmel, S. R. Waitukaitis, and M. van Hecke, “Jigsaw puzzle
    design of pluripotent origami,” <i>Nature Physics</i>, vol. 16, no. 1. Springer
    Nature, pp. 63–68, 2020.
  ista: Dieleman P, Vasmel N, Waitukaitis SR, van Hecke M. 2020. Jigsaw puzzle design
    of pluripotent origami. Nature Physics. 16(1), 63–68.
  mla: Dieleman, Peter, et al. “Jigsaw Puzzle Design of Pluripotent Origami.” <i>Nature
    Physics</i>, vol. 16, no. 1, Springer Nature, 2020, pp. 63–68, doi:<a href="https://doi.org/10.1038/s41567-019-0677-3">10.1038/s41567-019-0677-3</a>.
  short: P. Dieleman, N. Vasmel, S.R. Waitukaitis, M. van Hecke, Nature Physics 16
    (2020) 63–68.
date_created: 2019-10-31T07:51:44Z
date_published: 2020-01-01T00:00:00Z
date_updated: 2021-01-12T08:11:16Z
day: '01'
doi: 10.1038/s41567-019-0677-3
extern: '1'
intvolume: '        16'
issue: '1'
language:
- iso: eng
month: '01'
oa_version: None
page: 63–68
publication: Nature Physics
publication_identifier:
  eissn:
  - 1745-2481
  issn:
  - 1745-2473
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
status: public
title: Jigsaw puzzle design of pluripotent origami
type: journal_article
user_id: D865714E-FA4E-11E9-B85B-F5C5E5697425
volume: 16
year: '2020'
...
---
_id: '6997'
article_processing_charge: Yes (via OA deal)
article_type: original
author:
- first_name: Yuzhou
  full_name: Zhang, Yuzhou
  id: 3B6137F2-F248-11E8-B48F-1D18A9856A87
  last_name: Zhang
  orcid: 0000-0003-2627-6956
- first_name: Jiří
  full_name: Friml, Jiří
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Zhang Y, Friml J. Auxin guides roots to avoid obstacles during gravitropic
    growth. <i>New Phytologist</i>. 2020;225(3):1049-1052. doi:<a href="https://doi.org/10.1111/nph.16203">10.1111/nph.16203</a>
  apa: Zhang, Y., &#38; Friml, J. (2020). Auxin guides roots to avoid obstacles during
    gravitropic growth. <i>New Phytologist</i>. Wiley. <a href="https://doi.org/10.1111/nph.16203">https://doi.org/10.1111/nph.16203</a>
  chicago: Zhang, Yuzhou, and Jiří Friml. “Auxin Guides Roots to Avoid Obstacles during
    Gravitropic Growth.” <i>New Phytologist</i>. Wiley, 2020. <a href="https://doi.org/10.1111/nph.16203">https://doi.org/10.1111/nph.16203</a>.
  ieee: Y. Zhang and J. Friml, “Auxin guides roots to avoid obstacles during gravitropic
    growth,” <i>New Phytologist</i>, vol. 225, no. 3. Wiley, pp. 1049–1052, 2020.
  ista: Zhang Y, Friml J. 2020. Auxin guides roots to avoid obstacles during gravitropic
    growth. New Phytologist. 225(3), 1049–1052.
  mla: Zhang, Yuzhou, and Jiří Friml. “Auxin Guides Roots to Avoid Obstacles during
    Gravitropic Growth.” <i>New Phytologist</i>, vol. 225, no. 3, Wiley, 2020, pp.
    1049–52, doi:<a href="https://doi.org/10.1111/nph.16203">10.1111/nph.16203</a>.
  short: Y. Zhang, J. Friml, New Phytologist 225 (2020) 1049–1052.
corr_author: '1'
date_created: 2019-11-12T11:41:32Z
date_published: 2020-02-01T00:00:00Z
date_updated: 2025-04-14T07:45:04Z
day: '01'
ddc:
- '580'
department:
- _id: JiFr
doi: 10.1111/nph.16203
ec_funded: 1
external_id:
  isi:
  - '000489638800001'
  pmid:
  - '31603260'
file:
- access_level: open_access
  checksum: cd42ffdb381fd52812b9583d4d407139
  content_type: application/pdf
  creator: dernst
  date_created: 2020-11-18T16:42:48Z
  date_updated: 2020-11-18T16:42:48Z
  file_id: '8772'
  file_name: 2020_NewPhytologist_Zhang.pdf
  file_size: 717345
  relation: main_file
  success: 1
file_date_updated: 2020-11-18T16:42:48Z
has_accepted_license: '1'
intvolume: '       225'
isi: 1
issue: '3'
language:
- iso: eng
month: '02'
oa: 1
oa_version: Published Version
page: 1049-1052
pmid: 1
project:
- _id: 261099A6-B435-11E9-9278-68D0E5697425
  call_identifier: H2020
  grant_number: '742985'
  name: Tracing Evolution of Auxin Transport and Polarity in Plants
- _id: 26538374-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: I03630
  name: Molecular mechanisms of endocytic cargo recognition in plants
- _id: 25681D80-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '291734'
  name: International IST Postdoc Fellowship Programme
publication: New Phytologist
publication_identifier:
  eissn:
  - 1469-8137
  issn:
  - 0028-646x
publication_status: published
publisher: Wiley
quality_controlled: '1'
scopus_import: '1'
status: public
title: Auxin guides roots to avoid obstacles during gravitropic growth
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 225
year: '2020'
...
---
_id: '7004'
abstract:
- lang: eng
  text: We define an action of the (double of) Cohomological Hall algebra of Kontsevich
    and Soibelman on the cohomology of the moduli space of spiked instantons of Nekrasov.
    We identify this action with the one of the affine Yangian of gl(1). Based on
    that we derive the vertex algebra at the corner Wr1,r2,r3 of Gaiotto and Rapčák.
    We conjecture that our approach works for a big class of Calabi–Yau categories,
    including those associated with toric Calabi–Yau 3-folds.
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Miroslav
  full_name: Rapcak, Miroslav
  last_name: Rapcak
- first_name: Yan
  full_name: Soibelman, Yan
  last_name: Soibelman
- first_name: Yaping
  full_name: Yang, Yaping
  last_name: Yang
- first_name: Gufang
  full_name: Zhao, Gufang
  id: 2BC2AC5E-F248-11E8-B48F-1D18A9856A87
  last_name: Zhao
citation:
  ama: Rapcak M, Soibelman Y, Yang Y, Zhao G. Cohomological Hall algebras, vertex
    algebras and instantons. <i>Communications in Mathematical Physics</i>. 2020;376:1803-1873.
    doi:<a href="https://doi.org/10.1007/s00220-019-03575-5">10.1007/s00220-019-03575-5</a>
  apa: Rapcak, M., Soibelman, Y., Yang, Y., &#38; Zhao, G. (2020). Cohomological Hall
    algebras, vertex algebras and instantons. <i>Communications in Mathematical Physics</i>.
    Springer Nature. <a href="https://doi.org/10.1007/s00220-019-03575-5">https://doi.org/10.1007/s00220-019-03575-5</a>
  chicago: Rapcak, Miroslav, Yan Soibelman, Yaping Yang, and Gufang Zhao. “Cohomological
    Hall Algebras, Vertex Algebras and Instantons.” <i>Communications in Mathematical
    Physics</i>. Springer Nature, 2020. <a href="https://doi.org/10.1007/s00220-019-03575-5">https://doi.org/10.1007/s00220-019-03575-5</a>.
  ieee: M. Rapcak, Y. Soibelman, Y. Yang, and G. Zhao, “Cohomological Hall algebras,
    vertex algebras and instantons,” <i>Communications in Mathematical Physics</i>,
    vol. 376. Springer Nature, pp. 1803–1873, 2020.
  ista: Rapcak M, Soibelman Y, Yang Y, Zhao G. 2020. Cohomological Hall algebras,
    vertex algebras and instantons. Communications in Mathematical Physics. 376, 1803–1873.
  mla: Rapcak, Miroslav, et al. “Cohomological Hall Algebras, Vertex Algebras and
    Instantons.” <i>Communications in Mathematical Physics</i>, vol. 376, Springer
    Nature, 2020, pp. 1803–73, doi:<a href="https://doi.org/10.1007/s00220-019-03575-5">10.1007/s00220-019-03575-5</a>.
  short: M. Rapcak, Y. Soibelman, Y. Yang, G. Zhao, Communications in Mathematical
    Physics 376 (2020) 1803–1873.
date_created: 2019-11-12T14:01:27Z
date_published: 2020-06-01T00:00:00Z
date_updated: 2025-04-14T09:12:46Z
day: '01'
department:
- _id: TaHa
doi: 10.1007/s00220-019-03575-5
ec_funded: 1
external_id:
  arxiv:
  - '1810.10402'
  isi:
  - '000536255500004'
intvolume: '       376'
isi: 1
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1810.10402
month: '06'
oa: 1
oa_version: Preprint
page: 1803-1873
project:
- _id: 25E549F4-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '320593'
  name: Arithmetic and physics of Higgs moduli spaces
publication: Communications in Mathematical Physics
publication_identifier:
  eissn:
  - 1432-0916
  issn:
  - 0010-3616
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: Cohomological Hall algebras, vertex algebras and instantons
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 376
year: '2020'
...
---
_id: '7033'
abstract:
- lang: eng
  text: Removal of the Bax gene from mice completely protects the somas of retinal
    ganglion cells (RGCs) from apoptosis following optic nerve injury. This makes
    BAX a promising therapeutic target to prevent neurodegeneration. In this study,
    Bax+/− mice were used to test the hypothesis that lowering the quantity of BAX
    in RGCs would delay apoptosis following optic nerve injury. RGCs were damaged
    by performing optic nerve crush (ONC) and then immunostaining for phospho-cJUN,
    and quantitative PCR were used to monitor the status of the BAX activation mechanism
    in the months following injury. The apoptotic susceptibility of injured cells
    was directly tested by virally introducing GFP-BAX into Bax−/− RGCs after injury.
    The competency of quiescent RGCs to reactivate their BAX activation mechanism
    was tested by intravitreal injection of the JNK pathway agonist, anisomycin. Twenty-four
    weeks after ONC, Bax+/− mice had significantly less cell loss in their RGC layer
    than Bax+/+ mice 3 weeks after ONC. Bax+/− and Bax+/+ RGCs exhibited similar patterns
    of nuclear phospho-cJUN accumulation immediately after ONC, which persisted in
    Bax+/− RGCs for up to 7 weeks before abating. The transcriptional activation of
    BAX-activating genes was similar in Bax+/− and Bax+/+ RGCs following ONC. Intriguingly,
    cells deactivated their BAX activation mechanism between 7 and 12 weeks after
    crush. Introduction of GFP-BAX into Bax−/− cells at 4 weeks after ONC showed that
    these cells had a nearly normal capacity to activate this protein, but this capacity
    was lost 8 weeks after crush. Collectively, these data suggest that 8–12 weeks
    after crush, damaged cells no longer displayed increased susceptibility to BAX
    activation relative to their naïve counterparts. In this same timeframe, retinal
    glial activation and the signaling of the pro-apoptotic JNK pathway also abated.
    Quiescent RGCs did not show a timely reactivation of their JNK pathway following
    intravitreal injection with anisomycin. These findings demonstrate that lowering
    the quantity of BAX in RGCs is neuroprotective after acute injury. Damaged RGCs
    enter a quiescent state months after injury and are no longer responsive to an
    apoptotic stimulus. Quiescent RGCs will require rejuvenation to reacquire functionality.
acknowledgement: This work was supported by National Eye Institute grants R01 EY012223
  (RWN), R01 EY030123 (RWN), T32 EY027721 (Department of Ophthalmology and Visual
  Sciences, University of Wisconsin-Madison), and a Vision Science Core grant P30
  EY016665 (Department of Ophthalmology and Visual Sciences, University of Wisconsin-Madison),
  an unrestricted funding grant from Research to Prevent Blindness (Department of
  Ophthalmology and Visual Sciences, University of Wisconsin-Madison), the Frederick
  A. Davis Endowment (RWN), and the Mr. and Mrs. George Taylor Foundation (RWN).
article_processing_charge: No
article_type: original
author:
- first_name: RJ
  full_name: Donahue, RJ
  last_name: Donahue
- first_name: Margaret E
  full_name: Maes, Margaret E
  id: 3838F452-F248-11E8-B48F-1D18A9856A87
  last_name: Maes
  orcid: 0000-0001-9642-1085
- first_name: JA
  full_name: Grosser, JA
  last_name: Grosser
- first_name: RW
  full_name: Nickells, RW
  last_name: Nickells
citation:
  ama: Donahue R, Maes ME, Grosser J, Nickells R. BAX-depleted retinal ganglion cells
    survive and become quiescent following optic nerve damage. <i>Molecular Neurobiology</i>.
    2020;57(2):1070–1084. doi:<a href="https://doi.org/10.1007/s12035-019-01783-7">10.1007/s12035-019-01783-7</a>
  apa: Donahue, R., Maes, M. E., Grosser, J., &#38; Nickells, R. (2020). BAX-depleted
    retinal ganglion cells survive and become quiescent following optic nerve damage.
    <i>Molecular Neurobiology</i>. Springer Nature. <a href="https://doi.org/10.1007/s12035-019-01783-7">https://doi.org/10.1007/s12035-019-01783-7</a>
  chicago: Donahue, RJ, Margaret E Maes, JA Grosser, and RW Nickells. “BAX-Depleted
    Retinal Ganglion Cells Survive and Become Quiescent Following Optic Nerve Damage.”
    <i>Molecular Neurobiology</i>. Springer Nature, 2020. <a href="https://doi.org/10.1007/s12035-019-01783-7">https://doi.org/10.1007/s12035-019-01783-7</a>.
  ieee: R. Donahue, M. E. Maes, J. Grosser, and R. Nickells, “BAX-depleted retinal
    ganglion cells survive and become quiescent following optic nerve damage,” <i>Molecular
    Neurobiology</i>, vol. 57, no. 2. Springer Nature, pp. 1070–1084, 2020.
  ista: Donahue R, Maes ME, Grosser J, Nickells R. 2020. BAX-depleted retinal ganglion
    cells survive and become quiescent following optic nerve damage. Molecular Neurobiology.
    57(2), 1070–1084.
  mla: Donahue, RJ, et al. “BAX-Depleted Retinal Ganglion Cells Survive and Become
    Quiescent Following Optic Nerve Damage.” <i>Molecular Neurobiology</i>, vol. 57,
    no. 2, Springer Nature, 2020, pp. 1070–1084, doi:<a href="https://doi.org/10.1007/s12035-019-01783-7">10.1007/s12035-019-01783-7</a>.
  short: R. Donahue, M.E. Maes, J. Grosser, R. Nickells, Molecular Neurobiology 57
    (2020) 1070–1084.
date_created: 2019-11-18T14:18:39Z
date_published: 2020-02-01T00:00:00Z
date_updated: 2023-08-17T14:05:48Z
day: '01'
department:
- _id: SaSi
doi: 10.1007/s12035-019-01783-7
external_id:
  isi:
  - '000493754200001'
  pmid:
  - '31673950'
intvolume: '        57'
isi: 1
issue: '2'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7035206/
month: '02'
oa: 1
oa_version: Submitted Version
page: 1070–1084
pmid: 1
publication: Molecular Neurobiology
publication_identifier:
  eissn:
  - 1559-1182
  issn:
  - 0893-7648
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: BAX-depleted retinal ganglion cells survive and become quiescent following
  optic nerve damage
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 57
year: '2020'
...
---
_id: '7084'
abstract:
- lang: eng
  text: The unusual correlated state that emerges in URu2Si2 below THO = 17.5 K is
    known as “hidden order” because even basic characteristics of the order parameter,
    such as its dimensionality (whether it has one component or two), are “hidden.”
    We use resonant ultrasound spectroscopy to measure the symmetry-resolved elastic
    anomalies across THO. We observe no anomalies in the shear elastic moduli, providing
    strong thermodynamic evidence for a one-component order parameter. We develop
    a machine learning framework that reaches this conclusion directly from the raw
    data, even in a crystal that is too small for traditional resonant ultrasound.
    Our result rules out a broad class of theories of hidden order based on two-component
    order parameters, and constrains the nature of the fluctuations from which unconventional
    superconductivity emerges at lower temperature. Our machine learning framework
    is a powerful new tool for classifying the ubiquitous competing orders in correlated
    electron systems.
article_number: eaaz4074
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Sayak
  full_name: Ghosh, Sayak
  last_name: Ghosh
- first_name: Michael
  full_name: Matty, Michael
  last_name: Matty
- first_name: Ryan
  full_name: Baumbach, Ryan
  last_name: Baumbach
- first_name: Eric D.
  full_name: Bauer, Eric D.
  last_name: Bauer
- first_name: Kimberly A
  full_name: Modic, Kimberly A
  id: 13C26AC0-EB69-11E9-87C6-5F3BE6697425
  last_name: Modic
  orcid: 0000-0001-9760-3147
- first_name: Arkady
  full_name: Shekhter, Arkady
  last_name: Shekhter
- first_name: J. A.
  full_name: Mydosh, J. A.
  last_name: Mydosh
- first_name: Eun-Ah
  full_name: Kim, Eun-Ah
  last_name: Kim
- first_name: B. J.
  full_name: Ramshaw, B. J.
  last_name: Ramshaw
citation:
  ama: Ghosh S, Matty M, Baumbach R, et al. One-component order parameter in URu2Si2
    uncovered by resonant  ultrasound spectroscopy and machine learning. <i>Science
    Advances</i>. 2020;6(10). doi:<a href="https://doi.org/10.1126/sciadv.aaz4074">10.1126/sciadv.aaz4074</a>
  apa: Ghosh, S., Matty, M., Baumbach, R., Bauer, E. D., Modic, K. A., Shekhter, A.,
    … Ramshaw, B. J. (2020). One-component order parameter in URu2Si2 uncovered by
    resonant  ultrasound spectroscopy and machine learning. <i>Science Advances</i>.
    American Association for the Advancement of Science. <a href="https://doi.org/10.1126/sciadv.aaz4074">https://doi.org/10.1126/sciadv.aaz4074</a>
  chicago: Ghosh, Sayak, Michael Matty, Ryan Baumbach, Eric D. Bauer, Kimberly A Modic,
    Arkady Shekhter, J. A. Mydosh, Eun-Ah Kim, and B. J. Ramshaw. “One-Component Order
    Parameter in URu2Si2 Uncovered by Resonant  Ultrasound Spectroscopy and Machine
    Learning.” <i>Science Advances</i>. American Association for the Advancement of
    Science, 2020. <a href="https://doi.org/10.1126/sciadv.aaz4074">https://doi.org/10.1126/sciadv.aaz4074</a>.
  ieee: S. Ghosh <i>et al.</i>, “One-component order parameter in URu2Si2 uncovered
    by resonant  ultrasound spectroscopy and machine learning,” <i>Science Advances</i>,
    vol. 6, no. 10. American Association for the Advancement of Science, 2020.
  ista: Ghosh S, Matty M, Baumbach R, Bauer ED, Modic KA, Shekhter A, Mydosh JA, Kim
    E-A, Ramshaw BJ. 2020. One-component order parameter in URu2Si2 uncovered by resonant 
    ultrasound spectroscopy and machine learning. Science Advances. 6(10), eaaz4074.
  mla: Ghosh, Sayak, et al. “One-Component Order Parameter in URu2Si2 Uncovered by
    Resonant  Ultrasound Spectroscopy and Machine Learning.” <i>Science Advances</i>,
    vol. 6, no. 10, eaaz4074, American Association for the Advancement of Science,
    2020, doi:<a href="https://doi.org/10.1126/sciadv.aaz4074">10.1126/sciadv.aaz4074</a>.
  short: S. Ghosh, M. Matty, R. Baumbach, E.D. Bauer, K.A. Modic, A. Shekhter, J.A.
    Mydosh, E.-A. Kim, B.J. Ramshaw, Science Advances 6 (2020).
date_created: 2019-11-19T14:01:10Z
date_published: 2020-03-06T00:00:00Z
date_updated: 2022-08-25T15:08:41Z
day: '06'
doi: 10.1126/sciadv.aaz4074
extern: '1'
external_id:
  arxiv:
  - '1903.00552'
  pmid:
  - '32181367'
intvolume: '         6'
issue: '10'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1903.00552
month: '03'
oa: 1
oa_version: Preprint
pmid: 1
publication: Science Advances
publication_status: published
publisher: American Association for the Advancement of Science
quality_controlled: '1'
status: public
title: One-component order parameter in URu2Si2 uncovered by resonant  ultrasound
  spectroscopy and machine learning
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 6
year: '2020'
...
---
_id: '71'
abstract:
- lang: eng
  text: "We consider dynamical transport metrics for probability measures on discretisations
    of a bounded convex domain in ℝd. These metrics are natural discrete counterparts
    to the Kantorovich metric \U0001D54E2, defined using a Benamou-Brenier type formula.
    Under mild assumptions we prove an asymptotic upper bound for the discrete transport
    metric Wt in terms of \U0001D54E2, as the size of the mesh T tends to 0. However,
    we show that the corresponding lower bound may fail in general, even on certain
    one-dimensional and symmetric two-dimensional meshes. In addition, we show that
    the asymptotic lower bound holds under an isotropy assumption on the mesh, which
    turns out to be essentially necessary. This assumption is satisfied, e.g., for
    tilings by convex regular polygons, and it implies Gromov-Hausdorff convergence
    of the transport metric."
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Peter
  full_name: Gladbach, Peter
  last_name: Gladbach
- first_name: Eva
  full_name: Kopfer, Eva
  last_name: Kopfer
- first_name: Jan
  full_name: Maas, Jan
  id: 4C5696CE-F248-11E8-B48F-1D18A9856A87
  last_name: Maas
  orcid: 0000-0002-0845-1338
citation:
  ama: Gladbach P, Kopfer E, Maas J. Scaling limits of discrete optimal transport.
    <i>SIAM Journal on Mathematical Analysis</i>. 2020;52(3):2759-2802. doi:<a href="https://doi.org/10.1137/19M1243440">10.1137/19M1243440</a>
  apa: Gladbach, P., Kopfer, E., &#38; Maas, J. (2020). Scaling limits of discrete
    optimal transport. <i>SIAM Journal on Mathematical Analysis</i>. Society for Industrial
    and Applied Mathematics. <a href="https://doi.org/10.1137/19M1243440">https://doi.org/10.1137/19M1243440</a>
  chicago: Gladbach, Peter, Eva Kopfer, and Jan Maas. “Scaling Limits of Discrete
    Optimal Transport.” <i>SIAM Journal on Mathematical Analysis</i>. Society for
    Industrial and Applied Mathematics, 2020. <a href="https://doi.org/10.1137/19M1243440">https://doi.org/10.1137/19M1243440</a>.
  ieee: P. Gladbach, E. Kopfer, and J. Maas, “Scaling limits of discrete optimal transport,”
    <i>SIAM Journal on Mathematical Analysis</i>, vol. 52, no. 3. Society for Industrial
    and Applied Mathematics, pp. 2759–2802, 2020.
  ista: Gladbach P, Kopfer E, Maas J. 2020. Scaling limits of discrete optimal transport.
    SIAM Journal on Mathematical Analysis. 52(3), 2759–2802.
  mla: Gladbach, Peter, et al. “Scaling Limits of Discrete Optimal Transport.” <i>SIAM
    Journal on Mathematical Analysis</i>, vol. 52, no. 3, Society for Industrial and
    Applied Mathematics, 2020, pp. 2759–802, doi:<a href="https://doi.org/10.1137/19M1243440">10.1137/19M1243440</a>.
  short: P. Gladbach, E. Kopfer, J. Maas, SIAM Journal on Mathematical Analysis 52
    (2020) 2759–2802.
date_created: 2018-12-11T11:44:28Z
date_published: 2020-10-01T00:00:00Z
date_updated: 2025-07-10T11:54:14Z
day: '01'
department:
- _id: JaMa
doi: 10.1137/19M1243440
external_id:
  arxiv:
  - '1809.01092'
  isi:
  - '000546975100017'
intvolume: '        52'
isi: 1
issue: '3'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1809.01092
month: '10'
oa: 1
oa_version: Preprint
page: 2759-2802
publication: SIAM Journal on Mathematical Analysis
publication_identifier:
  eissn:
  - 1095-7154
  issn:
  - 0036-1410
publication_status: published
publisher: Society for Industrial and Applied Mathematics
publist_id: '7983'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Scaling limits of discrete optimal transport
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 52
year: '2020'
...
---
_id: '7142'
abstract:
- lang: eng
  text: The phytohormone auxin acts as an amazingly versatile coordinator of plant
    growth and development. With its morphogen-like properties, auxin controls sites
    and timing of differentiation and/or growth responses both, in quantitative and
    qualitative terms. Specificity in the auxin response depends largely on distinct
    modes of signal transmission, by which individual cells perceive and convert auxin
    signals into a remarkable diversity of responses. The best understood, or so-called
    canonical mechanism of auxin perception ultimately results in variable adjustments
    of the cellular transcriptome, via a short, nuclear signal transduction pathway.
    Additional findings that accumulated over decades implied that an additional,
    presumably, cell surface-based auxin perception mechanism mediates very rapid
    cellular responses and decisively contributes to the cell's overall hormonal response.
    Recent investigations into both, nuclear and cell surface auxin signalling challenged
    this assumed partition of roles for different auxin signalling pathways and revealed
    an unexpected complexity in transcriptional and non-transcriptional cellular responses
    mediated by auxin.
acknowledgement: Research in J.F. laboratory is funded by the European Union's Horizon
  2020 program (ERC grant agreement n° 742985); C.L. is supported by the Austrian
  Science Fund (FWF grant P 31493).
article_processing_charge: No
article_type: original
author:
- first_name: Michelle C
  full_name: Gallei, Michelle C
  id: 35A03822-F248-11E8-B48F-1D18A9856A87
  last_name: Gallei
  orcid: 0000-0003-1286-7368
- first_name: Christian
  full_name: Luschnig, Christian
  last_name: Luschnig
- first_name: Jiří
  full_name: Friml, Jiří
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: 'Gallei MC, Luschnig C, Friml J. Auxin signalling in growth: Schrödinger’s
    cat out of the bag. <i>Current Opinion in Plant Biology</i>. 2020;53(2):43-49.
    doi:<a href="https://doi.org/10.1016/j.pbi.2019.10.003">10.1016/j.pbi.2019.10.003</a>'
  apa: 'Gallei, M. C., Luschnig, C., &#38; Friml, J. (2020). Auxin signalling in growth:
    Schrödinger’s cat out of the bag. <i>Current Opinion in Plant Biology</i>. Elsevier.
    <a href="https://doi.org/10.1016/j.pbi.2019.10.003">https://doi.org/10.1016/j.pbi.2019.10.003</a>'
  chicago: 'Gallei, Michelle C, Christian Luschnig, and Jiří Friml. “Auxin Signalling
    in Growth: Schrödinger’s Cat out of the Bag.” <i>Current Opinion in Plant Biology</i>.
    Elsevier, 2020. <a href="https://doi.org/10.1016/j.pbi.2019.10.003">https://doi.org/10.1016/j.pbi.2019.10.003</a>.'
  ieee: 'M. C. Gallei, C. Luschnig, and J. Friml, “Auxin signalling in growth: Schrödinger’s
    cat out of the bag,” <i>Current Opinion in Plant Biology</i>, vol. 53, no. 2.
    Elsevier, pp. 43–49, 2020.'
  ista: 'Gallei MC, Luschnig C, Friml J. 2020. Auxin signalling in growth: Schrödinger’s
    cat out of the bag. Current Opinion in Plant Biology. 53(2), 43–49.'
  mla: 'Gallei, Michelle C., et al. “Auxin Signalling in Growth: Schrödinger’s Cat
    out of the Bag.” <i>Current Opinion in Plant Biology</i>, vol. 53, no. 2, Elsevier,
    2020, pp. 43–49, doi:<a href="https://doi.org/10.1016/j.pbi.2019.10.003">10.1016/j.pbi.2019.10.003</a>.'
  short: M.C. Gallei, C. Luschnig, J. Friml, Current Opinion in Plant Biology 53 (2020)
    43–49.
corr_author: '1'
date_created: 2019-12-02T12:05:26Z
date_published: 2020-02-01T00:00:00Z
date_updated: 2025-04-15T08:39:42Z
day: '01'
department:
- _id: JiFr
doi: 10.1016/j.pbi.2019.10.003
ec_funded: 1
external_id:
  isi:
  - '000521120600007'
  pmid:
  - '31760231'
intvolume: '        53'
isi: 1
issue: '2'
language:
- iso: eng
month: '02'
oa_version: None
page: 43-49
pmid: 1
project:
- _id: 261099A6-B435-11E9-9278-68D0E5697425
  call_identifier: H2020
  grant_number: '742985'
  name: Tracing Evolution of Auxin Transport and Polarity in Plants
publication: Current Opinion in Plant Biology
publication_identifier:
  eissn:
  - 1879-0356
  issn:
  - 1369-5266
publication_status: published
publisher: Elsevier
quality_controlled: '1'
related_material:
  record:
  - id: '11626'
    relation: dissertation_contains
    status: public
scopus_import: '1'
status: public
title: 'Auxin signalling in growth: Schrödinger''s cat out of the bag'
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 53
year: '2020'
...
---
_id: '7148'
abstract:
- lang: eng
  text: In the cerebellum, GluD2 is exclusively expressed in Purkinje cells, where
    it regulates synapse formation and regeneration, synaptic plasticity, and motor
    learning. Delayed cognitive development in humans with GluD2 gene mutations suggests
    extracerebellar functions of GluD2. However, extracerebellar expression of GluD2
    and its relationship with that of GluD1 are poorly understood. GluD2 mRNA and
    protein were widely detected, with relatively high levels observed in the olfactory
    glomerular layer, medial prefrontal cortex, cingulate cortex, retrosplenial granular
    cortex, olfactory tubercle, subiculum, striatum, lateral septum, anterodorsal
    thalamic nucleus, and arcuate hypothalamic nucleus. These regions were also enriched
    for GluD1, and many individual neurons coexpressed the two GluDs. In the retrosplenial
    granular cortex, GluD1 and GluD2 were selectively expressed at PSD‐95‐expressing
    glutamatergic synapses, and their coexpression on the same synapses was shown
    by SDS‐digested freeze‐fracture replica labeling. Biochemically, GluD1 and GluD2
    formed coimmunoprecipitable complex formation in HEK293T cells and in the cerebral
    cortex and hippocampus. We further estimated the relative protein amount by quantitative
    immunoblotting using GluA2/GluD2 and GluA2/GluD1 chimeric proteins as standards
    for titration of GluD1 and GluD2 antibodies. Intriguingly, the relative amount
    of GluD2 was almost comparable to that of GluD1 in the postsynaptic density fraction
    prepared from the cerebral cortex and hippocampus. In contrast, GluD2 was overwhelmingly
    predominant in the cerebellum. Thus, we have determined the relative extracerebellar
    expression of GluD1 and GluD2 at regional, neuronal, and synaptic levels. These
    data provide a molecular–anatomical basis for possible competitive and cooperative
    interactions of GluD family members at synapses in various brain regions.
acknowledgement: This study was supported by Grants-in-Aid for Scientific Research
  to K.K. (18K06813), Y.M. (17K08503, 17H0631319), and K.S. (16H04650) and a grant
  for Scientific Research on Innovative Areas to K.S (16H06276) from the Ministry
  of Education, Culture, Sports, Science and Technology of Japan (MEXT). We thank
  K. Akashi, I. Watanabe-Iida, Y. Suzuki, and H. Azechi for technical assistance and
  advice, and H. Uchida for valuable discussions. We thank E. Kushiya,I. Yabe, C.
  Ohori, Y. Mochizuki, Y. Ishikawa, and N. Ishimoto for technical assistance in generating
  GluD1-KO mice.
article_processing_charge: No
article_type: original
author:
- first_name: Chihiro
  full_name: Nakamoto, Chihiro
  last_name: Nakamoto
- first_name: Kohtarou
  full_name: Konno, Kohtarou
  last_name: Konno
- first_name: Taisuke
  full_name: Miyazaki, Taisuke
  last_name: Miyazaki
- first_name: Ena
  full_name: Nakatsukasa, Ena
  last_name: Nakatsukasa
- first_name: Rie
  full_name: Natsume, Rie
  last_name: Natsume
- first_name: Manabu
  full_name: Abe, Manabu
  last_name: Abe
- first_name: Meiko
  full_name: Kawamura, Meiko
  last_name: Kawamura
- first_name: Yugo
  full_name: Fukazawa, Yugo
  last_name: Fukazawa
- first_name: Ryuichi
  full_name: Shigemoto, Ryuichi
  id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
  last_name: Shigemoto
  orcid: 0000-0001-8761-9444
- first_name: Miwako
  full_name: Yamasaki, Miwako
  last_name: Yamasaki
- first_name: Kenji
  full_name: Sakimura, Kenji
  last_name: Sakimura
- first_name: Masahiko
  full_name: Watanabe, Masahiko
  last_name: Watanabe
citation:
  ama: Nakamoto C, Konno K, Miyazaki T, et al. Expression mapping, quantification,
    and complex formation of GluD1 and GluD2 glutamate receptors in adult mouse brain.
    <i>Journal of Comparative Neurology</i>. 2020;528(6):1003-1027. doi:<a href="https://doi.org/10.1002/cne.24792">10.1002/cne.24792</a>
  apa: Nakamoto, C., Konno, K., Miyazaki, T., Nakatsukasa, E., Natsume, R., Abe, M.,
    … Watanabe, M. (2020). Expression mapping, quantification, and complex formation
    of GluD1 and GluD2 glutamate receptors in adult mouse brain. <i>Journal of Comparative
    Neurology</i>. Wiley. <a href="https://doi.org/10.1002/cne.24792">https://doi.org/10.1002/cne.24792</a>
  chicago: Nakamoto, Chihiro, Kohtarou Konno, Taisuke Miyazaki, Ena Nakatsukasa, Rie
    Natsume, Manabu Abe, Meiko Kawamura, et al. “Expression Mapping, Quantification,
    and Complex Formation of GluD1 and GluD2 Glutamate Receptors in Adult Mouse Brain.”
    <i>Journal of Comparative Neurology</i>. Wiley, 2020. <a href="https://doi.org/10.1002/cne.24792">https://doi.org/10.1002/cne.24792</a>.
  ieee: C. Nakamoto <i>et al.</i>, “Expression mapping, quantification, and complex
    formation of GluD1 and GluD2 glutamate receptors in adult mouse brain,” <i>Journal
    of Comparative Neurology</i>, vol. 528, no. 6. Wiley, pp. 1003–1027, 2020.
  ista: Nakamoto C, Konno K, Miyazaki T, Nakatsukasa E, Natsume R, Abe M, Kawamura
    M, Fukazawa Y, Shigemoto R, Yamasaki M, Sakimura K, Watanabe M. 2020. Expression
    mapping, quantification, and complex formation of GluD1 and GluD2 glutamate receptors
    in adult mouse brain. Journal of Comparative Neurology. 528(6), 1003–1027.
  mla: Nakamoto, Chihiro, et al. “Expression Mapping, Quantification, and Complex
    Formation of GluD1 and GluD2 Glutamate Receptors in Adult Mouse Brain.” <i>Journal
    of Comparative Neurology</i>, vol. 528, no. 6, Wiley, 2020, pp. 1003–27, doi:<a
    href="https://doi.org/10.1002/cne.24792">10.1002/cne.24792</a>.
  short: C. Nakamoto, K. Konno, T. Miyazaki, E. Nakatsukasa, R. Natsume, M. Abe, M.
    Kawamura, Y. Fukazawa, R. Shigemoto, M. Yamasaki, K. Sakimura, M. Watanabe, Journal
    of Comparative Neurology 528 (2020) 1003–1027.
date_created: 2019-12-04T16:09:29Z
date_published: 2020-04-01T00:00:00Z
date_updated: 2023-08-17T14:06:50Z
day: '01'
ddc:
- '571'
- '599'
department:
- _id: RySh
doi: 10.1002/cne.24792
external_id:
  isi:
  - '000496410200001'
  pmid:
  - '31625608'
has_accepted_license: '1'
intvolume: '       528'
isi: 1
issue: '6'
language:
- iso: eng
month: '04'
oa_version: None
page: 1003-1027
pmid: 1
publication: Journal of Comparative Neurology
publication_identifier:
  eissn:
  - 1096-9861
  issn:
  - 0021-9967
publication_status: published
publisher: Wiley
quality_controlled: '1'
scopus_import: '1'
status: public
title: Expression mapping, quantification, and complex formation of GluD1 and GluD2
  glutamate receptors in adult mouse brain
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 528
year: '2020'
...
---
_id: '7149'
abstract:
- lang: eng
  text: In recent years, many genes have been associated with chromatinopathies classified
    as “Cornelia de Lange Syndrome‐like.” It is known that the phenotype of these
    patients becomes less recognizable, overlapping to features characteristic of
    other syndromes caused by genetic variants affecting different regulators of chromatin
    structure and function. Therefore, Cornelia de Lange syndrome diagnosis might
    be arduous due to the seldom discordance between unexpected molecular diagnosis
    and clinical evaluation. Here, we review the molecular features of Cornelia de
    Lange syndrome, supporting the hypothesis that “CdLS‐like syndromes” are part
    of a larger “rare disease family” sharing multiple clinical features and common
    disrupted molecular pathways.
acknowledgement: ' Dipartimento DiSS, Università degli Studi di Milano, Grant/Award
  Number: Linea 2; Fondazione Cariplo, Grant/Award Number: 2015-0783; German Federal
  Ministry of Education and Research (BMBF), Grant/Award Number: CHROMATIN-Net; Medical
  Faculty of the University of Lübeck, Grant/Award Number: J09-2017; Nickel & Co S.p.A.;
  Università degli Studi di Milano, Grant/Award Numbers: Molecular & Translational
  Medicine PhD Scholarship, Translational Medicine PhD Scholarship'
article_processing_charge: No
article_type: review
author:
- first_name: Laura
  full_name: Avagliano, Laura
  last_name: Avagliano
- first_name: Ilaria
  full_name: Parenti, Ilaria
  id: D93538B0-5B71-11E9-AC62-02EBE5697425
  last_name: Parenti
- first_name: Paolo
  full_name: Grazioli, Paolo
  last_name: Grazioli
- first_name: Elisabetta
  full_name: Di Fede, Elisabetta
  last_name: Di Fede
- first_name: Chiara
  full_name: Parodi, Chiara
  last_name: Parodi
- first_name: Milena
  full_name: Mariani, Milena
  last_name: Mariani
- first_name: Frank J.
  full_name: Kaiser, Frank J.
  last_name: Kaiser
- first_name: Angelo
  full_name: Selicorni, Angelo
  last_name: Selicorni
- first_name: Cristina
  full_name: Gervasini, Cristina
  last_name: Gervasini
- first_name: Valentina
  full_name: Massa, Valentina
  last_name: Massa
citation:
  ama: 'Avagliano L, Parenti I, Grazioli P, et al. Chromatinopathies: A focus on Cornelia
    de Lange syndrome. <i>Clinical Genetics</i>. 2020;97(1):3-11. doi:<a href="https://doi.org/10.1111/cge.13674">10.1111/cge.13674</a>'
  apa: 'Avagliano, L., Parenti, I., Grazioli, P., Di Fede, E., Parodi, C., Mariani,
    M., … Massa, V. (2020). Chromatinopathies: A focus on Cornelia de Lange syndrome.
    <i>Clinical Genetics</i>. Wiley. <a href="https://doi.org/10.1111/cge.13674">https://doi.org/10.1111/cge.13674</a>'
  chicago: 'Avagliano, Laura, Ilaria Parenti, Paolo Grazioli, Elisabetta Di Fede,
    Chiara Parodi, Milena Mariani, Frank J. Kaiser, Angelo Selicorni, Cristina Gervasini,
    and Valentina Massa. “Chromatinopathies: A Focus on Cornelia de Lange Syndrome.”
    <i>Clinical Genetics</i>. Wiley, 2020. <a href="https://doi.org/10.1111/cge.13674">https://doi.org/10.1111/cge.13674</a>.'
  ieee: 'L. Avagliano <i>et al.</i>, “Chromatinopathies: A focus on Cornelia de Lange
    syndrome,” <i>Clinical Genetics</i>, vol. 97, no. 1. Wiley, pp. 3–11, 2020.'
  ista: 'Avagliano L, Parenti I, Grazioli P, Di Fede E, Parodi C, Mariani M, Kaiser
    FJ, Selicorni A, Gervasini C, Massa V. 2020. Chromatinopathies: A focus on Cornelia
    de Lange syndrome. Clinical Genetics. 97(1), 3–11.'
  mla: 'Avagliano, Laura, et al. “Chromatinopathies: A Focus on Cornelia de Lange
    Syndrome.” <i>Clinical Genetics</i>, vol. 97, no. 1, Wiley, 2020, pp. 3–11, doi:<a
    href="https://doi.org/10.1111/cge.13674">10.1111/cge.13674</a>.'
  short: L. Avagliano, I. Parenti, P. Grazioli, E. Di Fede, C. Parodi, M. Mariani,
    F.J. Kaiser, A. Selicorni, C. Gervasini, V. Massa, Clinical Genetics 97 (2020)
    3–11.
date_created: 2019-12-04T16:10:59Z
date_published: 2020-01-01T00:00:00Z
date_updated: 2023-08-17T14:06:20Z
day: '01'
department:
- _id: GaNo
doi: 10.1111/cge.13674
external_id:
  isi:
  - '000562561800001'
  pmid:
  - '31721174'
intvolume: '        97'
isi: 1
issue: '1'
language:
- iso: eng
month: '01'
oa_version: None
page: 3-11
pmid: 1
publication: Clinical Genetics
publication_identifier:
  eissn:
  - 1399-0004
  issn:
  - 0009-9163
publication_status: published
publisher: Wiley
quality_controlled: '1'
scopus_import: '1'
status: public
title: 'Chromatinopathies: A focus on Cornelia de Lange syndrome'
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 97
year: '2020'
...
---
_id: '7160'
abstract:
- lang: eng
  text: 'Nocturnal animals that rely on their visual system for foraging, mating,
    and navigation usually exhibit specific traits associated with living in scotopic
    conditions. Most nocturnal birds have several visual specializations, such as
    enlarged eyes and an increased orbital convergence. However, the actual role of
    binocular vision in nocturnal foraging is still debated. Nightjars (Aves: Caprimulgidae)
    are predators that actively pursue and capture flying insects in crepuscular and
    nocturnal environments, mainly using a conspicuous “sit-and-wait” tactic on which
    pursuit begins with an insect flying over the bird that sits on the ground. In
    this study, we describe the visual system of the band-winged nightjar (Systellura
    longirostris), with emphasis on anatomical features previously described as relevant
    for nocturnal birds. Orbit convergence, determined by 3D scanning of the skull,
    was 73.28°. The visual field, determined by ophthalmoscopic reflex, exhibits an
    area of maximum binocular overlap of 42°, and it is dorsally oriented. The eyes
    showed a nocturnal-like normalized corneal aperture/axial length index. Retinal
    ganglion cells (RGCs) were relatively scant, and distributed in an unusual oblique-band
    pattern, with higher concentrations in the ventrotemporal quadrant. Together,
    these results indicate that the band-winged nightjar exhibits a retinal specialization
    associated with the binocular area of their dorsal visual field, a relevant area
    for pursuit triggering and prey attacks. The RGC distribution observed is unusual
    among birds, but similar to that of some visually dependent insectivorous bats,
    suggesting that those features might be convergent in relation to feeding strategies.'
article_processing_charge: No
article_type: original
author:
- first_name: Juan Esteban
  full_name: Salazar, Juan Esteban
  last_name: Salazar
- first_name: Daniel
  full_name: Severin, Daniel
  last_name: Severin
- first_name: Tomas A
  full_name: Vega Zuniga, Tomas A
  id: 2E7C4E78-F248-11E8-B48F-1D18A9856A87
  last_name: Vega Zuniga
- first_name: Pedro
  full_name: Fernández-Aburto, Pedro
  last_name: Fernández-Aburto
- first_name: Alfonso
  full_name: Deichler, Alfonso
  last_name: Deichler
- first_name: Michel
  full_name: Sallaberry A., Michel
  last_name: Sallaberry A.
- first_name: Jorge
  full_name: Mpodozis, Jorge
  last_name: Mpodozis
citation:
  ama: 'Salazar JE, Severin D, Vega Zuniga TA, et al. Anatomical specializations related
    to foraging in the visual system of a nocturnal insectivorous bird, the band-winged
    nightjar (Aves: Caprimulgiformes). <i>Brain, Behavior and Evolution</i>. 2020;94(1-4):27-36.
    doi:<a href="https://doi.org/10.1159/000504162">10.1159/000504162</a>'
  apa: 'Salazar, J. E., Severin, D., Vega Zuniga, T. A., Fernández-Aburto, P., Deichler,
    A., Sallaberry A., M., &#38; Mpodozis, J. (2020). Anatomical specializations related
    to foraging in the visual system of a nocturnal insectivorous bird, the band-winged
    nightjar (Aves: Caprimulgiformes). <i>Brain, Behavior and Evolution</i>. Karger
    Publishers. <a href="https://doi.org/10.1159/000504162">https://doi.org/10.1159/000504162</a>'
  chicago: 'Salazar, Juan Esteban, Daniel Severin, Tomas A Vega Zuniga, Pedro Fernández-Aburto,
    Alfonso Deichler, Michel Sallaberry A., and Jorge Mpodozis. “Anatomical Specializations
    Related to Foraging in the Visual System of a Nocturnal Insectivorous Bird, the
    Band-Winged Nightjar (Aves: Caprimulgiformes).” <i>Brain, Behavior and Evolution</i>.
    Karger Publishers, 2020. <a href="https://doi.org/10.1159/000504162">https://doi.org/10.1159/000504162</a>.'
  ieee: 'J. E. Salazar <i>et al.</i>, “Anatomical specializations related to foraging
    in the visual system of a nocturnal insectivorous bird, the band-winged nightjar
    (Aves: Caprimulgiformes),” <i>Brain, Behavior and Evolution</i>, vol. 94, no.
    1–4. Karger Publishers, pp. 27–36, 2020.'
  ista: 'Salazar JE, Severin D, Vega Zuniga TA, Fernández-Aburto P, Deichler A, Sallaberry A.
    M, Mpodozis J. 2020. Anatomical specializations related to foraging in the visual
    system of a nocturnal insectivorous bird, the band-winged nightjar (Aves: Caprimulgiformes).
    Brain, Behavior and Evolution. 94(1–4), 27–36.'
  mla: 'Salazar, Juan Esteban, et al. “Anatomical Specializations Related to Foraging
    in the Visual System of a Nocturnal Insectivorous Bird, the Band-Winged Nightjar
    (Aves: Caprimulgiformes).” <i>Brain, Behavior and Evolution</i>, vol. 94, no.
    1–4, Karger Publishers, 2020, pp. 27–36, doi:<a href="https://doi.org/10.1159/000504162">10.1159/000504162</a>.'
  short: J.E. Salazar, D. Severin, T.A. Vega Zuniga, P. Fernández-Aburto, A. Deichler,
    M. Sallaberry A., J. Mpodozis, Brain, Behavior and Evolution 94 (2020) 27–36.
date_created: 2019-12-09T09:04:13Z
date_published: 2020-01-01T00:00:00Z
date_updated: 2024-02-22T15:18:34Z
day: '01'
department:
- _id: MaJö
doi: 10.1159/000504162
external_id:
  isi:
  - '000522856600004'
  pmid:
  - '31751995'
intvolume: '        94'
isi: 1
issue: 1-4
language:
- iso: eng
month: '01'
oa_version: None
page: 27-36
pmid: 1
publication: Brain, Behavior and Evolution
publication_identifier:
  eissn:
  - 1421-9743
  issn:
  - 0006-8977
publication_status: published
publisher: Karger Publishers
quality_controlled: '1'
scopus_import: '1'
status: public
title: 'Anatomical specializations related to foraging in the visual system of a nocturnal
  insectivorous bird, the band-winged nightjar (Aves: Caprimulgiformes)'
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 94
year: '2020'
...
---
_id: '7166'
abstract:
- lang: eng
  text: In the living cell, we encounter a large variety of motile processes such
    as organelle transport and cytoskeleton remodeling. These processes are driven
    by motor proteins that generate force by transducing chemical free energy into
    mechanical work. In many cases, the molecular motors work in teams to collectively
    generate larger forces. Recent optical trapping experiments on small teams of
    cytoskeletal motors indicated that the collectively generated force increases
    with the size of the motor team but that this increase depends on the motor type
    and on whether the motors are studied in vitro or in vivo. Here, we use the theory
    of stochastic processes to describe the motion of N motors in a stationary optical
    trap and to compute the N-dependence of the collectively generated forces. We
    consider six distinct motor types, two kinesins, two dyneins, and two myosins.
    We show that the force increases always linearly with N but with a prefactor that
    depends on the performance of the single motor. Surprisingly, this prefactor increases
    for weaker motors with a lower stall force. This counter-intuitive behavior reflects
    the increased probability with which stronger motors detach from the filament
    during strain generation. Our theoretical results are in quantitative agreement
    with experimental data on small teams of kinesin-1 motors.
article_processing_charge: No
article_type: letter_note
author:
- first_name: Mehmet C
  full_name: Ucar, Mehmet C
  id: 50B2A802-6007-11E9-A42B-EB23E6697425
  last_name: Ucar
  orcid: 0000-0003-0506-4217
- first_name: Reinhard
  full_name: Lipowsky, Reinhard
  last_name: Lipowsky
citation:
  ama: Ucar MC, Lipowsky R. Collective force generation by molecular motors is determined
    by strain-induced unbinding. <i>Nano Letters</i>. 2020;20(1):669-676. doi:<a href="https://doi.org/10.1021/acs.nanolett.9b04445">10.1021/acs.nanolett.9b04445</a>
  apa: Ucar, M. C., &#38; Lipowsky, R. (2020). Collective force generation by molecular
    motors is determined by strain-induced unbinding. <i>Nano Letters</i>. American
    Chemical Society. <a href="https://doi.org/10.1021/acs.nanolett.9b04445">https://doi.org/10.1021/acs.nanolett.9b04445</a>
  chicago: Ucar, Mehmet C, and Reinhard Lipowsky. “Collective Force Generation by
    Molecular Motors Is Determined by Strain-Induced Unbinding.” <i>Nano Letters</i>.
    American Chemical Society, 2020. <a href="https://doi.org/10.1021/acs.nanolett.9b04445">https://doi.org/10.1021/acs.nanolett.9b04445</a>.
  ieee: M. C. Ucar and R. Lipowsky, “Collective force generation by molecular motors
    is determined by strain-induced unbinding,” <i>Nano Letters</i>, vol. 20, no.
    1. American Chemical Society, pp. 669–676, 2020.
  ista: Ucar MC, Lipowsky R. 2020. Collective force generation by molecular motors
    is determined by strain-induced unbinding. Nano Letters. 20(1), 669–676.
  mla: Ucar, Mehmet C., and Reinhard Lipowsky. “Collective Force Generation by Molecular
    Motors Is Determined by Strain-Induced Unbinding.” <i>Nano Letters</i>, vol. 20,
    no. 1, American Chemical Society, 2020, pp. 669–76, doi:<a href="https://doi.org/10.1021/acs.nanolett.9b04445">10.1021/acs.nanolett.9b04445</a>.
  short: M.C. Ucar, R. Lipowsky, Nano Letters 20 (2020) 669–676.
corr_author: '1'
date_created: 2019-12-10T15:36:05Z
date_published: 2020-01-08T00:00:00Z
date_updated: 2024-10-09T20:59:07Z
day: '08'
department:
- _id: EdHa
doi: 10.1021/acs.nanolett.9b04445
external_id:
  isi:
  - '000507151600087'
  pmid:
  - '31797672'
intvolume: '        20'
isi: 1
issue: '1'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://doi.org/10.1021/acs.nanolett.9b04445
month: '01'
oa: 1
oa_version: Published Version
page: 669-676
pmid: 1
publication: Nano Letters
publication_identifier:
  eissn:
  - 1530-6992
  issn:
  - 1530-6984
publication_status: published
publisher: American Chemical Society
quality_controlled: '1'
related_material:
  record:
  - id: '9726'
    relation: research_data
    status: public
  - id: '9885'
    relation: research_data
    status: public
scopus_import: '1'
status: public
title: Collective force generation by molecular motors is determined by strain-induced
  unbinding
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 20
year: '2020'
...
---
_id: '7196'
abstract:
- lang: eng
  text: 'In this thesis we study certain mathematical aspects of evolution. The two
    primary forces that drive an evolutionary process are mutation and selection.
    Mutation generates new variants in a population. Selection chooses among the variants
    depending on the reproductive rates of individuals. Evolutionary processes are
    intrinsically random – a new mutation that is initially present in the population
    at low frequency can go extinct, even if it confers a reproductive advantage.
    The overall rate of evolution is largely determined by two quantities: the probability
    that an invading advantageous mutation spreads through the population (called
    fixation probability) and the time until it does so (called fixation time). Both
    those quantities crucially depend not only on the strength of the invading mutation
    but also on the population structure. In this thesis, we aim to understand how
    the underlying population structure affects the overall rate of evolution. Specifically,
    we study population structures that increase the fixation probability of advantageous
    mutants (called amplifiers of selection). Broadly speaking, our results are of
    three different types: We present various strong amplifiers, we identify regimes
    under which only limited amplification is feasible, and we propose population
    structures that provide different tradeoffs between high fixation probability
    and short fixation time.'
alternative_title:
- ISTA Thesis
article_processing_charge: No
author:
- first_name: Josef
  full_name: Tkadlec, Josef
  id: 3F24CCC8-F248-11E8-B48F-1D18A9856A87
  last_name: Tkadlec
  orcid: 0000-0002-1097-9684
citation:
  ama: Tkadlec J. A role of graphs in evolutionary processes. 2020. doi:<a href="https://doi.org/10.15479/AT:ISTA:7196">10.15479/AT:ISTA:7196</a>
  apa: Tkadlec, J. (2020). <i>A role of graphs in evolutionary processes</i>. Institute
    of Science and Technology Austria. <a href="https://doi.org/10.15479/AT:ISTA:7196">https://doi.org/10.15479/AT:ISTA:7196</a>
  chicago: Tkadlec, Josef. “A Role of Graphs in Evolutionary Processes.” Institute
    of Science and Technology Austria, 2020. <a href="https://doi.org/10.15479/AT:ISTA:7196">https://doi.org/10.15479/AT:ISTA:7196</a>.
  ieee: J. Tkadlec, “A role of graphs in evolutionary processes,” Institute of Science
    and Technology Austria, 2020.
  ista: Tkadlec J. 2020. A role of graphs in evolutionary processes. Institute of
    Science and Technology Austria.
  mla: Tkadlec, Josef. <i>A Role of Graphs in Evolutionary Processes</i>. Institute
    of Science and Technology Austria, 2020, doi:<a href="https://doi.org/10.15479/AT:ISTA:7196">10.15479/AT:ISTA:7196</a>.
  short: J. Tkadlec, A Role of Graphs in Evolutionary Processes, Institute of Science
    and Technology Austria, 2020.
corr_author: '1'
date_created: 2019-12-20T12:26:36Z
date_published: 2020-01-12T00:00:00Z
date_updated: 2025-04-15T08:12:20Z
day: '12'
ddc:
- '519'
degree_awarded: PhD
department:
- _id: KrCh
- _id: GradSch
doi: 10.15479/AT:ISTA:7196
file:
- access_level: closed
  checksum: 451f8e64b0eb26bf297644ac72bfcbe9
  content_type: application/zip
  creator: jtkadlec
  date_created: 2020-01-12T11:49:49Z
  date_updated: 2020-07-14T12:47:52Z
  file_id: '7255'
  file_name: thesis.zip
  file_size: 21100497
  relation: source_file
- access_level: open_access
  checksum: d8c44cbc4f939c49a8efc9d4b8bb3985
  content_type: application/pdf
  creator: dernst
  date_created: 2020-01-28T07:32:42Z
  date_updated: 2020-07-14T12:47:52Z
  file_id: '7367'
  file_name: 2020_Tkadlec_Thesis.pdf
  file_size: 11670983
  relation: main_file
file_date_updated: 2020-07-14T12:47:52Z
has_accepted_license: '1'
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
page: '144'
publication_identifier:
  eissn:
  - 2663-337X
publication_status: published
publisher: Institute of Science and Technology Austria
related_material:
  record:
  - id: '7212'
    relation: dissertation_contains
    status: public
  - id: '5751'
    relation: dissertation_contains
    status: public
  - id: '7210'
    relation: dissertation_contains
    status: public
status: public
supervisor:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
title: A role of graphs in evolutionary processes
type: dissertation
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2020'
...
---
_id: '7204'
abstract:
- lang: eng
  text: Plant root architecture dynamically adapts to various environmental conditions,
    such as salt‐containing soil. The phytohormone abscisic acid (ABA) is involved
    among others also in these developmental adaptations, but the underlying molecular
    mechanism remains elusive. Here, a novel branch of the ABA signaling pathway in
    Arabidopsis involving PYR/PYL/RCAR (abbreviated as PYLs) receptor‐protein phosphatase
    2A (PP2A) complex that acts in parallel to the canonical PYLs‐protein phosphatase
    2C (PP2C) mechanism is identified. The PYLs‐PP2A signaling modulates root gravitropism
    and lateral root formation through regulating phytohormone auxin transport. In
    optimal conditions, PYLs ABA receptor interacts with the catalytic subunits of
    PP2A, increasing their phosphatase activity and thus counteracting PINOID (PID)
    kinase‐mediated phosphorylation of PIN‐FORMED (PIN) auxin transporters. By contrast,
    in salt and osmotic stress conditions, ABA binds to PYLs, inhibiting the PP2A
    activity, which leads to increased PIN phosphorylation and consequently modulated
    directional auxin transport leading to adapted root architecture. This work reveals
    an adaptive mechanism that may flexibly adjust plant root growth to withstand
    saline and osmotic stresses. It occurs via the cross‐talk between the stress hormone
    ABA and the versatile developmental regulator auxin.
article_number: '1901455'
article_processing_charge: No
article_type: original
author:
- first_name: Yang
  full_name: Li, Yang
  last_name: Li
- first_name: Yaping
  full_name: Wang, Yaping
  last_name: Wang
- first_name: Shutang
  full_name: Tan, Shutang
  id: 2DE75584-F248-11E8-B48F-1D18A9856A87
  last_name: Tan
  orcid: 0000-0002-0471-8285
- first_name: Zhen
  full_name: Li, Zhen
  last_name: Li
- first_name: Zhi
  full_name: Yuan, Zhi
  last_name: Yuan
- first_name: Matous
  full_name: Glanc, Matous
  id: 1AE1EA24-02D0-11E9-9BAA-DAF4881429F2
  last_name: Glanc
  orcid: 0000-0003-0619-7783
- first_name: David
  full_name: Domjan, David
  id: C684CD7A-257E-11EA-9B6F-D8588B4F947F
  last_name: Domjan
  orcid: 0000-0003-2267-106X
- first_name: Kai
  full_name: Wang, Kai
  last_name: Wang
- first_name: Wei
  full_name: Xuan, Wei
  last_name: Xuan
- first_name: Yan
  full_name: Guo, Yan
  last_name: Guo
- first_name: Zhizhong
  full_name: Gong, Zhizhong
  last_name: Gong
- first_name: Jiří
  full_name: Friml, Jiří
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Jing
  full_name: Zhang, Jing
  last_name: Zhang
citation:
  ama: Li Y, Wang Y, Tan S, et al. Root growth adaptation is mediated by PYLs ABA
    receptor-PP2A protein phosphatase complex. <i>Advanced Science</i>. 2020;7(3).
    doi:<a href="https://doi.org/10.1002/advs.201901455">10.1002/advs.201901455</a>
  apa: Li, Y., Wang, Y., Tan, S., Li, Z., Yuan, Z., Glanc, M., … Zhang, J. (2020).
    Root growth adaptation is mediated by PYLs ABA receptor-PP2A protein phosphatase
    complex. <i>Advanced Science</i>. Wiley. <a href="https://doi.org/10.1002/advs.201901455">https://doi.org/10.1002/advs.201901455</a>
  chicago: Li, Yang, Yaping Wang, Shutang Tan, Zhen Li, Zhi Yuan, Matous Glanc, David
    Domjan, et al. “Root Growth Adaptation Is Mediated by PYLs ABA Receptor-PP2A Protein
    Phosphatase Complex.” <i>Advanced Science</i>. Wiley, 2020. <a href="https://doi.org/10.1002/advs.201901455">https://doi.org/10.1002/advs.201901455</a>.
  ieee: Y. Li <i>et al.</i>, “Root growth adaptation is mediated by PYLs ABA receptor-PP2A
    protein phosphatase complex,” <i>Advanced Science</i>, vol. 7, no. 3. Wiley, 2020.
  ista: Li Y, Wang Y, Tan S, Li Z, Yuan Z, Glanc M, Domjan D, Wang K, Xuan W, Guo
    Y, Gong Z, Friml J, Zhang J. 2020. Root growth adaptation is mediated by PYLs
    ABA receptor-PP2A protein phosphatase complex. Advanced Science. 7(3), 1901455.
  mla: Li, Yang, et al. “Root Growth Adaptation Is Mediated by PYLs ABA Receptor-PP2A
    Protein Phosphatase Complex.” <i>Advanced Science</i>, vol. 7, no. 3, 1901455,
    Wiley, 2020, doi:<a href="https://doi.org/10.1002/advs.201901455">10.1002/advs.201901455</a>.
  short: Y. Li, Y. Wang, S. Tan, Z. Li, Z. Yuan, M. Glanc, D. Domjan, K. Wang, W.
    Xuan, Y. Guo, Z. Gong, J. Friml, J. Zhang, Advanced Science 7 (2020).
date_created: 2019-12-22T23:00:43Z
date_published: 2020-02-05T00:00:00Z
date_updated: 2023-08-17T14:13:17Z
day: '05'
ddc:
- '580'
department:
- _id: JiFr
doi: 10.1002/advs.201901455
external_id:
  isi:
  - '000501912800001'
  pmid:
  - '32042554'
file:
- access_level: open_access
  checksum: 016eeab5860860af038e2da95ffe75c3
  content_type: application/pdf
  creator: dernst
  date_created: 2020-02-24T14:29:54Z
  date_updated: 2020-07-14T12:47:53Z
  file_id: '7519'
  file_name: 2020_AdvScience_Li.pdf
  file_size: 3586924
  relation: main_file
file_date_updated: 2020-07-14T12:47:53Z
has_accepted_license: '1'
intvolume: '         7'
isi: 1
issue: '3'
language:
- iso: eng
month: '02'
oa: 1
oa_version: Published Version
pmid: 1
publication: Advanced Science
publication_identifier:
  eissn:
  - 2198-3844
publication_status: published
publisher: Wiley
quality_controlled: '1'
scopus_import: '1'
status: public
title: Root growth adaptation is mediated by PYLs ABA receptor-PP2A protein phosphatase
  complex
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 7
year: '2020'
...
---
_id: '7205'
abstract:
- lang: eng
  text: Genetic incompatibilities contribute to reproductive isolation between many
    diverging populations, but it is still unclear to what extent they play a role
    if divergence happens with gene flow. In contact zones between the "Crab" and
    "Wave" ecotypes of the snail Littorina saxatilis, divergent selection forms strong
    barriers to gene flow, while the role of post‐zygotic barriers due to selection
    against hybrids remains unclear. High embryo abortion rates in this species could
    indicate the presence of such barriers. Post‐zygotic barriers might include genetic
    incompatibilities (e.g. Dobzhansky–Muller incompatibilities) but also maladaptation,
    both expected to be most pronounced in contact zones. In addition, embryo abortion
    might reflect physiological stress on females and embryos independent of any genetic
    stress. We examined all embryos of >500 females sampled outside and inside contact
    zones of three populations in Sweden. Females' clutch size ranged from 0 to 1,011
    embryos (mean 130 ± 123), and abortion rates varied between 0% and 100% (mean
    12%). We described female genotypes by using a hybrid index based on hundreds
    of SNPs differentiated between ecotypes with which we characterized female genotypes.
    We also calculated female SNP heterozygosity and inversion karyotype. Clutch size
    did not vary with female hybrid index, and abortion rates were only weakly related
    to hybrid index in two sites but not at all in a third site. No additional variation
    in abortion rate was explained by female SNP heterozygosity, but increased female
    inversion heterozygosity added slightly to increased abortion. Our results show
    only weak and probably biologically insignificant post‐zygotic barriers contributing
    to ecotype divergence, and the high and variable abortion rates were marginally,
    if at all, explained by hybrid index of females.
article_processing_charge: No
article_type: original
author:
- first_name: Kerstin
  full_name: Johannesson, Kerstin
  last_name: Johannesson
- first_name: Zuzanna
  full_name: Zagrodzka, Zuzanna
  last_name: Zagrodzka
- first_name: Rui
  full_name: Faria, Rui
  last_name: Faria
- first_name: Anja M
  full_name: Westram, Anja M
  id: 3C147470-F248-11E8-B48F-1D18A9856A87
  last_name: Westram
  orcid: 0000-0003-1050-4969
- first_name: Roger K.
  full_name: Butlin, Roger K.
  last_name: Butlin
citation:
  ama: Johannesson K, Zagrodzka Z, Faria R, Westram AM, Butlin RK. Is embryo abortion
    a post-zygotic barrier to gene flow between Littorina ecotypes? <i>Journal of
    Evolutionary Biology</i>. 2020;33(3):342-351. doi:<a href="https://doi.org/10.1111/jeb.13570">10.1111/jeb.13570</a>
  apa: Johannesson, K., Zagrodzka, Z., Faria, R., Westram, A. M., &#38; Butlin, R.
    K. (2020). Is embryo abortion a post-zygotic barrier to gene flow between Littorina
    ecotypes? <i>Journal of Evolutionary Biology</i>. Wiley. <a href="https://doi.org/10.1111/jeb.13570">https://doi.org/10.1111/jeb.13570</a>
  chicago: Johannesson, Kerstin, Zuzanna Zagrodzka, Rui Faria, Anja M Westram, and
    Roger K. Butlin. “Is Embryo Abortion a Post-Zygotic Barrier to Gene Flow between
    Littorina Ecotypes?” <i>Journal of Evolutionary Biology</i>. Wiley, 2020. <a href="https://doi.org/10.1111/jeb.13570">https://doi.org/10.1111/jeb.13570</a>.
  ieee: K. Johannesson, Z. Zagrodzka, R. Faria, A. M. Westram, and R. K. Butlin, “Is
    embryo abortion a post-zygotic barrier to gene flow between Littorina ecotypes?,”
    <i>Journal of Evolutionary Biology</i>, vol. 33, no. 3. Wiley, pp. 342–351, 2020.
  ista: Johannesson K, Zagrodzka Z, Faria R, Westram AM, Butlin RK. 2020. Is embryo
    abortion a post-zygotic barrier to gene flow between Littorina ecotypes? Journal
    of Evolutionary Biology. 33(3), 342–351.
  mla: Johannesson, Kerstin, et al. “Is Embryo Abortion a Post-Zygotic Barrier to
    Gene Flow between Littorina Ecotypes?” <i>Journal of Evolutionary Biology</i>,
    vol. 33, no. 3, Wiley, 2020, pp. 342–51, doi:<a href="https://doi.org/10.1111/jeb.13570">10.1111/jeb.13570</a>.
  short: K. Johannesson, Z. Zagrodzka, R. Faria, A.M. Westram, R.K. Butlin, Journal
    of Evolutionary Biology 33 (2020) 342–351.
date_created: 2019-12-22T23:00:43Z
date_published: 2020-03-01T00:00:00Z
date_updated: 2025-07-10T11:54:22Z
day: '01'
ddc:
- '570'
department:
- _id: NiBa
doi: 10.1111/jeb.13570
external_id:
  isi:
  - '000500954800001'
  pmid:
  - '31724256'
file:
- access_level: open_access
  checksum: 7534ff0839709c0c5265c12d29432f03
  content_type: application/pdf
  creator: dernst
  date_created: 2020-09-22T09:42:18Z
  date_updated: 2020-09-22T09:42:18Z
  file_id: '8553'
  file_name: 2020_EvolBiology_Johannesson.pdf
  file_size: 885611
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  success: 1
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intvolume: '        33'
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language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
page: 342-351
pmid: 1
publication: Journal of Evolutionary Biology
publication_identifier:
  eissn:
  - 1420-9101
  issn:
  - 1010-061X
publication_status: published
publisher: Wiley
quality_controlled: '1'
related_material:
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scopus_import: '1'
status: public
title: Is embryo abortion a post-zygotic barrier to gene flow between Littorina ecotypes?
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 33
year: '2020'
...
---
_id: '7207'
abstract:
- lang: eng
  text: The hippocampus plays key roles in learning and memory and is a main target
    of Alzheimer's disease (AD), which causes progressive memory impairments. Despite
    numerous investigations about the processes required for the normal hippocampal
    functions, the neurotransmitter receptors involved in the synaptic deficits by
    which AD disables the hippocampus are not yet characterized. By combining histoblots,
    western blots, immunohistochemistry and high‐resolution immunoelectron microscopic
    methods for GABAB receptors, this study provides a quantitative description of
    the expression and the subcellular localization of GABAB1 in the hippocampus in
    a mouse model of AD at 1, 6 and 12 months of age. Western blots and histoblots
    showed that the total amount of protein and the laminar expression pattern of
    GABAB1 were similar in APP/PS1 mice and in age‐matched wild‐type mice. In contrast,
    immunoelectron microscopic techniques showed that the subcellular localization
    of GABAB1 subunit did not change significantly in APP/PS1 mice at 1 month of age,
    was significantly reduced in the stratum lacunosum‐moleculare of CA1 pyramidal
    cells at 6 months of age and significantly reduced at the membrane surface of
    CA1 pyramidal cells at 12 months of age. This reduction of plasma membrane GABAB1
    was paralleled by a significant increase of the subunit at the intracellular sites.
    We further observed a decrease of membrane‐targeted GABAB receptors in axon terminals
    contacting CA1 pyramidal cells. Our data demonstrate compartment‐ and age‐dependent
    reduction of plasma membrane‐targeted GABAB receptors in the CA1 region of the
    hippocampus, suggesting that this decrease might be enough to alter the GABAB‐mediated
    synaptic transmission taking place in AD.
article_processing_charge: No
article_type: original
author:
- first_name: Alejandro
  full_name: Martín-Belmonte, Alejandro
  last_name: Martín-Belmonte
- first_name: Carolina
  full_name: Aguado, Carolina
  last_name: Aguado
- first_name: Rocío
  full_name: Alfaro-Ruíz, Rocío
  last_name: Alfaro-Ruíz
- first_name: Ana Esther
  full_name: Moreno-Martínez, Ana Esther
  last_name: Moreno-Martínez
- first_name: Luis
  full_name: De La Ossa, Luis
  last_name: De La Ossa
- first_name: José
  full_name: Martínez-Hernández, José
  last_name: Martínez-Hernández
- first_name: Alain
  full_name: Buisson, Alain
  last_name: Buisson
- first_name: Simon
  full_name: Früh, Simon
  last_name: Früh
- first_name: Bernhard
  full_name: Bettler, Bernhard
  last_name: Bettler
- first_name: Ryuichi
  full_name: Shigemoto, Ryuichi
  id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
  last_name: Shigemoto
  orcid: 0000-0001-8761-9444
- first_name: Yugo
  full_name: Fukazawa, Yugo
  last_name: Fukazawa
- first_name: Rafael
  full_name: Luján, Rafael
  last_name: Luján
citation:
  ama: Martín-Belmonte A, Aguado C, Alfaro-Ruíz R, et al. Reduction in the neuronal
    surface of post and presynaptic GABA&#62;B&#60; receptors in the hippocampus in
    a mouse model of Alzheimer’s disease. <i>Brain Pathology</i>. 2020;30(3):554-575.
    doi:<a href="https://doi.org/10.1111/bpa.12802">10.1111/bpa.12802</a>
  apa: Martín-Belmonte, A., Aguado, C., Alfaro-Ruíz, R., Moreno-Martínez, A. E., De
    La Ossa, L., Martínez-Hernández, J., … Luján, R. (2020). Reduction in the neuronal
    surface of post and presynaptic GABA&#62;B&#60; receptors in the hippocampus in
    a mouse model of Alzheimer’s disease. <i>Brain Pathology</i>. Wiley. <a href="https://doi.org/10.1111/bpa.12802">https://doi.org/10.1111/bpa.12802</a>
  chicago: Martín-Belmonte, Alejandro, Carolina Aguado, Rocío Alfaro-Ruíz, Ana Esther
    Moreno-Martínez, Luis De La Ossa, José Martínez-Hernández, Alain Buisson, et al.
    “Reduction in the Neuronal Surface of Post and Presynaptic GABA&#62;B&#60; Receptors
    in the Hippocampus in a Mouse Model of Alzheimer’s Disease.” <i>Brain Pathology</i>.
    Wiley, 2020. <a href="https://doi.org/10.1111/bpa.12802">https://doi.org/10.1111/bpa.12802</a>.
  ieee: A. Martín-Belmonte <i>et al.</i>, “Reduction in the neuronal surface of post
    and presynaptic GABA&#62;B&#60; receptors in the hippocampus in a mouse model
    of Alzheimer’s disease,” <i>Brain Pathology</i>, vol. 30, no. 3. Wiley, pp. 554–575,
    2020.
  ista: Martín-Belmonte A, Aguado C, Alfaro-Ruíz R, Moreno-Martínez AE, De La Ossa
    L, Martínez-Hernández J, Buisson A, Früh S, Bettler B, Shigemoto R, Fukazawa Y,
    Luján R. 2020. Reduction in the neuronal surface of post and presynaptic GABA&#62;B&#60;
    receptors in the hippocampus in a mouse model of Alzheimer’s disease. Brain Pathology.
    30(3), 554–575.
  mla: Martín-Belmonte, Alejandro, et al. “Reduction in the Neuronal Surface of Post
    and Presynaptic GABA&#62;B&#60; Receptors in the Hippocampus in a Mouse Model
    of Alzheimer’s Disease.” <i>Brain Pathology</i>, vol. 30, no. 3, Wiley, 2020,
    pp. 554–75, doi:<a href="https://doi.org/10.1111/bpa.12802">10.1111/bpa.12802</a>.
  short: A. Martín-Belmonte, C. Aguado, R. Alfaro-Ruíz, A.E. Moreno-Martínez, L. De
    La Ossa, J. Martínez-Hernández, A. Buisson, S. Früh, B. Bettler, R. Shigemoto,
    Y. Fukazawa, R. Luján, Brain Pathology 30 (2020) 554–575.
date_created: 2019-12-22T23:00:43Z
date_published: 2020-05-01T00:00:00Z
date_updated: 2025-07-10T11:54:22Z
day: '01'
ddc:
- '570'
department:
- _id: RySh
doi: 10.1111/bpa.12802
ec_funded: 1
external_id:
  isi:
  - '000502270900001'
  pmid:
  - '31729777'
file:
- access_level: open_access
  checksum: 549cc1b18f638a21d17a939ba5563fa9
  content_type: application/pdf
  creator: dernst
  date_created: 2020-09-22T09:47:19Z
  date_updated: 2020-09-22T09:47:19Z
  file_id: '8554'
  file_name: 2020_BrainPathology_MartinBelmonte.pdf
  file_size: 4220935
  relation: main_file
  success: 1
file_date_updated: 2020-09-22T09:47:19Z
has_accepted_license: '1'
intvolume: '        30'
isi: 1
issue: '3'
language:
- iso: eng
month: '05'
oa: 1
oa_version: Published Version
page: 554-575
pmid: 1
project:
- _id: 25CBA828-B435-11E9-9278-68D0E5697425
  call_identifier: H2020
  grant_number: '720270'
  name: Human Brain Project Specific Grant Agreement 1
- _id: 26436750-B435-11E9-9278-68D0E5697425
  call_identifier: H2020
  grant_number: '785907'
  name: Human Brain Project Specific Grant Agreement 2
publication: Brain Pathology
publication_identifier:
  eissn:
  - 1750-3639
  issn:
  - 1015-6305
publication_status: published
publisher: Wiley
quality_controlled: '1'
scopus_import: '1'
status: public
title: Reduction in the neuronal surface of post and presynaptic GABA>B< receptors
  in the hippocampus in a mouse model of Alzheimer's disease
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 30
year: '2020'
...
---
_id: '7212'
abstract:
- lang: eng
  text: The fixation probability of a single mutant invading a population of residents
    is among the most widely-studied quantities in evolutionary dynamics. Amplifiers
    of natural selection are population structures that increase the fixation probability
    of advantageous mutants, compared to well-mixed populations. Extensive studies
    have shown that many amplifiers exist for the Birth-death Moran process, some
    of them substantially increasing the fixation probability or even guaranteeing
    fixation in the limit of large population size. On the other hand, no amplifiers
    are known for the death-Birth Moran process, and computer-assisted exhaustive
    searches have failed to discover amplification. In this work we resolve this disparity,
    by showing that any amplification under death-Birth updating is necessarily bounded
    and transient. Our boundedness result states that even if a population structure
    does amplify selection, the resulting fixation probability is close to that of
    the well-mixed population. Our transience result states that for any population
    structure there exists a threshold r⋆ such that the population structure ceases
    to amplify selection if the mutant fitness advantage r is larger than r⋆. Finally,
    we also extend the above results to δ-death-Birth updating, which is a combination
    of Birth-death and death-Birth updating. On the positive side, we identify population
    structures that maintain amplification for a wide range of values r and δ. These
    results demonstrate that amplification of natural selection depends on the specific
    mechanisms of the evolutionary process.
article_number: e1007494
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Josef
  full_name: Tkadlec, Josef
  id: 3F24CCC8-F248-11E8-B48F-1D18A9856A87
  last_name: Tkadlec
  orcid: 0000-0002-1097-9684
- first_name: Andreas
  full_name: Pavlogiannis, Andreas
  id: 49704004-F248-11E8-B48F-1D18A9856A87
  last_name: Pavlogiannis
  orcid: 0000-0002-8943-0722
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Martin A.
  full_name: Nowak, Martin A.
  last_name: Nowak
citation:
  ama: Tkadlec J, Pavlogiannis A, Chatterjee K, Nowak MA. Limits on amplifiers of
    natural selection under death-Birth updating. <i>PLoS computational biology</i>.
    2020;16. doi:<a href="https://doi.org/10.1371/journal.pcbi.1007494">10.1371/journal.pcbi.1007494</a>
  apa: Tkadlec, J., Pavlogiannis, A., Chatterjee, K., &#38; Nowak, M. A. (2020). Limits
    on amplifiers of natural selection under death-Birth updating. <i>PLoS Computational
    Biology</i>. Public Library of Science. <a href="https://doi.org/10.1371/journal.pcbi.1007494">https://doi.org/10.1371/journal.pcbi.1007494</a>
  chicago: Tkadlec, Josef, Andreas Pavlogiannis, Krishnendu Chatterjee, and Martin
    A. Nowak. “Limits on Amplifiers of Natural Selection under Death-Birth Updating.”
    <i>PLoS Computational Biology</i>. Public Library of Science, 2020. <a href="https://doi.org/10.1371/journal.pcbi.1007494">https://doi.org/10.1371/journal.pcbi.1007494</a>.
  ieee: J. Tkadlec, A. Pavlogiannis, K. Chatterjee, and M. A. Nowak, “Limits on amplifiers
    of natural selection under death-Birth updating,” <i>PLoS computational biology</i>,
    vol. 16. Public Library of Science, 2020.
  ista: Tkadlec J, Pavlogiannis A, Chatterjee K, Nowak MA. 2020. Limits on amplifiers
    of natural selection under death-Birth updating. PLoS computational biology. 16,
    e1007494.
  mla: Tkadlec, Josef, et al. “Limits on Amplifiers of Natural Selection under Death-Birth
    Updating.” <i>PLoS Computational Biology</i>, vol. 16, e1007494, Public Library
    of Science, 2020, doi:<a href="https://doi.org/10.1371/journal.pcbi.1007494">10.1371/journal.pcbi.1007494</a>.
  short: J. Tkadlec, A. Pavlogiannis, K. Chatterjee, M.A. Nowak, PLoS Computational
    Biology 16 (2020).
date_created: 2019-12-23T13:45:11Z
date_published: 2020-01-17T00:00:00Z
date_updated: 2025-04-15T06:29:58Z
day: '17'
ddc:
- '000'
department:
- _id: KrCh
doi: 10.1371/journal.pcbi.1007494
ec_funded: 1
external_id:
  arxiv:
  - '1906.02785'
  isi:
  - '000510916500025'
file:
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  checksum: ce32ee2d2f53aed832f78bbd47e882df
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  creator: dernst
  date_created: 2020-02-03T07:32:42Z
  date_updated: 2020-07-14T12:47:53Z
  file_id: '7441'
  file_name: 2020_PlosCompBio_Tkadlec.pdf
  file_size: 1817531
  relation: main_file
file_date_updated: 2020-07-14T12:47:53Z
has_accepted_license: '1'
intvolume: '        16'
isi: 1
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
project:
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
publication: PLoS computational biology
publication_identifier:
  eissn:
  - '15537358'
publication_status: published
publisher: Public Library of Science
quality_controlled: '1'
related_material:
  record:
  - id: '7196'
    relation: part_of_dissertation
    status: public
scopus_import: '1'
status: public
title: Limits on amplifiers of natural selection under death-Birth updating
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 16
year: '2020'
...
---
_id: '7213'
abstract:
- lang: eng
  text: Persistent homology is a powerful tool in Topological Data Analysis (TDA)
    to capture the topological properties of data succinctly at different spatial
    resolutions. For graphical data, the shape, and structure of the neighborhood
    of individual data items (nodes) are an essential means of characterizing their
    properties. We propose the use of persistent homology methods to capture structural
    and topological properties of graphs and use it to address the problem of link
    prediction. We achieve encouraging results on nine different real-world datasets
    that attest to the potential of persistent homology-based methods for network
    analysis.
alternative_title:
- SCI
article_processing_charge: No
author:
- first_name: Sumit
  full_name: Bhatia, Sumit
  last_name: Bhatia
- first_name: Bapi
  full_name: Chatterjee, Bapi
  id: 3C41A08A-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-2742-4028
- first_name: Deepak
  full_name: Nathani, Deepak
  last_name: Nathani
- first_name: Manohar
  full_name: Kaul, Manohar
  last_name: Kaul
citation:
  ama: 'Bhatia S, Chatterjee B, Nathani D, Kaul M. A persistent homology perspective
    to the link prediction problem. In: <i>Complex Networks and Their Applications
    VIII</i>. Vol 881. Springer Nature; 2020:27-39. doi:<a href="https://doi.org/10.1007/978-3-030-36687-2_3">10.1007/978-3-030-36687-2_3</a>'
  apa: 'Bhatia, S., Chatterjee, B., Nathani, D., &#38; Kaul, M. (2020). A persistent
    homology perspective to the link prediction problem. In <i>Complex Networks and
    their applications VIII</i> (Vol. 881, pp. 27–39). Lisbon, Portugal: Springer
    Nature. <a href="https://doi.org/10.1007/978-3-030-36687-2_3">https://doi.org/10.1007/978-3-030-36687-2_3</a>'
  chicago: Bhatia, Sumit, Bapi Chatterjee, Deepak Nathani, and Manohar Kaul. “A Persistent
    Homology Perspective to the Link Prediction Problem.” In <i>Complex Networks and
    Their Applications VIII</i>, 881:27–39. Springer Nature, 2020. <a href="https://doi.org/10.1007/978-3-030-36687-2_3">https://doi.org/10.1007/978-3-030-36687-2_3</a>.
  ieee: S. Bhatia, B. Chatterjee, D. Nathani, and M. Kaul, “A persistent homology
    perspective to the link prediction problem,” in <i>Complex Networks and their
    applications VIII</i>, Lisbon, Portugal, 2020, vol. 881, pp. 27–39.
  ista: 'Bhatia S, Chatterjee B, Nathani D, Kaul M. 2020. A persistent homology perspective
    to the link prediction problem. Complex Networks and their applications VIII.
    COMPLEX: International Conference on Complex Networks and their Applications,
    SCI, vol. 881, 27–39.'
  mla: Bhatia, Sumit, et al. “A Persistent Homology Perspective to the Link Prediction
    Problem.” <i>Complex Networks and Their Applications VIII</i>, vol. 881, Springer
    Nature, 2020, pp. 27–39, doi:<a href="https://doi.org/10.1007/978-3-030-36687-2_3">10.1007/978-3-030-36687-2_3</a>.
  short: S. Bhatia, B. Chatterjee, D. Nathani, M. Kaul, in:, Complex Networks and
    Their Applications VIII, Springer Nature, 2020, pp. 27–39.
conference:
  end_date: 2019-12-12
  location: Lisbon, Portugal
  name: 'COMPLEX: International Conference on Complex Networks and their Applications'
  start_date: 2019-12-10
date_created: 2019-12-29T23:00:45Z
date_published: 2020-01-01T00:00:00Z
date_updated: 2025-07-10T11:54:23Z
day: '01'
ddc:
- '004'
department:
- _id: DaAl
doi: 10.1007/978-3-030-36687-2_3
ec_funded: 1
external_id:
  isi:
  - '000843927300003'
file:
- access_level: open_access
  checksum: 8951f094c8c7dae9ff8db885199bc296
  content_type: application/pdf
  creator: bchatter
  date_created: 2020-10-08T08:16:48Z
  date_updated: 2020-10-08T08:16:48Z
  file_id: '8625'
  file_name: main.pdf
  file_size: 310598
  relation: main_file
  success: 1
file_date_updated: 2020-10-08T08:16:48Z
has_accepted_license: '1'
intvolume: '       881'
isi: 1
language:
- iso: eng
month: '01'
oa: 1
oa_version: Submitted Version
page: 27-39
project:
- _id: 260C2330-B435-11E9-9278-68D0E5697425
  call_identifier: H2020
  grant_number: '754411'
  name: ISTplus - Postdoctoral Fellowships
publication: Complex Networks and their applications VIII
publication_identifier:
  eissn:
  - 1860-9503
  isbn:
  - '9783030366865'
  issn:
  - 1860-949X
publication_status: published
publisher: Springer Nature
quality_controlled: '1'
scopus_import: '1'
status: public
title: A persistent homology perspective to the link prediction problem
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 881
year: '2020'
...
---
_id: '7218'
abstract:
- lang: eng
  text: The combined resection of skull-infiltrating tumours and immediate cranioplastic
    reconstruction predominantly relies on freehand-moulded solutions. Techniques
    that enable this procedure to be performed easily in routine clinical practice
    would be useful. A cadaveric study was developed in which a new software tool
    was used to perform single-stage reconstructions with prefabricated implants after
    the resection of skull-infiltrating pathologies. A novel 3D visualization and
    interaction framework was developed to create 10 virtual craniotomies in five
    cadaveric specimens. Polyether ether ketone (PEEK) implants were manufactured
    according to the bone defects. The image-guided craniotomy was reconstructed with
    PEEK and compared to polymethyl methacrylate (PMMA). Navigational accuracy and
    surgical precision were assessed. The PEEK workflow resulted in up to 10-fold
    shorter reconstruction times than the standard technique. Surgical precision was
    reflected by the mean 1.1 ± 0.29 mm distance between the virtual and real craniotomy,
    with submillimetre precision in 50%. Assessment of the global offset between virtual
    and actual craniotomy revealed an average shift of 4.5 ± 3.6 mm. The results validated
    the ‘elective single-stage cranioplasty’ technique as a state-of-the-art virtual
    planning method and surgical workflow. This patient-tailored workflow could significantly
    reduce surgical times compared to the traditional, intraoperative acrylic moulding
    method and may be an option for the reconstruction of bone defects in the craniofacial
    region.
article_processing_charge: No
article_type: original
author:
- first_name: Philippe
  full_name: Dodier, Philippe
  last_name: Dodier
- first_name: Fabian
  full_name: Winter, Fabian
  last_name: Winter
- first_name: Thomas
  full_name: Auzinger, Thomas
  id: 4718F954-F248-11E8-B48F-1D18A9856A87
  last_name: Auzinger
  orcid: 0000-0002-1546-3265
- first_name: Gabriel
  full_name: Mistelbauer, Gabriel
  last_name: Mistelbauer
- first_name: Josa M.
  full_name: Frischer, Josa M.
  last_name: Frischer
- first_name: Wei Te
  full_name: Wang, Wei Te
  last_name: Wang
- first_name: Ammar
  full_name: Mallouhi, Ammar
  last_name: Mallouhi
- first_name: Wolfgang
  full_name: Marik, Wolfgang
  last_name: Marik
- first_name: Stefan
  full_name: Wolfsberger, Stefan
  last_name: Wolfsberger
- first_name: Lukas
  full_name: Reissig, Lukas
  last_name: Reissig
- first_name: Firas
  full_name: Hammadi, Firas
  last_name: Hammadi
- first_name: Christian
  full_name: Matula, Christian
  last_name: Matula
- first_name: Arnulf
  full_name: Baumann, Arnulf
  last_name: Baumann
- first_name: Gerhard
  full_name: Bavinzski, Gerhard
  last_name: Bavinzski
citation:
  ama: 'Dodier P, Winter F, Auzinger T, et al. Single-stage bone resection and cranioplastic
    reconstruction: Comparison of a novel software-derived PEEK workflow with the
    standard reconstructive method. <i>International Journal of Oral and Maxillofacial
    Surgery</i>. 2020;49(8):P1007-1015. doi:<a href="https://doi.org/10.1016/j.ijom.2019.11.011">10.1016/j.ijom.2019.11.011</a>'
  apa: 'Dodier, P., Winter, F., Auzinger, T., Mistelbauer, G., Frischer, J. M., Wang,
    W. T., … Bavinzski, G. (2020). Single-stage bone resection and cranioplastic reconstruction:
    Comparison of a novel software-derived PEEK workflow with the standard reconstructive
    method. <i>International Journal of Oral and Maxillofacial Surgery</i>. Elsevier.
    <a href="https://doi.org/10.1016/j.ijom.2019.11.011">https://doi.org/10.1016/j.ijom.2019.11.011</a>'
  chicago: 'Dodier, Philippe, Fabian Winter, Thomas Auzinger, Gabriel Mistelbauer,
    Josa M. Frischer, Wei Te Wang, Ammar Mallouhi, et al. “Single-Stage Bone Resection
    and Cranioplastic Reconstruction: Comparison of a Novel Software-Derived PEEK
    Workflow with the Standard Reconstructive Method.” <i>International Journal of
    Oral and Maxillofacial Surgery</i>. Elsevier, 2020. <a href="https://doi.org/10.1016/j.ijom.2019.11.011">https://doi.org/10.1016/j.ijom.2019.11.011</a>.'
  ieee: 'P. Dodier <i>et al.</i>, “Single-stage bone resection and cranioplastic reconstruction:
    Comparison of a novel software-derived PEEK workflow with the standard reconstructive
    method,” <i>International Journal of Oral and Maxillofacial Surgery</i>, vol.
    49, no. 8. Elsevier, pp. P1007-1015, 2020.'
  ista: 'Dodier P, Winter F, Auzinger T, Mistelbauer G, Frischer JM, Wang WT, Mallouhi
    A, Marik W, Wolfsberger S, Reissig L, Hammadi F, Matula C, Baumann A, Bavinzski
    G. 2020. Single-stage bone resection and cranioplastic reconstruction: Comparison
    of a novel software-derived PEEK workflow with the standard reconstructive method.
    International Journal of Oral and Maxillofacial Surgery. 49(8), P1007-1015.'
  mla: 'Dodier, Philippe, et al. “Single-Stage Bone Resection and Cranioplastic Reconstruction:
    Comparison of a Novel Software-Derived PEEK Workflow with the Standard Reconstructive
    Method.” <i>International Journal of Oral and Maxillofacial Surgery</i>, vol.
    49, no. 8, Elsevier, 2020, pp. P1007-1015, doi:<a href="https://doi.org/10.1016/j.ijom.2019.11.011">10.1016/j.ijom.2019.11.011</a>.'
  short: P. Dodier, F. Winter, T. Auzinger, G. Mistelbauer, J.M. Frischer, W.T. Wang,
    A. Mallouhi, W. Marik, S. Wolfsberger, L. Reissig, F. Hammadi, C. Matula, A. Baumann,
    G. Bavinzski, International Journal of Oral and Maxillofacial Surgery 49 (2020)
    P1007-1015.
date_created: 2019-12-29T23:00:47Z
date_published: 2020-08-01T00:00:00Z
date_updated: 2023-08-17T14:15:22Z
day: '01'
department:
- _id: BeBi
doi: 10.1016/j.ijom.2019.11.011
external_id:
  isi:
  - '000556819800005'
  pmid:
  - '31866145'
intvolume: '        49'
isi: 1
issue: '8'
language:
- iso: eng
month: '08'
oa_version: None
page: P1007-1015
pmid: 1
publication: International Journal of Oral and Maxillofacial Surgery
publication_identifier:
  eissn:
  - 1399-0020
  issn:
  - 0901-5027
publication_status: published
publisher: Elsevier
quality_controlled: '1'
scopus_import: '1'
status: public
title: 'Single-stage bone resection and cranioplastic reconstruction: Comparison of
  a novel software-derived PEEK workflow with the standard reconstructive method'
type: journal_article
user_id: 4359f0d1-fa6c-11eb-b949-802e58b17ae8
volume: 49
year: '2020'
...
---
_id: '7219'
abstract:
- lang: eng
  text: Root system architecture (RSA), governed by the phytohormone auxin, endows
    plants with an adaptive advantage in particular environments. Using geographically
    representative arabidopsis (Arabidopsis thaliana) accessions as a resource for
    GWA mapping, Waidmann et al. and Ogura et al. recently identified two novel components
    involved in modulating auxin-mediated RSA and conferring plant fitness in particular
    habitats.
article_processing_charge: No
article_type: original
author:
- first_name: Guanghui
  full_name: Xiao, Guanghui
  last_name: Xiao
- first_name: Yuzhou
  full_name: Zhang, Yuzhou
  id: 3B6137F2-F248-11E8-B48F-1D18A9856A87
  last_name: Zhang
  orcid: 0000-0003-2627-6956
citation:
  ama: 'Xiao G, Zhang Y. Adaptive growth: Shaping auxin-mediated root system architecture.
    <i>Trends in Plant Science</i>. 2020;25(2):P121-123. doi:<a href="https://doi.org/10.1016/j.tplants.2019.12.001">10.1016/j.tplants.2019.12.001</a>'
  apa: 'Xiao, G., &#38; Zhang, Y. (2020). Adaptive growth: Shaping auxin-mediated
    root system architecture. <i>Trends in Plant Science</i>. Elsevier. <a href="https://doi.org/10.1016/j.tplants.2019.12.001">https://doi.org/10.1016/j.tplants.2019.12.001</a>'
  chicago: 'Xiao, Guanghui, and Yuzhou Zhang. “Adaptive Growth: Shaping Auxin-Mediated
    Root System Architecture.” <i>Trends in Plant Science</i>. Elsevier, 2020. <a
    href="https://doi.org/10.1016/j.tplants.2019.12.001">https://doi.org/10.1016/j.tplants.2019.12.001</a>.'
  ieee: 'G. Xiao and Y. Zhang, “Adaptive growth: Shaping auxin-mediated root system
    architecture,” <i>Trends in Plant Science</i>, vol. 25, no. 2. Elsevier, pp. P121-123,
    2020.'
  ista: 'Xiao G, Zhang Y. 2020. Adaptive growth: Shaping auxin-mediated root system
    architecture. Trends in Plant Science. 25(2), P121-123.'
  mla: 'Xiao, Guanghui, and Yuzhou Zhang. “Adaptive Growth: Shaping Auxin-Mediated
    Root System Architecture.” <i>Trends in Plant Science</i>, vol. 25, no. 2, Elsevier,
    2020, pp. P121-123, doi:<a href="https://doi.org/10.1016/j.tplants.2019.12.001">10.1016/j.tplants.2019.12.001</a>.'
  short: G. Xiao, Y. Zhang, Trends in Plant Science 25 (2020) P121-123.
corr_author: '1'
date_created: 2019-12-29T23:00:48Z
date_published: 2020-02-01T00:00:00Z
date_updated: 2025-07-10T11:54:24Z
day: '01'
department:
- _id: JiFr
doi: 10.1016/j.tplants.2019.12.001
external_id:
  isi:
  - '000508637500001'
  pmid:
  - '31843370'
intvolume: '        25'
isi: 1
issue: '2'
language:
- iso: eng
month: '02'
oa_version: None
page: P121-123
pmid: 1
publication: Trends in Plant Science
publication_identifier:
  issn:
  - 1360-1385
publication_status: published
publisher: Elsevier
quality_controlled: '1'
scopus_import: '1'
status: public
title: 'Adaptive growth: Shaping auxin-mediated root system architecture'
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 25
year: '2020'
...
