[{"title":"Functionals on triangulations of delaunay sets","abstract":[{"lang":"eng","text":"We study densities of functionals over uniformly bounded triangulations of a Delaunay set of vertices, and prove that the minimum is attained for the Delaunay triangulation if this is the case for finite sets."}],"publisher":"Independent University of Moscow","issue":"3","status":"public","quality_controlled":"1","doi":"10.17323/1609-4514-2014-14-3-491-504","year":"2014","volume":14,"citation":{"apa":"Dolbilin, N., Edelsbrunner, H., Glazyrin, A., &#38; Musin, O. (2014). Functionals on triangulations of delaunay sets. <i>Moscow Mathematical Journal</i>. Independent University of Moscow. <a href=\"https://doi.org/10.17323/1609-4514-2014-14-3-491-504\">https://doi.org/10.17323/1609-4514-2014-14-3-491-504</a>","chicago":"Dolbilin, Nikolai, Herbert Edelsbrunner, Alexey Glazyrin, and Oleg Musin. “Functionals on Triangulations of Delaunay Sets.” <i>Moscow Mathematical Journal</i>. Independent University of Moscow, 2014. <a href=\"https://doi.org/10.17323/1609-4514-2014-14-3-491-504\">https://doi.org/10.17323/1609-4514-2014-14-3-491-504</a>.","ieee":"N. Dolbilin, H. Edelsbrunner, A. Glazyrin, and O. Musin, “Functionals on triangulations of delaunay sets,” <i>Moscow Mathematical Journal</i>, vol. 14, no. 3. Independent University of Moscow, pp. 491–504, 2014.","short":"N. Dolbilin, H. Edelsbrunner, A. Glazyrin, O. Musin, Moscow Mathematical Journal 14 (2014) 491–504.","ista":"Dolbilin N, Edelsbrunner H, Glazyrin A, Musin O. 2014. Functionals on triangulations of delaunay sets. Moscow Mathematical Journal. 14(3), 491–504.","mla":"Dolbilin, Nikolai, et al. “Functionals on Triangulations of Delaunay Sets.” <i>Moscow Mathematical Journal</i>, vol. 14, no. 3, Independent University of Moscow, 2014, pp. 491–504, doi:<a href=\"https://doi.org/10.17323/1609-4514-2014-14-3-491-504\">10.17323/1609-4514-2014-14-3-491-504</a>.","ama":"Dolbilin N, Edelsbrunner H, Glazyrin A, Musin O. Functionals on triangulations of delaunay sets. <i>Moscow Mathematical Journal</i>. 2014;14(3):491-504. doi:<a href=\"https://doi.org/10.17323/1609-4514-2014-14-3-491-504\">10.17323/1609-4514-2014-14-3-491-504</a>"},"publication_status":"published","oa":1,"date_updated":"2025-07-10T11:51:26Z","intvolume":"        14","publication":"Moscow Mathematical Journal","oa_version":"Submitted Version","month":"07","_id":"1876","article_type":"original","department":[{"_id":"HeEd"}],"date_created":"2018-12-11T11:54:29Z","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","article_processing_charge":"No","type":"journal_article","day":"01","publist_id":"5220","author":[{"first_name":"Nikolai","full_name":"Dolbilin, Nikolai","last_name":"Dolbilin"},{"orcid":"0000-0002-9823-6833","last_name":"Edelsbrunner","id":"3FB178DA-F248-11E8-B48F-1D18A9856A87","first_name":"Herbert","full_name":"Edelsbrunner, Herbert"},{"full_name":"Glazyrin, Alexey","first_name":"Alexey","last_name":"Glazyrin"},{"full_name":"Musin, Oleg","first_name":"Oleg","last_name":"Musin"}],"publication_identifier":{"issn":["1609-3321"]},"scopus_import":"1","main_file_link":[{"url":"http://arxiv.org/abs/1211.7053","open_access":"1"}],"page":"491 - 504","language":[{"iso":"eng"}],"arxiv":1,"external_id":{"arxiv":["1211.7053"]},"date_published":"2014-07-01T00:00:00Z","fulldoi":"https://doi.org/10.17323/1609-4514-2014-14-3-491-504"},{"corr_author":"1","page":"441 - 442","publist_id":"5219","author":[{"last_name":"Sixt","orcid":"0000-0002-6620-9179","full_name":"Sixt, Michael K","id":"41E9FBEA-F248-11E8-B48F-1D18A9856A87","first_name":"Michael K"},{"first_name":"Kari","id":"368EE576-F248-11E8-B48F-1D18A9856A87","full_name":"Vaahtomeri, Kari","last_name":"Vaahtomeri","orcid":"0000-0001-7829-3518"}],"scopus_import":"1","day":"23","article_processing_charge":"No","type":"journal_article","fulldoi":"https://doi.org/10.1038/514441a","date_published":"2014-10-23T00:00:00Z","external_id":{"isi":["000343775900028"]},"language":[{"iso":"eng"}],"volume":514,"year":"2014","publication_status":"published","citation":{"apa":"Sixt, M. K., &#38; Vaahtomeri, K. (2014). Physiology: Relax and come in. <i>Nature</i>. Springer Nature. <a href=\"https://doi.org/10.1038/514441a\">https://doi.org/10.1038/514441a</a>","chicago":"Sixt, Michael K, and Kari Vaahtomeri. “Physiology: Relax and Come In.” <i>Nature</i>. Springer Nature, 2014. <a href=\"https://doi.org/10.1038/514441a\">https://doi.org/10.1038/514441a</a>.","ieee":"M. K. Sixt and K. Vaahtomeri, “Physiology: Relax and come in,” <i>Nature</i>, vol. 514, no. 7523. Springer Nature, pp. 441–442, 2014.","short":"M.K. Sixt, K. Vaahtomeri, Nature 514 (2014) 441–442.","ista":"Sixt MK, Vaahtomeri K. 2014. Physiology: Relax and come in. Nature. 514(7523), 441–442.","mla":"Sixt, Michael K., and Kari Vaahtomeri. “Physiology: Relax and Come In.” <i>Nature</i>, vol. 514, no. 7523, Springer Nature, 2014, pp. 441–42, doi:<a href=\"https://doi.org/10.1038/514441a\">10.1038/514441a</a>.","ama":"Sixt MK, Vaahtomeri K. Physiology: Relax and come in. <i>Nature</i>. 2014;514(7523):441-442. doi:<a href=\"https://doi.org/10.1038/514441a\">10.1038/514441a</a>"},"doi":"10.1038/514441a","publisher":"Springer Nature","issue":"7523","status":"public","quality_controlled":"1","abstract":[{"lang":"eng","text":"During inflammation, lymph nodes swell with an influx of immune cells. New findings identify a signalling pathway that induces relaxation in the contractile cells that give structure to these organs."}],"title":"Physiology: Relax and come in","article_type":"letter_note","department":[{"_id":"MiSi"}],"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:30Z","month":"10","_id":"1877","intvolume":"       514","oa_version":"None","publication":"Nature","isi":1,"date_updated":"2025-09-29T13:09:03Z"},{"publist_id":"5211","author":[{"full_name":"Landau, Dan","first_name":"Dan","last_name":"Landau"},{"first_name":"Chip","full_name":"Stewart, Chip","last_name":"Stewart"},{"orcid":"0000-0002-0170-7353","last_name":"Reiter","full_name":"Reiter, Johannes","first_name":"Johannes","id":"4A918E98-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Lawrence, Michael","first_name":"Michael","last_name":"Lawrence"},{"full_name":"Sougnez, Carrie","first_name":"Carrie","last_name":"Sougnez"},{"first_name":"Jennifer","full_name":"Brown, Jennifer","last_name":"Brown"},{"last_name":"Lopez Guillermo","full_name":"Lopez Guillermo, Armando","first_name":"Armando"},{"first_name":"Stacey","full_name":"Gabriel, Stacey","last_name":"Gabriel"},{"full_name":"Lander, Eric","first_name":"Eric","last_name":"Lander"},{"full_name":"Neuberg, Donna","first_name":"Donna","last_name":"Neuberg"},{"last_name":"López Otín","first_name":"Carlos","full_name":"López Otín, Carlos"},{"full_name":"Campo, Elias","first_name":"Elias","last_name":"Campo"},{"full_name":"Getz, Gad","first_name":"Gad","last_name":"Getz"},{"full_name":"Wu, Catherine","first_name":"Catherine","last_name":"Wu"}],"main_file_link":[{"url":"http://www.bloodjournal.org/content/124/21/1952?sso-checked=true"}],"year":"2014","volume":124,"publication_status":"published","page":"1952 - 1952","citation":{"ieee":"D. Landau <i>et al.</i>, “Novel putative driver gene mutations in chronic lymphocytic leukemia (CLL): results from a combined analysis of whole exome sequencing of 262 primary CLL aamples,” <i>Blood</i>, vol. 124, no. 21. American Society of Hematology, pp. 1952–1952, 2014.","short":"D. Landau, C. Stewart, J. Reiter, M. Lawrence, C. Sougnez, J. Brown, A. Lopez Guillermo, S. Gabriel, E. Lander, D. Neuberg, C. López Otín, E. Campo, G. Getz, C. Wu, Blood 124 (2014) 1952–1952.","apa":"Landau, D., Stewart, C., Reiter, J., Lawrence, M., Sougnez, C., Brown, J., … Wu, C. (2014). Novel putative driver gene mutations in chronic lymphocytic leukemia (CLL): results from a combined analysis of whole exome sequencing of 262 primary CLL aamples. <i>Blood</i>. American Society of Hematology.","chicago":"Landau, Dan, Chip Stewart, Johannes Reiter, Michael Lawrence, Carrie Sougnez, Jennifer Brown, Armando Lopez Guillermo, et al. “Novel Putative Driver Gene Mutations in Chronic Lymphocytic Leukemia (CLL): Results from a Combined Analysis of Whole Exome Sequencing of 262 Primary CLL Aamples.” <i>Blood</i>. American Society of Hematology, 2014.","mla":"Landau, Dan, et al. “Novel Putative Driver Gene Mutations in Chronic Lymphocytic Leukemia (CLL): Results from a Combined Analysis of Whole Exome Sequencing of 262 Primary CLL Aamples.” <i>Blood</i>, vol. 124, no. 21, American Society of Hematology, 2014, pp. 1952–1952.","ama":"Landau D, Stewart C, Reiter J, et al. Novel putative driver gene mutations in chronic lymphocytic leukemia (CLL): results from a combined analysis of whole exome sequencing of 262 primary CLL aamples. <i>Blood</i>. 2014;124(21):1952-1952.","ista":"Landau D, Stewart C, Reiter J, Lawrence M, Sougnez C, Brown J, Lopez Guillermo A, Gabriel S, Lander E, Neuberg D, López Otín C, Campo E, Getz G, Wu C. 2014. Novel putative driver gene mutations in chronic lymphocytic leukemia (CLL): results from a combined analysis of whole exome sequencing of 262 primary CLL aamples. Blood. 124(21), 1952–1952."},"title":"Novel putative driver gene mutations in chronic lymphocytic leukemia (CLL): results from a combined analysis of whole exome sequencing of 262 primary CLL aamples","abstract":[{"text":"Unbiased high-throughput massively parallel sequencing methods have transformed the process of discovery of novel putative driver gene mutations in cancer. In chronic lymphocytic leukemia (CLL), these methods have yielded several unexpected findings, including the driver genes SF3B1, NOTCH1 and POT1. Recent analysis, utilizing down-sampling of existing datasets, has shown that the discovery process of putative drivers is far from complete across cancer. In CLL, while driver gene mutations affecting >10% of patients were efficiently discovered with previously published CLL cohorts of up to 160 samples subjected to whole exome sequencing (WES), this sample size has only 0.78 power to detect drivers affecting 5% of patients, and only 0.12 power for drivers affecting 2% of patients. These calculations emphasize the need to apply unbiased WES to larger patient cohorts.","lang":"eng"}],"type":"journal_article","day":"04","publisher":"American Society of Hematology","status":"public","issue":"21","month":"12","_id":"1884","department":[{"_id":"KrCh"}],"date_created":"2018-12-11T11:54:32Z","user_id":"4435EBFC-F248-11E8-B48F-1D18A9856A87","date_updated":"2021-01-12T06:53:50Z","language":[{"iso":"eng"}],"intvolume":"       124","date_published":"2014-12-04T00:00:00Z","oa_version":"None","publication":"Blood"},{"has_accepted_license":"1","ddc":["570"],"fulldoi":"https://doi.org/10.7554/eLife.03722","external_id":{"isi":["000209685300001"]},"date_published":"2014-11-14T00:00:00Z","language":[{"iso":"eng"}],"pubrep_id":"420","article_number":"e03722","scopus_import":"1","author":[{"last_name":"Hermundstad","first_name":"Ann","full_name":"Hermundstad, Ann"},{"first_name":"John","full_name":"Briguglio, John","last_name":"Briguglio"},{"last_name":"Conte","full_name":"Conte, Mary","first_name":"Mary"},{"first_name":"Jonathan","full_name":"Victor, Jonathan","last_name":"Victor"},{"last_name":"Balasubramanian","first_name":"Vijay","full_name":"Balasubramanian, Vijay"},{"orcid":"0000-0002-6699-1455","last_name":"Tkacik","id":"3D494DCA-F248-11E8-B48F-1D18A9856A87","first_name":"Gasper","full_name":"Tkacik, Gasper"}],"publist_id":"5209","day":"14","type":"journal_article","article_processing_charge":"No","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:32Z","department":[{"_id":"GaTk"}],"_id":"1886","month":"11","oa_version":"Published Version","publication":"eLife","file_date_updated":"2020-07-14T12:45:20Z","date_updated":"2025-09-29T13:08:33Z","isi":1,"file":[{"date_created":"2018-12-12T10:12:04Z","content_type":"application/pdf","access_level":"open_access","relation":"main_file","file_size":5117086,"checksum":"766ac8999ac6e3364f10065a06024b8f","creator":"system","file_name":"IST-2016-420-v1+1_e03722.full.pdf","date_updated":"2020-07-14T12:45:20Z","file_id":"4922"}],"oa":1,"citation":{"ama":"Hermundstad A, Briguglio J, Conte M, Victor J, Balasubramanian V, Tkačik G. Variance predicts salience in central sensory processing. <i>eLife</i>. 2014;(November). doi:<a href=\"https://doi.org/10.7554/eLife.03722\">10.7554/eLife.03722</a>","mla":"Hermundstad, Ann, et al. “Variance Predicts Salience in Central Sensory Processing.” <i>ELife</i>, no. November, e03722, eLife Sciences Publications, 2014, doi:<a href=\"https://doi.org/10.7554/eLife.03722\">10.7554/eLife.03722</a>.","ista":"Hermundstad A, Briguglio J, Conte M, Victor J, Balasubramanian V, Tkačik G. 2014. Variance predicts salience in central sensory processing. eLife. (November), e03722.","short":"A. Hermundstad, J. Briguglio, M. Conte, J. Victor, V. Balasubramanian, G. Tkačik, ELife (2014).","ieee":"A. Hermundstad, J. Briguglio, M. Conte, J. Victor, V. Balasubramanian, and G. Tkačik, “Variance predicts salience in central sensory processing,” <i>eLife</i>, no. November. eLife Sciences Publications, 2014.","chicago":"Hermundstad, Ann, John Briguglio, Mary Conte, Jonathan Victor, Vijay Balasubramanian, and Gašper Tkačik. “Variance Predicts Salience in Central Sensory Processing.” <i>ELife</i>. eLife Sciences Publications, 2014. <a href=\"https://doi.org/10.7554/eLife.03722\">https://doi.org/10.7554/eLife.03722</a>.","apa":"Hermundstad, A., Briguglio, J., Conte, M., Victor, J., Balasubramanian, V., &#38; Tkačik, G. (2014). Variance predicts salience in central sensory processing. <i>ELife</i>. eLife Sciences Publications. <a href=\"https://doi.org/10.7554/eLife.03722\">https://doi.org/10.7554/eLife.03722</a>"},"publication_status":"published","year":"2014","tmp":{"image":"/images/cc_by.png","short":"CC BY (4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"doi":"10.7554/eLife.03722","quality_controlled":"1","issue":"November","status":"public","project":[{"name":"Sensitivity to higher-order statistics in natural scenes","call_identifier":"FWF","_id":"254D1A94-B435-11E9-9278-68D0E5697425","grant_number":"P 25651-N26"}],"publisher":"eLife Sciences Publications","abstract":[{"lang":"eng","text":"Information processing in the sensory periphery is shaped by natural stimulus statistics. In the periphery, a transmission bottleneck constrains performance; thus efficient coding implies that natural signal components with a predictably wider range should be compressed. In a different regime—when sampling limitations constrain performance—efficient coding implies that more resources should be allocated to informative features that are more variable. We propose that this regime is relevant for sensory cortex when it extracts complex features from limited numbers of sensory samples. To test this prediction, we use central visual processing as a model: we show that visual sensitivity for local multi-point spatial correlations, described by dozens of independently-measured parameters, can be quantitatively predicted from the structure of natural images. This suggests that efficient coding applies centrally, where it extends to higher-order sensory features and operates in a regime in which sensitivity increases with feature variability."}],"title":"Variance predicts salience in central sensory processing"},{"article_processing_charge":"No","title":"Gemeinsame Krankheitsabwehr in Ameisengesellschaften","type":"journal_article","status":"public","day":"01","publisher":"Deutsche Zoologische Gesellschaft","quality_controlled":"1","author":[{"first_name":"Sylvia","id":"2F64EC8C-F248-11E8-B48F-1D18A9856A87","full_name":"Cremer, Sylvia","orcid":"0000-0002-2193-3868","last_name":"Cremer"}],"publist_id":"5208","main_file_link":[{"open_access":"1","url":"https://www.dzg-ev.de/wp-content/uploads/2019/05/zoologie2014.pdf"}],"year":"2014","page":"23 - 30","corr_author":"1","citation":{"apa":"Cremer, S. (2014). Gemeinsame Krankheitsabwehr in Ameisengesellschaften. <i>Zoologie</i>. Deutsche Zoologische Gesellschaft.","chicago":"Cremer, Sylvia. “Gemeinsame Krankheitsabwehr in Ameisengesellschaften.” <i>Zoologie</i>. Deutsche Zoologische Gesellschaft, 2014.","ieee":"S. Cremer, “Gemeinsame Krankheitsabwehr in Ameisengesellschaften,” <i>Zoologie</i>. Deutsche Zoologische Gesellschaft, pp. 23–30, 2014.","short":"S. Cremer, Zoologie (2014) 23–30.","ista":"Cremer S. 2014. Gemeinsame Krankheitsabwehr in Ameisengesellschaften. Zoologie., 23–30.","mla":"Cremer, Sylvia. “Gemeinsame Krankheitsabwehr in Ameisengesellschaften.” <i>Zoologie</i>, Deutsche Zoologische Gesellschaft, 2014, pp. 23–30.","ama":"Cremer S. Gemeinsame Krankheitsabwehr in Ameisengesellschaften. <i>Zoologie</i>. 2014:23-30."},"publication_status":"published","language":[{"iso":"eng"}],"date_updated":"2026-06-18T18:02:48Z","oa":1,"publication":"Zoologie","oa_version":"Published Version","date_published":"2014-01-01T00:00:00Z","ddc":["570"],"month":"01","_id":"1887","department":[{"_id":"SyCr"}],"article_type":"original","date_created":"2018-12-11T11:54:33Z","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87"},{"type":"book_chapter","abstract":[{"text":"Im Rahmen meiner Arbeit mit der kollektiven Krankheitsabwehr in Ameisengesellschaften interessiert mich vor allem, wie sich die Kolonien als Ganzes gegen Krankheiten wehren können. Warum ist dieses Thema der Krankheitsdynamik in Gruppen so wichtig? Ein Vergleich von solitär lebenden Individuen mit Individuen, die in sozialen Gruppen zusammenleben, zeigt die Kosten und die Vorteile des Gruppenlebens: Einerseits haben Individuen in sozialen Gruppen aufgrund der hohen Dichte, in der die Tiere zusammenleben, den hohen Interaktionsraten, die sie miteinander haben, und der engen Verwandtschaft, die sie verbindet, ein höheres Ansteckungsrisiko. Andererseits kann die individuelle Krankheitsabwehr durch die kollektive Abwehr in den Gruppen ergänzt werden.","lang":"ger"}],"article_processing_charge":"No","title":"Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie der Wissenschaften","quality_controlled":"1","publisher":"Verlag Dr. Friedrich Pfeil","day":"01","status":"public","publication_identifier":{"issn":["2366-2875"]},"publist_id":"5207","author":[{"last_name":"Cremer","orcid":"0000-0002-2193-3868","full_name":"Cremer, Sylvia","first_name":"Sylvia","id":"2F64EC8C-F248-11E8-B48F-1D18A9856A87"}],"page":"65 - 72","corr_author":"1","publication_status":"published","citation":{"chicago":"Cremer, Sylvia. “Soziale Immunität: Wie Sich Der Staat Gegen Pathogene Wehrt  Bayerische Akademie Der Wissenschaften.” In <i>Soziale Insekten in Einer Sich Wandelnden Welt</i>, 43:65–72. Verlag Dr. Friedrich Pfeil, 2014.","apa":"Cremer, S. (2014). Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie der Wissenschaften. In <i>Soziale Insekten in einer sich wandelnden Welt</i> (Vol. 43, pp. 65–72). Verlag Dr. Friedrich Pfeil.","short":"S. Cremer, in:, Soziale Insekten in Einer Sich Wandelnden Welt, Verlag Dr. Friedrich Pfeil, 2014, pp. 65–72.","ieee":"S. Cremer, “Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie der Wissenschaften,” in <i>Soziale Insekten in einer sich wandelnden Welt</i>, vol. 43, Verlag Dr. Friedrich Pfeil, 2014, pp. 65–72.","ista":"Cremer S. 2014.Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie der Wissenschaften. In: Soziale Insekten in einer sich wandelnden Welt. Rundgespräche der Kommission für Ökologie, vol. 43, 65–72.","ama":"Cremer S. Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie der Wissenschaften. In: <i>Soziale Insekten in Einer Sich Wandelnden Welt</i>. Vol 43. Verlag Dr. Friedrich Pfeil; 2014:65-72.","mla":"Cremer, Sylvia. “Soziale Immunität: Wie Sich Der Staat Gegen Pathogene Wehrt  Bayerische Akademie Der Wissenschaften.” <i>Soziale Insekten in Einer Sich Wandelnden Welt</i>, vol. 43, Verlag Dr. Friedrich Pfeil, 2014, pp. 65–72."},"year":"2014","volume":43,"language":[{"iso":"eng"}],"date_updated":"2024-10-09T20:55:52Z","date_published":"2014-01-01T00:00:00Z","alternative_title":["Rundgespräche der Kommission für Ökologie"],"oa_version":"None","publication":"Soziale Insekten in einer sich wandelnden Welt","intvolume":"        43","_id":"1888","month":"01","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","date_created":"2018-12-11T11:54:33Z","department":[{"_id":"SyCr"}]},{"article_processing_charge":"No","type":"journal_article","day":"01","publist_id":"5206","author":[{"last_name":"Bräunlich","full_name":"Bräunlich, Gerhard","first_name":"Gerhard"},{"last_name":"Hainzl","full_name":"Hainzl, Christian","first_name":"Christian"},{"full_name":"Seiringer, Robert","first_name":"Robert","id":"4AFD0470-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0002-6781-0521","last_name":"Seiringer"}],"scopus_import":"1","main_file_link":[{"url":"http://arxiv.org/abs/1305.5135","open_access":"1"}],"article_number":"1450012","language":[{"iso":"eng"}],"arxiv":1,"external_id":{"isi":["000341933500002"],"arxiv":["1305.5135"]},"date_published":"2014-08-01T00:00:00Z","fulldoi":"https://doi.org/10.1142/S0129055X14500123","abstract":[{"text":"We study translation-invariant quasi-free states for a system of fermions with two-particle interactions. The associated energy functional is similar to the BCS functional but also includes direct and exchange energies. We show that for suitable short-range interactions, these latter terms only lead to a renormalization of the chemical potential, with the usual properties of the BCS functional left unchanged. Our analysis thus represents a rigorous justification of part of the BCS approximation. We give bounds on the critical temperature below which the system displays superfluidity.","lang":"eng"}],"title":"Translation-invariant quasi-free states for fermionic systems and the BCS approximation","acknowledgement":"We would like to thank Max Lein and Andreas Deuchert for valuable suggestions and remarks. Partial financial support by the NSERC (R.S.) is gratefully acknowledged.","publisher":"World Scientific Publishing","status":"public","issue":"7","quality_controlled":"1","doi":"10.1142/S0129055X14500123","volume":26,"year":"2014","citation":{"ista":"Bräunlich G, Hainzl C, Seiringer R. 2014. Translation-invariant quasi-free states for fermionic systems and the BCS approximation. Reviews in Mathematical Physics. 26(7), 1450012.","ama":"Bräunlich G, Hainzl C, Seiringer R. Translation-invariant quasi-free states for fermionic systems and the BCS approximation. <i>Reviews in Mathematical Physics</i>. 2014;26(7). doi:<a href=\"https://doi.org/10.1142/S0129055X14500123\">10.1142/S0129055X14500123</a>","mla":"Bräunlich, Gerhard, et al. “Translation-Invariant Quasi-Free States for Fermionic Systems and the BCS Approximation.” <i>Reviews in Mathematical Physics</i>, vol. 26, no. 7, 1450012, World Scientific Publishing, 2014, doi:<a href=\"https://doi.org/10.1142/S0129055X14500123\">10.1142/S0129055X14500123</a>.","chicago":"Bräunlich, Gerhard, Christian Hainzl, and Robert Seiringer. “Translation-Invariant Quasi-Free States for Fermionic Systems and the BCS Approximation.” <i>Reviews in Mathematical Physics</i>. World Scientific Publishing, 2014. <a href=\"https://doi.org/10.1142/S0129055X14500123\">https://doi.org/10.1142/S0129055X14500123</a>.","apa":"Bräunlich, G., Hainzl, C., &#38; Seiringer, R. (2014). Translation-invariant quasi-free states for fermionic systems and the BCS approximation. <i>Reviews in Mathematical Physics</i>. World Scientific Publishing. <a href=\"https://doi.org/10.1142/S0129055X14500123\">https://doi.org/10.1142/S0129055X14500123</a>","short":"G. Bräunlich, C. Hainzl, R. Seiringer, Reviews in Mathematical Physics 26 (2014).","ieee":"G. Bräunlich, C. Hainzl, and R. Seiringer, “Translation-invariant quasi-free states for fermionic systems and the BCS approximation,” <i>Reviews in Mathematical Physics</i>, vol. 26, no. 7. World Scientific Publishing, 2014."},"publication_status":"published","oa":1,"date_updated":"2025-09-29T13:07:59Z","isi":1,"intvolume":"        26","publication":"Reviews in Mathematical Physics","oa_version":"Submitted Version","month":"08","_id":"1889","article_type":"original","department":[{"_id":"RoSe"}],"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:33Z"},{"title":"Sequential effects in continued visual search: Using fixation-related potentials to compare distractor processing before and after target detection","abstract":[{"text":"To search for a target in a complex environment is an everyday behavior that ends with finding the target. When we search for two identical targets, however, we must continue the search after finding the first target and memorize its location. We used fixation-related potentials to investigate the neural correlates of different stages of the search, that is, before and after finding the first target. Having found the first target influenced subsequent distractor processing. Compared to distractor fixations before the first target fixation, a negative shift was observed for three subsequent distractor fixations. These results suggest that processing a target in continued search modulates the brain's response, either transiently by reflecting temporary working memory processes or permanently by reflecting working memory retention.","lang":"eng"}],"acknowledgement":"Funded by Austrian Science Fund (FWF) Grant Number: P 22189-B18; European Union within the 6th Framework Programme Grant Number: 517590; State government of Styria Grant Number: PN 4055","publisher":"Wiley-Blackwell","status":"public","issue":"4","doi":"10.1111/psyp.12062","volume":51,"tmp":{"image":"/images/cc_by.png","short":"CC BY (4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"year":"2014","publication_status":"published","citation":{"ieee":"C. Körner, V. Braunstein, M. Stangl, A. Schlögl, C. Neuper, and A. Ischebeck, “Sequential effects in continued visual search: Using fixation-related potentials to compare distractor processing before and after target detection,” <i>Psychophysiology</i>, vol. 51, no. 4. Wiley-Blackwell, pp. 385–395, 2014.","short":"C. Körner, V. Braunstein, M. Stangl, A. Schlögl, C. Neuper, A. Ischebeck, Psychophysiology 51 (2014) 385–395.","apa":"Körner, C., Braunstein, V., Stangl, M., Schlögl, A., Neuper, C., &#38; Ischebeck, A. (2014). Sequential effects in continued visual search: Using fixation-related potentials to compare distractor processing before and after target detection. <i>Psychophysiology</i>. Wiley-Blackwell. <a href=\"https://doi.org/10.1111/psyp.12062\">https://doi.org/10.1111/psyp.12062</a>","chicago":"Körner, Christof, Verena Braunstein, Matthias Stangl, Alois Schlögl, Christa Neuper, and Anja Ischebeck. “Sequential Effects in Continued Visual Search: Using Fixation-Related Potentials to Compare Distractor Processing before and after Target Detection.” <i>Psychophysiology</i>. Wiley-Blackwell, 2014. <a href=\"https://doi.org/10.1111/psyp.12062\">https://doi.org/10.1111/psyp.12062</a>.","mla":"Körner, Christof, et al. “Sequential Effects in Continued Visual Search: Using Fixation-Related Potentials to Compare Distractor Processing before and after Target Detection.” <i>Psychophysiology</i>, vol. 51, no. 4, Wiley-Blackwell, 2014, pp. 385–95, doi:<a href=\"https://doi.org/10.1111/psyp.12062\">10.1111/psyp.12062</a>.","ama":"Körner C, Braunstein V, Stangl M, Schlögl A, Neuper C, Ischebeck A. Sequential effects in continued visual search: Using fixation-related potentials to compare distractor processing before and after target detection. <i>Psychophysiology</i>. 2014;51(4):385-395. doi:<a href=\"https://doi.org/10.1111/psyp.12062\">10.1111/psyp.12062</a>","ista":"Körner C, Braunstein V, Stangl M, Schlögl A, Neuper C, Ischebeck A. 2014. Sequential effects in continued visual search: Using fixation-related potentials to compare distractor processing before and after target detection. Psychophysiology. 51(4), 385–395."},"oa":1,"file":[{"file_size":543243,"checksum":"4255b6185e774acce1d99f8e195c564d","relation":"main_file","access_level":"open_access","date_created":"2018-12-12T10:16:44Z","content_type":"application/pdf","date_updated":"2020-07-14T12:45:20Z","file_id":"5233","creator":"system","file_name":"IST-2016-442-v1+1_K-rner_et_al-2014-Psychophysiology.pdf"}],"date_updated":"2025-09-29T13:07:21Z","isi":1,"intvolume":"        51","file_date_updated":"2020-07-14T12:45:20Z","oa_version":"Published Version","publication":"Psychophysiology","month":"02","_id":"1890","department":[{"_id":"ScienComp"},{"_id":"PeJo"}],"date_created":"2018-12-11T11:54:34Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","article_processing_charge":"No","type":"journal_article","day":"11","publist_id":"5205","author":[{"last_name":"Körner","first_name":"Christof","full_name":"Körner, Christof"},{"last_name":"Braunstein","first_name":"Verena","full_name":"Braunstein, Verena"},{"first_name":"Matthias","full_name":"Stangl, Matthias","last_name":"Stangl"},{"full_name":"Schlögl, Alois","id":"45BF87EE-F248-11E8-B48F-1D18A9856A87","first_name":"Alois","last_name":"Schlögl","orcid":"0000-0002-5621-8100"},{"last_name":"Neuper","first_name":"Christa","full_name":"Neuper, Christa"},{"last_name":"Ischebeck","full_name":"Ischebeck, Anja","first_name":"Anja"}],"scopus_import":"1","page":"385 - 395","pubrep_id":"442","language":[{"iso":"eng"}],"date_published":"2014-02-11T00:00:00Z","external_id":{"isi":["000332585900010"]},"fulldoi":"https://doi.org/10.1111/psyp.12062","ddc":["000"],"has_accepted_license":"1"},{"publisher":"Wiley-Blackwell","status":"public","issue":"5","acknowledgement":"Grant Nr. 2011/01/N/ST3/02475","abstract":[{"lang":"eng","text":"We provide theoretical tests of a novel experimental technique to determine mechanostability of proteins based on stretching a mechanically protected protein by single-molecule force spectroscopy. This technique involves stretching a homogeneous or heterogeneous chain of reference proteins (single-molecule markers) in which one of them acts as host to the guest protein under study. The guest protein is grafted into the host through genetic engineering. It is expected that unraveling of the host precedes the unraveling of the guest removing ambiguities in the reading of the force-extension patterns of the guest protein. We study examples of such systems within a coarse-grained structure-based model. We consider systems with various ratios of mechanostability for the host and guest molecules and compare them to experimental results involving cohesin I as the guest molecule. For a comparison, we also study the force-displacement patterns in proteins that are linked in a serial fashion. We find that the mechanostability of the guest is similar to that of the isolated or serially linked protein. We also demonstrate that the ideal configuration of this strategy would be one in which the host is much more mechanostable than the single-molecule markers. We finally show that it is troublesome to use the highly stable cystine knot proteins as a host to graft a guest in stretching studies because this would involve a cleaving procedure."}],"title":"Theoretical tests of the mechanical protection strategy in protein nanomechanics","publication_status":"published","citation":{"chicago":"Chwastyk, Mateusz, Albert Galera Prat, Mateusz K Sikora, Àngel Gómez Sicilia, Mariano Carrión Vázquez, and Marek Cieplak. “Theoretical Tests of the Mechanical Protection Strategy in Protein Nanomechanics.” <i>Proteins: Structure, Function and Bioinformatics</i>. Wiley-Blackwell, 2014. <a href=\"https://doi.org/10.1002/prot.24436\">https://doi.org/10.1002/prot.24436</a>.","apa":"Chwastyk, M., Galera Prat, A., Sikora, M. K., Gómez Sicilia, À., Carrión Vázquez, M., &#38; Cieplak, M. (2014). Theoretical tests of the mechanical protection strategy in protein nanomechanics. <i>Proteins: Structure, Function and Bioinformatics</i>. Wiley-Blackwell. <a href=\"https://doi.org/10.1002/prot.24436\">https://doi.org/10.1002/prot.24436</a>","short":"M. Chwastyk, A. Galera Prat, M.K. Sikora, À. Gómez Sicilia, M. Carrión Vázquez, M. Cieplak, Proteins: Structure, Function and Bioinformatics 82 (2014) 717–726.","ieee":"M. Chwastyk, A. Galera Prat, M. K. Sikora, À. Gómez Sicilia, M. Carrión Vázquez, and M. Cieplak, “Theoretical tests of the mechanical protection strategy in protein nanomechanics,” <i>Proteins: Structure, Function and Bioinformatics</i>, vol. 82, no. 5. Wiley-Blackwell, pp. 717–726, 2014.","ista":"Chwastyk M, Galera Prat A, Sikora MK, Gómez Sicilia À, Carrión Vázquez M, Cieplak M. 2014. Theoretical tests of the mechanical protection strategy in protein nanomechanics. Proteins: Structure, Function and Bioinformatics. 82(5), 717–726.","ama":"Chwastyk M, Galera Prat A, Sikora MK, Gómez Sicilia À, Carrión Vázquez M, Cieplak M. Theoretical tests of the mechanical protection strategy in protein nanomechanics. <i>Proteins: Structure, Function and Bioinformatics</i>. 2014;82(5):717-726. doi:<a href=\"https://doi.org/10.1002/prot.24436\">10.1002/prot.24436</a>","mla":"Chwastyk, Mateusz, et al. “Theoretical Tests of the Mechanical Protection Strategy in Protein Nanomechanics.” <i>Proteins: Structure, Function and Bioinformatics</i>, vol. 82, no. 5, Wiley-Blackwell, 2014, pp. 717–26, doi:<a href=\"https://doi.org/10.1002/prot.24436\">10.1002/prot.24436</a>."},"volume":82,"year":"2014","doi":"10.1002/prot.24436","oa_version":"None","publication":"Proteins: Structure, Function and Bioinformatics","intvolume":"        82","date_updated":"2025-09-29T13:06:49Z","isi":1,"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:34Z","department":[{"_id":"CaHe"}],"_id":"1891","month":"05","day":"01","type":"journal_article","article_processing_charge":"No","page":"717 - 726","scopus_import":"1","publist_id":"5204","author":[{"last_name":"Chwastyk","full_name":"Chwastyk, Mateusz","first_name":"Mateusz"},{"last_name":"Galera Prat","full_name":"Galera Prat, Albert","first_name":"Albert"},{"full_name":"Sikora, Mateusz K","first_name":"Mateusz K","id":"2F74BCDE-F248-11E8-B48F-1D18A9856A87","last_name":"Sikora"},{"full_name":"Gómez Sicilia, Àngel","first_name":"Àngel","last_name":"Gómez Sicilia"},{"last_name":"Carrión Vázquez","full_name":"Carrión Vázquez, Mariano","first_name":"Mariano"},{"last_name":"Cieplak","full_name":"Cieplak, Marek","first_name":"Marek"}],"date_published":"2014-05-01T00:00:00Z","external_id":{"isi":["000337280400003"]},"language":[{"iso":"eng"}],"fulldoi":"https://doi.org/10.1002/prot.24436"},{"publication":"PNAS","oa_version":"Submitted Version","intvolume":"       111","date_updated":"2025-09-29T13:05:44Z","isi":1,"oa":1,"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:34Z","department":[{"_id":"JiFr"}],"ec_funded":1,"_id":"1893","month":"02","issue":"7","status":"public","project":[{"name":"Polarity and subcellular dynamics in plants","call_identifier":"FP7","grant_number":"282300","_id":"25716A02-B435-11E9-9278-68D0E5697425"}],"publisher":"National Academy of Sciences","acknowledgement":"This work was supported by grants from the Research Foundation-Flanders (Odysseus).","abstract":[{"text":"Phosphatidylinositol (PtdIns) is a structural phospholipid that can be phosphorylated into various lipid signaling molecules, designated polyphosphoinositides (PPIs). The reversible phosphorylation of PPIs on the 3, 4, or 5 position of inositol is performed by a set of organelle-specific kinases and phosphatases, and the characteristic head groups make these molecules ideal for regulating biological processes in time and space. In yeast and mammals, PtdIns3P and PtdIns(3,5)P2 play crucial roles in trafficking toward the lytic compartments, whereas the role in plants is not yet fully understood. Here we identified the role of a land plant-specific subgroup of PPI phosphatases, the suppressor of actin 2 (SAC2) to SAC5, during vacuolar trafficking and morphogenesis in Arabidopsis thaliana. SAC2-SAC5 localize to the tonoplast along with PtdIns3P, the presumable product of their activity. In SAC gain- and loss-of-function mutants, the levels of PtdIns monophosphates and bisphosphates were changed, with opposite effects on the morphology of storage and lytic vacuoles, and the trafficking toward the vacuoles was defective. Moreover, multiple sac knockout mutants had an increased number of smaller storage and lytic vacuoles, whereas extralarge vacuoles were observed in the overexpression lines, correlating with various growth and developmental defects. The fragmented vacuolar phenotype of sac mutants could be mimicked by treating wild-type seedlings with PtdIns(3,5)P2, corroborating that this PPI is important for vacuole morphology. Taken together, these results provide evidence that PPIs, together with their metabolic enzymes SAC2-SAC5, are crucial for vacuolar trafficking and for vacuolar morphology and function in plants.","lang":"eng"}],"title":"SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function in Arabidopsis","citation":{"ieee":"P. Marhavá <i>et al.</i>, “SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function in Arabidopsis,” <i>PNAS</i>, vol. 111, no. 7. National Academy of Sciences, pp. 2818–2823, 2014.","short":"P. Marhavá, S. Hirsch, E. Feraru, R. Tejos, R. Van Wijk, T. Viaene, M. Heilmann, J. Lerche, R. De Rycke, M. Feraru, P. Grones, M. Van Montagu, I. Heilmann, T. Munnik, J. Friml, PNAS 111 (2014) 2818–2823.","apa":"Marhavá, P., Hirsch, S., Feraru, E., Tejos, R., Van Wijk, R., Viaene, T., … Friml, J. (2014). SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function in Arabidopsis. <i>PNAS</i>. National Academy of Sciences. <a href=\"https://doi.org/10.1073/pnas.1324264111\">https://doi.org/10.1073/pnas.1324264111</a>","chicago":"Marhavá, Petra, Sibylle Hirsch, Elena Feraru, Ricardo Tejos, Ringo Van Wijk, Tom Viaene, Mareike Heilmann, et al. “SAC Phosphoinositide Phosphatases at the Tonoplast Mediate Vacuolar Function in Arabidopsis.” <i>PNAS</i>. National Academy of Sciences, 2014. <a href=\"https://doi.org/10.1073/pnas.1324264111\">https://doi.org/10.1073/pnas.1324264111</a>.","mla":"Marhavá, Petra, et al. “SAC Phosphoinositide Phosphatases at the Tonoplast Mediate Vacuolar Function in Arabidopsis.” <i>PNAS</i>, vol. 111, no. 7, National Academy of Sciences, 2014, pp. 2818–23, doi:<a href=\"https://doi.org/10.1073/pnas.1324264111\">10.1073/pnas.1324264111</a>.","ama":"Marhavá P, Hirsch S, Feraru E, et al. SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function in Arabidopsis. <i>PNAS</i>. 2014;111(7):2818-2823. doi:<a href=\"https://doi.org/10.1073/pnas.1324264111\">10.1073/pnas.1324264111</a>","ista":"Marhavá P, Hirsch S, Feraru E, Tejos R, Van Wijk R, Viaene T, Heilmann M, Lerche J, De Rycke R, Feraru M, Grones P, Van Montagu M, Heilmann I, Munnik T, Friml J. 2014. SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function in Arabidopsis. PNAS. 111(7), 2818–2823."},"publication_status":"published","year":"2014","volume":111,"doi":"10.1073/pnas.1324264111","external_id":{"isi":["000331396500082"]},"date_published":"2014-02-18T00:00:00Z","language":[{"iso":"eng"}],"fulldoi":"https://doi.org/10.1073/pnas.1324264111","day":"18","type":"journal_article","article_processing_charge":"No","page":"2818 - 2823","corr_author":"1","main_file_link":[{"open_access":"1","url":"http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3932866/"}],"scopus_import":"1","author":[{"first_name":"Petra","id":"44E59624-F248-11E8-B48F-1D18A9856A87","full_name":"Nováková, Petra","last_name":"Nováková"},{"last_name":"Hirsch","first_name":"Sibylle","full_name":"Hirsch, Sibylle"},{"last_name":"Feraru","first_name":"Elena","full_name":"Feraru, Elena"},{"last_name":"Tejos","full_name":"Tejos, Ricardo","first_name":"Ricardo"},{"full_name":"Van Wijk, Ringo","first_name":"Ringo","last_name":"Van Wijk"},{"first_name":"Tom","full_name":"Viaene, Tom","last_name":"Viaene"},{"last_name":"Heilmann","first_name":"Mareike","full_name":"Heilmann, Mareike"},{"full_name":"Lerche, Jennifer","first_name":"Jennifer","last_name":"Lerche"},{"full_name":"De Rycke, Riet","first_name":"Riet","last_name":"De Rycke"},{"first_name":"Mugurel","full_name":"Feraru, Mugurel","last_name":"Feraru"},{"last_name":"Grones","full_name":"Grones, Peter","id":"399876EC-F248-11E8-B48F-1D18A9856A87","first_name":"Peter"},{"last_name":"Van Montagu","first_name":"Marc","full_name":"Van Montagu, Marc"},{"full_name":"Heilmann, Ingo","first_name":"Ingo","last_name":"Heilmann"},{"full_name":"Munnik, Teun","first_name":"Teun","last_name":"Munnik"},{"last_name":"Friml","orcid":"0000-0002-8302-7596","full_name":"Friml, Jirí","first_name":"Jirí","id":"4159519E-F248-11E8-B48F-1D18A9856A87"}],"publist_id":"5202"},{"language":[{"iso":"eng"}],"external_id":{"isi":["000341231500064"]},"date_published":"2014-09-02T00:00:00Z","ddc":["570"],"fulldoi":"https://doi.org/10.1371/journal.pone.0106247","has_accepted_license":"1","article_processing_charge":"No","type":"journal_article","day":"02","author":[{"full_name":"Grabowska, Anna","first_name":"Anna","last_name":"Grabowska"},{"last_name":"Wywiał","full_name":"Wywiał, Ewa","first_name":"Ewa"},{"last_name":"Dunin Horkawicz","first_name":"Stanislaw","full_name":"Dunin Horkawicz, Stanislaw"},{"full_name":"Łasica, Anna","first_name":"Anna","last_name":"Łasica"},{"last_name":"Wösten","full_name":"Wösten, Marc","first_name":"Marc"},{"id":"3ABC5BA6-F248-11E8-B48F-1D18A9856A87","first_name":"Anna A","full_name":"Nagy-Staron, Anna A","last_name":"Nagy-Staron","orcid":"0000-0002-1391-8377"},{"first_name":"Renata","full_name":"Godlewska, Renata","last_name":"Godlewska"},{"full_name":"Bocian Ostrzycka, Katarzyna","first_name":"Katarzyna","last_name":"Bocian Ostrzycka"},{"full_name":"Pieńkowska, Katarzyna","first_name":"Katarzyna","last_name":"Pieńkowska"},{"first_name":"Paweł","full_name":"Łaniewski, Paweł","last_name":"Łaniewski"},{"full_name":"Bujnicki, Janusz","first_name":"Janusz","last_name":"Bujnicki"},{"full_name":"Van Putten, Jos","first_name":"Jos","last_name":"Van Putten"},{"first_name":"Elzbieta","full_name":"Jagusztyn Krynicka, Elzbieta","last_name":"Jagusztyn Krynicka"}],"publist_id":"5201","scopus_import":"1","pubrep_id":"438","article_number":"e106247","date_updated":"2025-09-29T13:05:10Z","isi":1,"file":[{"date_updated":"2020-07-14T12:45:20Z","file_id":"5205","file_name":"IST-2016-438-v1+1_journal.pone.0106247.pdf","creator":"system","file_size":4248801,"checksum":"7d02c3da7f72b82bb5d7932d80c3251f","relation":"main_file","access_level":"open_access","content_type":"application/pdf","date_created":"2018-12-12T10:16:19Z"}],"oa":1,"file_date_updated":"2020-07-14T12:45:20Z","intvolume":"         9","oa_version":"Published Version","publication":"PLoS One","month":"09","_id":"1894","department":[{"_id":"CaGu"}],"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:35Z","abstract":[{"text":"Background: Bacterial Dsb enzymes are involved in the oxidative folding of many proteins, through the formation of disulfide bonds between their cysteine residues. The Dsb protein network has been well characterized in cells of the model microorganism Escherichia coli. To gain insight into the functioning of the Dsb system in epsilon-Proteobacteria, where it plays an important role in the colonization process, we studied two homologs of the main Escherichia coli Dsb oxidase (EcDsbA) that are present in the cells of the enteric pathogen Campylobacter jejuni, the most frequently reported bacterial cause of human enteritis in the world. Methods and Results: Phylogenetic analysis suggests the horizontal transfer of the epsilon-Proteobacterial DsbAs from a common ancestor to gamma-Proteobacteria, which then gave rise to the DsbL lineage. Phenotype and enzymatic assays suggest that the two C. jejuni DsbAs play different roles in bacterial cells and have divergent substrate spectra. CjDsbA1 is essential for the motility and autoagglutination phenotypes, while CjDsbA2 has no impact on those processes. CjDsbA1 plays a critical role in the oxidative folding that ensures the activity of alkaline phosphatase CjPhoX, whereas CjDsbA2 is crucial for the activity of arylsulfotransferase CjAstA, encoded within the dsbA2-dsbB-astA operon. Conclusions: Our results show that CjDsbA1 is the primary thiol-oxidoreductase affecting life processes associated with bacterial spread and host colonization, as well as ensuring the oxidative folding of particular protein substrates. In contrast, CjDsbA2 activity does not affect the same processes and so far its oxidative folding activity has been demonstrated for one substrate, arylsulfotransferase CjAstA. The results suggest the cooperation between CjDsbA2 and CjDsbB. In the case of the CjDsbA1, this cooperation is not exclusive and there is probably another protein to be identified in C. jejuni cells that acts to re-oxidize CjDsbA1. Altogether the data presented here constitute the considerable insight to the Epsilonproteobacterial Dsb systems, which have been poorly understood so far.","lang":"eng"}],"title":"Functional and bioinformatics analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA","status":"public","issue":"9","publisher":"Public Library of Science","quality_controlled":"1","doi":"10.1371/journal.pone.0106247","tmp":{"image":"/images/cc_by.png","short":"CC BY (4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"year":"2014","volume":9,"publication_status":"published","citation":{"apa":"Grabowska, A., Wywiał, E., Dunin Horkawicz, S., Łasica, A., Wösten, M., Nagy-Staron, A. A., … Jagusztyn Krynicka, E. (2014). Functional and bioinformatics analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA. <i>PLoS One</i>. Public Library of Science. <a href=\"https://doi.org/10.1371/journal.pone.0106247\">https://doi.org/10.1371/journal.pone.0106247</a>","chicago":"Grabowska, Anna, Ewa Wywiał, Stanislaw Dunin Horkawicz, Anna Łasica, Marc Wösten, Anna A Nagy-Staron, Renata Godlewska, et al. “Functional and Bioinformatics Analysis of Two Campylobacter Jejuni Homologs of the Thiol-Disulfide Oxidoreductase, DsbA.” <i>PLoS One</i>. Public Library of Science, 2014. <a href=\"https://doi.org/10.1371/journal.pone.0106247\">https://doi.org/10.1371/journal.pone.0106247</a>.","ieee":"A. Grabowska <i>et al.</i>, “Functional and bioinformatics analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA,” <i>PLoS One</i>, vol. 9, no. 9. Public Library of Science, 2014.","short":"A. Grabowska, E. Wywiał, S. Dunin Horkawicz, A. Łasica, M. Wösten, A.A. Nagy-Staron, R. Godlewska, K. Bocian Ostrzycka, K. Pieńkowska, P. Łaniewski, J. Bujnicki, J. Van Putten, E. Jagusztyn Krynicka, PLoS One 9 (2014).","ista":"Grabowska A, Wywiał E, Dunin Horkawicz S, Łasica A, Wösten M, Nagy-Staron AA, Godlewska R, Bocian Ostrzycka K, Pieńkowska K, Łaniewski P, Bujnicki J, Van Putten J, Jagusztyn Krynicka E. 2014. Functional and bioinformatics analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA. PLoS One. 9(9), e106247.","mla":"Grabowska, Anna, et al. “Functional and Bioinformatics Analysis of Two Campylobacter Jejuni Homologs of the Thiol-Disulfide Oxidoreductase, DsbA.” <i>PLoS One</i>, vol. 9, no. 9, e106247, Public Library of Science, 2014, doi:<a href=\"https://doi.org/10.1371/journal.pone.0106247\">10.1371/journal.pone.0106247</a>.","ama":"Grabowska A, Wywiał E, Dunin Horkawicz S, et al. Functional and bioinformatics analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA. <i>PLoS One</i>. 2014;9(9). doi:<a href=\"https://doi.org/10.1371/journal.pone.0106247\">10.1371/journal.pone.0106247</a>"}},{"acknowledgement":"This work was supported in part by a Grant-in-Aid for Scientific Research on Innovative Areas (Comprehensive Brain Science Network) and (B) 17330153, from the Ministry of Education, Culture, Sports, Science and Technology of Japan.","title":"Functional deficiency of MHC class i enhances LTP and abolishes LTD in the nucleus accumbens of mice","abstract":[{"lang":"eng","text":"Major histocompatibility complex class I (MHCI) molecules were recently identified as novel regulators of synaptic plasticity. These molecules are expressed in various brain areas, especially in regions undergoing activity-dependent synaptic plasticity, but their role in the nucleus accumbens (NAc) is unknown. In this study, we investigated the effects of genetic disruption of MHCI function, through deletion of β2-microblobulin, which causes lack of cell surface expression of MHCI. First, we confirmed that MHCI molecules are expressed in the NAc core in wild-type mice. Second, we performed electrophysiological recordings with NAc core slices from wild-type and β2-microglobulin knock-out mice lacking cell surface expression of MHCI. We found that low frequency stimulation induced long-term depression in wild-type but not knock-out mice, whereas high frequency stimulation induced long-term potentiation in both genotypes, with a larger magnitude in knock-out mice. Furthermore, we demonstrated that knock-out mice showed more persistent behavioral sensitization to cocaine, which is a NAc-related behavior. Using this model, we analyzed the density of total AMPA receptors and their subunits GluR1 and GluR2 in the NAc core, by SDS-digested freeze-fracture replica labeling. After repeated cocaine exposure, the density of GluR1 was increased, but there was no change in total AMPA receptors and GluR2 levels in wildtype mice. In contrast, following repeated cocaine exposure, increased densities of total AMPA receptors, GluR1 and GluR2 were observed in knock-out mice. These results indicate that functional deficiency of MHCI enhances synaptic potentiation, induced by electrical and pharmacological stimulation."}],"status":"public","issue":"9","publisher":"Public Library of Science","doi":"10.1371/journal.pone.0107099","publication_status":"published","citation":{"apa":"Edamura, M., Murakami, G., Meng, H., Itakura, M., Shigemoto, R., Fukuda, A., &#38; Nakahara, D. (2014). Functional deficiency of MHC class i enhances LTP and abolishes LTD in the nucleus accumbens of mice. <i>PLoS One</i>. Public Library of Science. <a href=\"https://doi.org/10.1371/journal.pone.0107099\">https://doi.org/10.1371/journal.pone.0107099</a>","chicago":"Edamura, Mitsuhiro, Gen Murakami, Hongrui Meng, Makoto Itakura, Ryuichi Shigemoto, Atsuo Fukuda, and Daiichiro Nakahara. “Functional Deficiency of MHC Class i Enhances LTP and Abolishes LTD in the Nucleus Accumbens of Mice.” <i>PLoS One</i>. Public Library of Science, 2014. <a href=\"https://doi.org/10.1371/journal.pone.0107099\">https://doi.org/10.1371/journal.pone.0107099</a>.","ieee":"M. Edamura <i>et al.</i>, “Functional deficiency of MHC class i enhances LTP and abolishes LTD in the nucleus accumbens of mice,” <i>PLoS One</i>, vol. 9, no. 9. Public Library of Science, 2014.","short":"M. Edamura, G. Murakami, H. Meng, M. Itakura, R. Shigemoto, A. Fukuda, D. Nakahara, PLoS One 9 (2014).","ista":"Edamura M, Murakami G, Meng H, Itakura M, Shigemoto R, Fukuda A, Nakahara D. 2014. Functional deficiency of MHC class i enhances LTP and abolishes LTD in the nucleus accumbens of mice. PLoS One. 9(9), e107099.","mla":"Edamura, Mitsuhiro, et al. “Functional Deficiency of MHC Class i Enhances LTP and Abolishes LTD in the Nucleus Accumbens of Mice.” <i>PLoS One</i>, vol. 9, no. 9, e107099, Public Library of Science, 2014, doi:<a href=\"https://doi.org/10.1371/journal.pone.0107099\">10.1371/journal.pone.0107099</a>.","ama":"Edamura M, Murakami G, Meng H, et al. Functional deficiency of MHC class i enhances LTP and abolishes LTD in the nucleus accumbens of mice. <i>PLoS One</i>. 2014;9(9). doi:<a href=\"https://doi.org/10.1371/journal.pone.0107099\">10.1371/journal.pone.0107099</a>"},"volume":9,"tmp":{"image":"/images/cc_by.png","short":"CC BY (4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"year":"2014","date_updated":"2025-09-29T13:04:38Z","file":[{"file_size":6262085,"checksum":"1f3be936be93114596d61ba44cacee69","relation":"main_file","access_level":"open_access","content_type":"application/pdf","date_created":"2018-12-12T10:09:01Z","file_id":"4724","date_updated":"2020-07-14T12:45:20Z","file_name":"IST-2016-439-v1+1_journal.pone.0107099.pdf","creator":"system"}],"isi":1,"oa":1,"publication":"PLoS One","oa_version":"Published Version","file_date_updated":"2020-07-14T12:45:20Z","intvolume":"         9","_id":"1895","month":"09","date_created":"2018-12-11T11:54:35Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","department":[{"_id":"RySh"}],"type":"journal_article","article_processing_charge":"No","day":"30","scopus_import":"1","author":[{"full_name":"Edamura, Mitsuhiro","first_name":"Mitsuhiro","last_name":"Edamura"},{"full_name":"Murakami, Gen","first_name":"Gen","last_name":"Murakami"},{"full_name":"Meng, Hongrui","first_name":"Hongrui","last_name":"Meng"},{"last_name":"Itakura","first_name":"Makoto","full_name":"Itakura, Makoto"},{"id":"499F3ABC-F248-11E8-B48F-1D18A9856A87","first_name":"Ryuichi","full_name":"Shigemoto, Ryuichi","orcid":"0000-0001-8761-9444","last_name":"Shigemoto"},{"last_name":"Fukuda","full_name":"Fukuda, Atsuo","first_name":"Atsuo"},{"first_name":"Daiichiro","full_name":"Nakahara, Daiichiro","last_name":"Nakahara"}],"publist_id":"5200","pubrep_id":"439","article_number":"e107099","language":[{"iso":"eng"}],"date_published":"2014-09-30T00:00:00Z","external_id":{"isi":["000343671700028"]},"has_accepted_license":"1","ddc":["570"],"fulldoi":"https://doi.org/10.1371/journal.pone.0107099"},{"main_file_link":[{"open_access":"1","url":"http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4145132/"}],"scopus_import":"1","author":[{"last_name":"Naramoto","full_name":"Naramoto, Satoshi","first_name":"Satoshi"},{"last_name":"Otegui","first_name":"Marisa","full_name":"Otegui, Marisa"},{"first_name":"Natsumaro","full_name":"Kutsuna, Natsumaro","last_name":"Kutsuna"},{"full_name":"De Rycke, Riet","first_name":"Riet","last_name":"De Rycke"},{"last_name":"Dainobu","full_name":"Dainobu, Tomoko","first_name":"Tomoko"},{"full_name":"Karampelias, Michael","first_name":"Michael","last_name":"Karampelias"},{"last_name":"Fujimoto","full_name":"Fujimoto, Masaru","first_name":"Masaru"},{"first_name":"Elena","full_name":"Feraru, Elena","last_name":"Feraru"},{"first_name":"Daisuke","full_name":"Miki, Daisuke","last_name":"Miki"},{"full_name":"Fukuda, Hiroo","first_name":"Hiroo","last_name":"Fukuda"},{"last_name":"Nakano","full_name":"Nakano, Akihiko","first_name":"Akihiko"},{"orcid":"0000-0002-8302-7596","last_name":"Friml","id":"4159519E-F248-11E8-B48F-1D18A9856A87","first_name":"Jirí","full_name":"Friml, Jirí"}],"publist_id":"5199","page":"3062 - 3076","type":"journal_article","article_processing_charge":"No","day":"01","fulldoi":"https://doi.org/10.1105/tpc.114.125880","language":[{"iso":"eng"}],"external_id":{"isi":["000342076200027"]},"date_published":"2014-07-01T00:00:00Z","doi":"10.1105/tpc.114.125880","publication_status":"published","citation":{"mla":"Naramoto, Satoshi, et al. “Insights into the Localization and Function of the Membrane Trafficking Regulator GNOM ARF-GEF at the Golgi Apparatus in Arabidopsis.” <i>Plant Cell</i>, vol. 26, no. 7, American Society of Plant Biologists, 2014, pp. 3062–76, doi:<a href=\"https://doi.org/10.1105/tpc.114.125880\">10.1105/tpc.114.125880</a>.","ama":"Naramoto S, Otegui M, Kutsuna N, et al. Insights into the localization and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis. <i>Plant Cell</i>. 2014;26(7):3062-3076. doi:<a href=\"https://doi.org/10.1105/tpc.114.125880\">10.1105/tpc.114.125880</a>","ista":"Naramoto S, Otegui M, Kutsuna N, De Rycke R, Dainobu T, Karampelias M, Fujimoto M, Feraru E, Miki D, Fukuda H, Nakano A, Friml J. 2014. Insights into the localization and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis. Plant Cell. 26(7), 3062–3076.","ieee":"S. Naramoto <i>et al.</i>, “Insights into the localization and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis,” <i>Plant Cell</i>, vol. 26, no. 7. American Society of Plant Biologists, pp. 3062–3076, 2014.","short":"S. Naramoto, M. Otegui, N. Kutsuna, R. De Rycke, T. Dainobu, M. Karampelias, M. Fujimoto, E. Feraru, D. Miki, H. Fukuda, A. Nakano, J. Friml, Plant Cell 26 (2014) 3062–3076.","apa":"Naramoto, S., Otegui, M., Kutsuna, N., De Rycke, R., Dainobu, T., Karampelias, M., … Friml, J. (2014). Insights into the localization and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis. <i>Plant Cell</i>. American Society of Plant Biologists. <a href=\"https://doi.org/10.1105/tpc.114.125880\">https://doi.org/10.1105/tpc.114.125880</a>","chicago":"Naramoto, Satoshi, Marisa Otegui, Natsumaro Kutsuna, Riet De Rycke, Tomoko Dainobu, Michael Karampelias, Masaru Fujimoto, et al. “Insights into the Localization and Function of the Membrane Trafficking Regulator GNOM ARF-GEF at the Golgi Apparatus in Arabidopsis.” <i>Plant Cell</i>. American Society of Plant Biologists, 2014. <a href=\"https://doi.org/10.1105/tpc.114.125880\">https://doi.org/10.1105/tpc.114.125880</a>."},"volume":26,"year":"2014","acknowledgement":"This work was supported by the Odysseus Program of the Research Foundation-Flanders (J.F.).","title":"Insights into the localization and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis","abstract":[{"text":"GNOM is one of the most characterized membrane trafficking regulators in plants, with crucial roles in development. GNOM encodes an ARF-guanine nucleotide exchange factor (ARF-GEF) that activates small GTPases of the ARF (ADP ribosylation factor) class to mediate vesicle budding at endomembranes. The crucial role of GNOM in recycling of PIN auxin transporters and other proteins to the plasma membrane was identified in studies using the ARF-GEF inhibitor brefeldin A (BFA). GNOM, the most prominent regulator of recycling in plants, has been proposed to act and localize at so far elusive recycling endosomes. Here, we report the GNOM localization in context of its cellular function in Arabidopsis thaliana. State-of-the-art imaging, pharmacological interference, and ultrastructure analysis show that GNOM predominantly localizes to Golgi apparatus. Super-resolution confocal live imaging microscopy identified GNOM and its closest homolog GNOM-like 1 at distinct subdomains on Golgi cisternae. Short-term BFA treatment stabilizes GNOM at the Golgi apparatus, whereas prolonged exposures results in GNOM translocation to trans-Golgi network (TGN)/early endosomes (EEs). Malformed TGN/EE in gnom mutants suggests a role for GNOM in maintaining TGN/EE function. Our results redefine the subcellular action of GNOM and reevaluate the identity and function of recycling endosomes in plants.","lang":"eng"}],"status":"public","issue":"7","publisher":"American Society of Plant Biologists","_id":"1897","month":"07","date_created":"2018-12-11T11:54:36Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","department":[{"_id":"JiFr"}],"date_updated":"2025-09-29T13:04:06Z","isi":1,"oa":1,"oa_version":"Submitted Version","publication":"Plant Cell","intvolume":"        26"},{"doi":"10.1016/j.neuron.2014.08.036","volume":84,"year":"2014","publication_status":"published","citation":{"ieee":"A. Ritzau Jost <i>et al.</i>, “Ultrafast action potentials mediate kilohertz signaling at a central synapse,” <i>Neuron</i>, vol. 84, no. 1. Elsevier, pp. 152–163, 2014.","short":"A. Ritzau Jost, I. Delvendahl, A. Rings, N. Byczkowicz, H. Harada, R. Shigemoto, J. Hirrlinger, J. Eilers, S. Hallermann, Neuron 84 (2014) 152–163.","apa":"Ritzau Jost, A., Delvendahl, I., Rings, A., Byczkowicz, N., Harada, H., Shigemoto, R., … Hallermann, S. (2014). Ultrafast action potentials mediate kilohertz signaling at a central synapse. <i>Neuron</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.neuron.2014.08.036\">https://doi.org/10.1016/j.neuron.2014.08.036</a>","chicago":"Ritzau Jost, Andreas, Igor Delvendahl, Annika Rings, Niklas Byczkowicz, Harumi Harada, Ryuichi Shigemoto, Johannes Hirrlinger, Jens Eilers, and Stefan Hallermann. “Ultrafast Action Potentials Mediate Kilohertz Signaling at a Central Synapse.” <i>Neuron</i>. Elsevier, 2014. <a href=\"https://doi.org/10.1016/j.neuron.2014.08.036\">https://doi.org/10.1016/j.neuron.2014.08.036</a>.","mla":"Ritzau Jost, Andreas, et al. “Ultrafast Action Potentials Mediate Kilohertz Signaling at a Central Synapse.” <i>Neuron</i>, vol. 84, no. 1, Elsevier, 2014, pp. 152–63, doi:<a href=\"https://doi.org/10.1016/j.neuron.2014.08.036\">10.1016/j.neuron.2014.08.036</a>.","ama":"Ritzau Jost A, Delvendahl I, Rings A, et al. Ultrafast action potentials mediate kilohertz signaling at a central synapse. <i>Neuron</i>. 2014;84(1):152-163. doi:<a href=\"https://doi.org/10.1016/j.neuron.2014.08.036\">10.1016/j.neuron.2014.08.036</a>","ista":"Ritzau Jost A, Delvendahl I, Rings A, Byczkowicz N, Harada H, Shigemoto R, Hirrlinger J, Eilers J, Hallermann S. 2014. Ultrafast action potentials mediate kilohertz signaling at a central synapse. Neuron. 84(1), 152–163."},"title":"Ultrafast action potentials mediate kilohertz signaling at a central synapse","abstract":[{"text":"Fast synaptic transmission is important for rapid information processing. To explore the maximal rate of neuronal signaling and to analyze the presynaptic mechanisms, we focused on the input layer of the cerebellar cortex, where exceptionally high action potential (AP) frequencies have been reported invivo. With paired recordings between presynaptic cerebellar mossy fiber boutons and postsynaptic granule cells, we demonstrate reliable neurotransmission upto ~1 kHz. Presynaptic APs are ultrafast, with ~100μs half-duration. Both Kv1 and Kv3 potassium channels mediate the fast repolarization, rapidly inactivating sodium channels ensure metabolic efficiency, and little AP broadening occurs during bursts of up to 1.5 kHz. Presynaptic Cav2.1 (P/Q-type) calcium channels open efficiently during ultrafast APs. Furthermore, a subset of synaptic vesicles is tightly coupled to Ca2+ channels, and vesicles are rapidly recruited to the release site. These data reveal mechanisms of presynaptic AP generation and transmitter release underlying neuronal kHz signaling.","lang":"eng"}],"status":"public","issue":"1","publisher":"Elsevier","quality_controlled":"1","month":"10","_id":"1898","department":[{"_id":"RySh"}],"date_created":"2018-12-11T11:54:36Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_updated":"2025-09-29T13:03:03Z","isi":1,"intvolume":"        84","publication":"Neuron","oa_version":"None","author":[{"full_name":"Ritzau Jost, Andreas","first_name":"Andreas","last_name":"Ritzau Jost"},{"first_name":"Igor","full_name":"Delvendahl, Igor","last_name":"Delvendahl"},{"full_name":"Rings, Annika","first_name":"Annika","last_name":"Rings"},{"first_name":"Niklas","full_name":"Byczkowicz, Niklas","last_name":"Byczkowicz"},{"orcid":"0000-0001-7429-7896","last_name":"Harada","id":"2E55CDF2-F248-11E8-B48F-1D18A9856A87","first_name":"Harumi","full_name":"Harada, Harumi"},{"orcid":"0000-0001-8761-9444","last_name":"Shigemoto","first_name":"Ryuichi","id":"499F3ABC-F248-11E8-B48F-1D18A9856A87","full_name":"Shigemoto, Ryuichi"},{"full_name":"Hirrlinger, Johannes","first_name":"Johannes","last_name":"Hirrlinger"},{"last_name":"Eilers","first_name":"Jens","full_name":"Eilers, Jens"},{"full_name":"Hallermann, Stefan","first_name":"Stefan","last_name":"Hallermann"}],"publist_id":"5197","scopus_import":"1","page":"152 - 163","article_processing_charge":"No","type":"journal_article","day":"01","fulldoi":"https://doi.org/10.1016/j.neuron.2014.08.036","language":[{"iso":"eng"}],"date_published":"2014-10-01T00:00:00Z","external_id":{"isi":["000342502800017"]}},{"title":"Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN","abstract":[{"text":"Asymmetric cell divisions allow stem cells to balance proliferation and differentiation. During embryogenesis, murine epidermis expands rapidly from a single layer of unspecified basal layer progenitors to a stratified, differentiated epithelium. Morphogenesis involves perpendicular (asymmetric) divisions and the spindle orientation protein LGN, but little is known about how the apical localization of LGN is regulated. Here, we combine conventional genetics and lentiviral-mediated in vivo RNAi to explore the functions of the LGN-interacting proteins Par3, mInsc and Gα i3. Whereas loss of each gene alone leads to randomized division angles, combined loss of Gnai3 and mInsc causes a phenotype of mostly planar divisions, akin to loss of LGN. These findings lend experimental support for the hitherto untested model that Par3-mInsc and Gα i3 act cooperatively to polarize LGN and promote perpendicular divisions. Finally, we uncover a developmental switch between delamination-driven early stratification and spindle-orientation-dependent differentiation that occurs around E15, revealing a two-step mechanism underlying epidermal maturation.","lang":"eng"}],"quality_controlled":"1","publisher":"Nature Publishing Group","status":"public","issue":"8","doi":"10.1038/ncb3001","citation":{"ieee":"S. Williams, L. Ratliff, M. P. Postiglione, J. Knoblich, and E. Fuchs, “Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN,” <i>Nature Cell Biology</i>, vol. 16, no. 8. Nature Publishing Group, pp. 758–769, 2014.","short":"S. Williams, L. Ratliff, M.P. Postiglione, J. Knoblich, E. Fuchs, Nature Cell Biology 16 (2014) 758–769.","apa":"Williams, S., Ratliff, L., Postiglione, M. P., Knoblich, J., &#38; Fuchs, E. (2014). Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN. <i>Nature Cell Biology</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/ncb3001\">https://doi.org/10.1038/ncb3001</a>","chicago":"Williams, Scott, Lyndsay Ratliff, Maria P Postiglione, Juergen Knoblich, and Elaine Fuchs. “Par3-MInsc and Gα I3 Cooperate to Promote Oriented Epidermal Cell Divisions through LGN.” <i>Nature Cell Biology</i>. Nature Publishing Group, 2014. <a href=\"https://doi.org/10.1038/ncb3001\">https://doi.org/10.1038/ncb3001</a>.","mla":"Williams, Scott, et al. “Par3-MInsc and Gα I3 Cooperate to Promote Oriented Epidermal Cell Divisions through LGN.” <i>Nature Cell Biology</i>, vol. 16, no. 8, Nature Publishing Group, 2014, pp. 758–69, doi:<a href=\"https://doi.org/10.1038/ncb3001\">10.1038/ncb3001</a>.","ama":"Williams S, Ratliff L, Postiglione MP, Knoblich J, Fuchs E. Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN. <i>Nature Cell Biology</i>. 2014;16(8):758-769. doi:<a href=\"https://doi.org/10.1038/ncb3001\">10.1038/ncb3001</a>","ista":"Williams S, Ratliff L, Postiglione MP, Knoblich J, Fuchs E. 2014. Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN. Nature Cell Biology. 16(8), 758–769."},"publication_status":"published","volume":16,"year":"2014","oa":1,"isi":1,"date_updated":"2025-09-29T12:32:05Z","oa_version":"Submitted Version","publication":"Nature Cell Biology","intvolume":"        16","_id":"1899","month":"07","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:36Z","article_type":"original","department":[{"_id":"SiHi"}],"type":"journal_article","article_processing_charge":"No","day":"13","scopus_import":"1","main_file_link":[{"url":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4159251/","open_access":"1"}],"publist_id":"5196","author":[{"first_name":"Scott","full_name":"Williams, Scott","last_name":"Williams"},{"last_name":"Ratliff","first_name":"Lyndsay","full_name":"Ratliff, Lyndsay"},{"last_name":"Postiglione","id":"2C67902A-F248-11E8-B48F-1D18A9856A87","first_name":"Maria P","full_name":"Postiglione, Maria P"},{"last_name":"Knoblich","first_name":"Juergen","full_name":"Knoblich, Juergen"},{"full_name":"Fuchs, Elaine","first_name":"Elaine","last_name":"Fuchs"}],"page":"758 - 769","language":[{"iso":"eng"}],"external_id":{"pmid":["25016959"],"isi":["000339904900008"]},"date_published":"2014-07-13T00:00:00Z","pmid":1,"fulldoi":"https://doi.org/10.1038/ncb3001"},{"isi":1,"date_updated":"2025-09-29T12:31:29Z","oa_version":"None","publication":"Nature Cell Biology","intvolume":"        16","_id":"1900","month":"01","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:37Z","department":[{"_id":"CaHe"}],"abstract":[{"text":"Epithelial cell layers need to be tightly regulated to maintain their integrity and correct function. Cell integration into epithelial sheets is now shown to depend on the N-WASP-regulated stabilization of cortical F-actin, which generates distinct patterns of apical-lateral contractility at E-cadherin-based cell-cell junctions.","lang":"eng"}],"title":"Lateral junction dynamics lead the way out","quality_controlled":"1","status":"public","issue":"2","publisher":"Nature Publishing Group","doi":"10.1038/ncb2913","citation":{"ama":"Behrndt M, Heisenberg C-PJ. Lateral junction dynamics lead the way out. <i>Nature Cell Biology</i>. 2014;16(2):127-129. doi:<a href=\"https://doi.org/10.1038/ncb2913\">10.1038/ncb2913</a>","mla":"Behrndt, Martin, and Carl-Philipp J. Heisenberg. “Lateral Junction Dynamics Lead the Way Out.” <i>Nature Cell Biology</i>, vol. 16, no. 2, Nature Publishing Group, 2014, pp. 127–29, doi:<a href=\"https://doi.org/10.1038/ncb2913\">10.1038/ncb2913</a>.","ista":"Behrndt M, Heisenberg C-PJ. 2014. Lateral junction dynamics lead the way out. Nature Cell Biology. 16(2), 127–129.","short":"M. Behrndt, C.-P.J. Heisenberg, Nature Cell Biology 16 (2014) 127–129.","ieee":"M. Behrndt and C.-P. J. Heisenberg, “Lateral junction dynamics lead the way out,” <i>Nature Cell Biology</i>, vol. 16, no. 2. Nature Publishing Group, pp. 127–129, 2014.","chicago":"Behrndt, Martin, and Carl-Philipp J Heisenberg. “Lateral Junction Dynamics Lead the Way Out.” <i>Nature Cell Biology</i>. Nature Publishing Group, 2014. <a href=\"https://doi.org/10.1038/ncb2913\">https://doi.org/10.1038/ncb2913</a>.","apa":"Behrndt, M., &#38; Heisenberg, C.-P. J. (2014). Lateral junction dynamics lead the way out. <i>Nature Cell Biology</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/ncb2913\">https://doi.org/10.1038/ncb2913</a>"},"publication_status":"published","volume":16,"year":"2014","language":[{"iso":"eng"}],"date_published":"2014-01-31T00:00:00Z","external_id":{"isi":["000331161400001"]},"fulldoi":"https://doi.org/10.1038/ncb2913","type":"journal_article","article_processing_charge":"No","day":"31","scopus_import":"1","author":[{"full_name":"Behrndt, Martin","id":"3ECECA3A-F248-11E8-B48F-1D18A9856A87","first_name":"Martin","last_name":"Behrndt"},{"last_name":"Heisenberg","orcid":"0000-0002-0912-4566","id":"39427864-F248-11E8-B48F-1D18A9856A87","first_name":"Carl-Philipp J","full_name":"Heisenberg, Carl-Philipp J"}],"publist_id":"5195","corr_author":"1","page":"127 - 129"},{"publist_id":"5194","author":[{"full_name":"Tian, Huiyu","first_name":"Huiyu","last_name":"Tian"},{"first_name":"Krzysztof T","full_name":"Wabnik, Krzysztof T","last_name":"Wabnik"},{"last_name":"Niu","full_name":"Niu, Tiantian","first_name":"Tiantian"},{"last_name":"Li","full_name":"Li, Hongjiang","first_name":"Hongjiang"},{"last_name":"Yu","first_name":"Qianqian","full_name":"Yu, Qianqian"},{"last_name":"Pollmann","full_name":"Pollmann, Stephan","first_name":"Stephan"},{"last_name":"Vanneste","full_name":"Vanneste, Steffen","first_name":"Steffen"},{"last_name":"Govaerts","first_name":"Willy","full_name":"Govaerts, Willy"},{"first_name":"Jakub","full_name":"Rolčík, Jakub","last_name":"Rolčík"},{"first_name":"Markus","full_name":"Geisler, Markus","last_name":"Geisler"},{"orcid":"0000-0002-8302-7596","last_name":"Friml","first_name":"Jirí","id":"4159519E-F248-11E8-B48F-1D18A9856A87","full_name":"Friml, Jirí"},{"last_name":"Ding","full_name":"Ding, Zhaojun","first_name":"Zhaojun"}],"scopus_import":"1","page":"277 - 289","article_processing_charge":"No","type":"journal_article","day":"01","fulldoi":"https://doi.org/10.1093/mp/sst118","language":[{"iso":"eng"}],"date_published":"2014-02-01T00:00:00Z","external_id":{"isi":["000330841400003"]},"doi":"10.1093/mp/sst118","year":"2014","volume":7,"publication_status":"published","citation":{"short":"H. Tian, K.T. Wabnik, T. Niu, H. Li, Q. Yu, S. Pollmann, S. Vanneste, W. Govaerts, J. Rolčík, M. Geisler, J. Friml, Z. Ding, Molecular Plant 7 (2014) 277–289.","ieee":"H. Tian <i>et al.</i>, “WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for the patterning of root stem cell niches in arabidopsis,” <i>Molecular Plant</i>, vol. 7, no. 2. Oxford University Press, pp. 277–289, 2014.","chicago":"Tian, Huiyu, Krzysztof T Wabnik, Tiantian Niu, Hongjiang Li, Qianqian Yu, Stephan Pollmann, Steffen Vanneste, et al. “WOX5-IAA17 Feedback Circuit-Mediated Cellular Auxin Response Is Crucial for the Patterning of Root Stem Cell Niches in Arabidopsis.” <i>Molecular Plant</i>. Oxford University Press, 2014. <a href=\"https://doi.org/10.1093/mp/sst118\">https://doi.org/10.1093/mp/sst118</a>.","apa":"Tian, H., Wabnik, K. T., Niu, T., Li, H., Yu, Q., Pollmann, S., … Ding, Z. (2014). WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for the patterning of root stem cell niches in arabidopsis. <i>Molecular Plant</i>. Oxford University Press. <a href=\"https://doi.org/10.1093/mp/sst118\">https://doi.org/10.1093/mp/sst118</a>","ama":"Tian H, Wabnik KT, Niu T, et al. WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for the patterning of root stem cell niches in arabidopsis. <i>Molecular Plant</i>. 2014;7(2):277-289. doi:<a href=\"https://doi.org/10.1093/mp/sst118\">10.1093/mp/sst118</a>","mla":"Tian, Huiyu, et al. “WOX5-IAA17 Feedback Circuit-Mediated Cellular Auxin Response Is Crucial for the Patterning of Root Stem Cell Niches in Arabidopsis.” <i>Molecular Plant</i>, vol. 7, no. 2, Oxford University Press, 2014, pp. 277–89, doi:<a href=\"https://doi.org/10.1093/mp/sst118\">10.1093/mp/sst118</a>.","ista":"Tian H, Wabnik KT, Niu T, Li H, Yu Q, Pollmann S, Vanneste S, Govaerts W, Rolčík J, Geisler M, Friml J, Ding Z. 2014. WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for the patterning of root stem cell niches in arabidopsis. Molecular Plant. 7(2), 277–289."},"abstract":[{"text":"In plants, the patterning of stem cell-enriched meristems requires a graded auxin response maximum that emerges from the concerted action of polar auxin transport, auxin biosynthesis, auxin metabolism, and cellular auxin response machinery. However, mechanisms underlying this auxin response maximum-mediated root stem cell maintenance are not fully understood. Here, we present unexpected evidence that WUSCHEL-RELATED HOMEOBOX 5 (WOX5) transcription factor modulates expression of auxin biosynthetic genes in the quiescent center (QC) of the root and thus provides a robust mechanism for the maintenance of auxin response maximum in the root tip. This WOX5 action is balanced through the activity of indole-3-acetic acid 17 (IAA17) auxin response repressor. Our combined genetic, cell biology, and computational modeling studies revealed a previously uncharacterized feedback loop linking WOX5-mediated auxin production to IAA17-dependent repression of auxin responses. This WOX5-IAA17 feedback circuit further assures the maintenance of auxin response maximum in the root tip and thereby contributes to the maintenance of distal stem cell (DSC) populations. Our experimental studies and in silico computer simulations both demonstrate that the WOX5-IAA17 feedback circuit is essential for the maintenance of auxin gradient in the root tip and the auxin-mediated root DSC differentiation.","lang":"eng"}],"title":"WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for the patterning of root stem cell niches in arabidopsis","acknowledgement":"This work was supported by funding from the projects CZ.1.07/2.3.00/20.0043 and CZ.1.05/1.1.00/02.0068 (to CEITEC, Central European Institute of Technology) and the Odysseus program of the Research Foundation-Flanders to J.F\r\n","publisher":"Oxford University Press","issue":"2","status":"public","month":"02","_id":"1901","department":[{"_id":"JiFr"}],"user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","date_created":"2018-12-11T11:54:37Z","date_updated":"2025-09-29T12:30:56Z","isi":1,"intvolume":"         7","oa_version":"None","publication":"Molecular Plant"},{"fulldoi":"https://doi.org/10.1093/molbev/mst187","ddc":["570"],"date_published":"2014-01-01T00:00:00Z","external_id":{"pmid":["24170494"],"isi":["000329253200022"]},"pmid":1,"language":[{"iso":"eng"}],"page":"232 - 238","publist_id":"5193","author":[{"last_name":"Hall","full_name":"Hall, Barry","first_name":"Barry"},{"last_name":"Acar","orcid":"0000-0003-1986-9753","id":"2DDF136A-F248-11E8-B48F-1D18A9856A87","first_name":"Hande","full_name":"Acar, Hande"},{"first_name":"Anna","full_name":"Nandipati, Anna","last_name":"Nandipati"},{"last_name":"Barlow","first_name":"Miriam","full_name":"Barlow, Miriam"}],"publication_identifier":{"eissn":["1537-1719"],"issn":["0737-4038"]},"scopus_import":"1","main_file_link":[{"url":"https://doi.org/10.1093/molbev/mst187","open_access":"1"}],"day":"01","article_processing_charge":"No","type":"journal_article","article_type":"original","department":[{"_id":"JoBo"}],"date_created":"2018-12-11T11:54:37Z","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","month":"01","_id":"1902","intvolume":"        31","publication":"Molecular Biology and Evolution","oa_version":"Published Version","oa":1,"date_updated":"2026-06-18T18:03:30Z","isi":1,"volume":31,"year":"2014","publication_status":"published","OA_place":"publisher","citation":{"short":"B. Hall, H. Acar, A. Nandipati, M. Barlow, Molecular Biology and Evolution 31 (2014) 232–238.","ieee":"B. Hall, H. Acar, A. Nandipati, and M. Barlow, “Growth rates made easy,” <i>Molecular Biology and Evolution</i>, vol. 31, no. 1. Oxford University Press, pp. 232–238, 2014.","chicago":"Hall, Barry, Hande Acar, Anna Nandipati, and Miriam Barlow. “Growth Rates Made Easy.” <i>Molecular Biology and Evolution</i>. Oxford University Press, 2014. <a href=\"https://doi.org/10.1093/molbev/mst187\">https://doi.org/10.1093/molbev/mst187</a>.","apa":"Hall, B., Acar, H., Nandipati, A., &#38; Barlow, M. (2014). Growth rates made easy. <i>Molecular Biology and Evolution</i>. Oxford University Press. <a href=\"https://doi.org/10.1093/molbev/mst187\">https://doi.org/10.1093/molbev/mst187</a>","ama":"Hall B, Acar H, Nandipati A, Barlow M. Growth rates made easy. <i>Molecular Biology and Evolution</i>. 2014;31(1):232-238. doi:<a href=\"https://doi.org/10.1093/molbev/mst187\">10.1093/molbev/mst187</a>","mla":"Hall, Barry, et al. “Growth Rates Made Easy.” <i>Molecular Biology and Evolution</i>, vol. 31, no. 1, Oxford University Press, 2014, pp. 232–38, doi:<a href=\"https://doi.org/10.1093/molbev/mst187\">10.1093/molbev/mst187</a>.","ista":"Hall B, Acar H, Nandipati A, Barlow M. 2014. Growth rates made easy. Molecular Biology and Evolution. 31(1), 232–238."},"OA_type":"free access","doi":"10.1093/molbev/mst187","publisher":"Oxford University Press","issue":"1","status":"public","quality_controlled":"1","title":"Growth rates made easy","abstract":[{"lang":"eng","text":"In the 1960s-1980s, determination of bacterial growth rates was an important tool in microbial genetics, biochemistry, molecular biology, and microbial physiology. The exciting technical developments of the 1990s and the 2000s eclipsed that tool; as a result, many investigators today lack experience with growth rate measurements. Recently, investigators in a number of areas have started to use measurements of bacterial growth rates for a variety of purposes. Those measurements have been greatly facilitated by the availability of microwell plate readers that permit the simultaneous measurements on up to 384 different cultures. Only the exponential (logarithmic) portions of the resulting growth curves are useful for determining growth rates, and manual determination of that portion and calculation of growth rates can be tedious for high-throughput purposes. Here, we introduce the program GrowthRates that uses plate reader output files to automatically determine the exponential portion of the curve and to automatically calculate the growth rate, the maximum culture density, and the duration of the growth lag phase. GrowthRates is freely available for Macintosh, Windows, and Linux.We discuss the effects of culture volume, the classical bacterial growth curve, and the differences between determinations in rich media and minimal (mineral salts) media. This protocol covers calibration of the plate reader, growth of culture inocula for both rich and minimal media, and experimental setup. As a guide to reliability, we report typical day-to-day variation in growth rates and variation within experiments with respect to position of wells within the plates."}]},{"doi":"10.1111/jeb.12370","OA_type":"free access","publication_status":"published","citation":{"chicago":"Tobler, Michael, Martin Plath, Rüdiger Riesch, Ingo Schlupp, Anna V Grasse, Gopi Munimanda, C Setzer, Dustin Penn, and Yoshan Moodley. “Selection from Parasites Favours Immunogenetic Diversity but Not Divergence among Locally Adapted Host Populations.” <i>Journal of Evolutionary Biology</i>. Wiley, 2014. <a href=\"https://doi.org/10.1111/jeb.12370\">https://doi.org/10.1111/jeb.12370</a>.","apa":"Tobler, M., Plath, M., Riesch, R., Schlupp, I., Grasse, A. V., Munimanda, G., … Moodley, Y. (2014). Selection from parasites favours immunogenetic diversity but not divergence among locally adapted host populations. <i>Journal of Evolutionary Biology</i>. Wiley. <a href=\"https://doi.org/10.1111/jeb.12370\">https://doi.org/10.1111/jeb.12370</a>","short":"M. Tobler, M. Plath, R. Riesch, I. Schlupp, A.V. Grasse, G. Munimanda, C. Setzer, D. Penn, Y. Moodley, Journal of Evolutionary Biology 27 (2014) 960–974.","ieee":"M. Tobler <i>et al.</i>, “Selection from parasites favours immunogenetic diversity but not divergence among locally adapted host populations,” <i>Journal of Evolutionary Biology</i>, vol. 27, no. 5. Wiley, pp. 960–974, 2014.","ista":"Tobler M, Plath M, Riesch R, Schlupp I, Grasse AV, Munimanda G, Setzer C, Penn D, Moodley Y. 2014. Selection from parasites favours immunogenetic diversity but not divergence among locally adapted host populations. Journal of Evolutionary Biology. 27(5), 960–974.","ama":"Tobler M, Plath M, Riesch R, et al. Selection from parasites favours immunogenetic diversity but not divergence among locally adapted host populations. <i>Journal of Evolutionary Biology</i>. 2014;27(5):960-974. doi:<a href=\"https://doi.org/10.1111/jeb.12370\">10.1111/jeb.12370</a>","mla":"Tobler, Michael, et al. “Selection from Parasites Favours Immunogenetic Diversity but Not Divergence among Locally Adapted Host Populations.” <i>Journal of Evolutionary Biology</i>, vol. 27, no. 5, Wiley, 2014, pp. 960–74, doi:<a href=\"https://doi.org/10.1111/jeb.12370\">10.1111/jeb.12370</a>."},"year":"2014","volume":27,"acknowledgement":"This study was funded by grants from the National Science Foundation (NSF) to MT (IOS-1121832) and IS (DEB-0743406) and from the German Science Foundation (DFG; PL 470/1-2) and ‘LOEWE − Landesoffensive zur Entwicklung wissenschaftlich-ökonomischer Exzellenz’ of Hesse's Ministry of Higher Education, Research, and the Arts, to MP.","abstract":[{"lang":"eng","text":"The unprecedented polymorphism in the major histocompatibility complex (MHC) genes is thought to be maintained by balancing selection from parasites. However, do parasites also drive divergence at MHC loci between host populations, or do the effects of balancing selection maintain similarities among populations? We examined MHC variation in populations of the livebearing fish Poecilia mexicana and characterized their parasite communities. Poecilia mexicana populations in the Cueva del Azufre system are locally adapted to darkness and the presence of toxic hydrogen sulphide, representing highly divergent ecotypes or incipient species. Parasite communities differed significantly across populations, and populations with higher parasite loads had higher levels of diversity at class II MHC genes. However, despite different parasite communities, marked divergence in adaptive traits and in neutral genetic markers, we found MHC alleles to be remarkably similar among host populations. Our findings indicate that balancing selection from parasites maintains immunogenetic diversity of hosts, but this process does not promote MHC divergence in this system. On the contrary, we suggest that balancing selection on immunogenetic loci may outweigh divergent selection causing divergence, thereby hindering host divergence and speciation. Our findings support the hypothesis that balancing selection maintains MHC similarities among lineages during and after speciation (trans-species evolution)."}],"title":"Selection from parasites favours immunogenetic diversity but not divergence among locally adapted host populations","quality_controlled":"1","issue":"5","status":"public","publisher":"Wiley","_id":"1905","month":"04","date_created":"2018-12-11T11:54:38Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","department":[{"_id":"SyCr"}],"article_type":"original","date_updated":"2025-09-29T12:28:21Z","isi":1,"oa":1,"publication":"Journal of Evolutionary Biology","oa_version":"None","intvolume":"        27","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1111/jeb.12370"}],"publication_identifier":{"eissn":["1420-9101"],"issn":["1010-061X"]},"scopus_import":"1","author":[{"last_name":"Tobler","first_name":"Michael","full_name":"Tobler, Michael"},{"first_name":"Martin","full_name":"Plath, Martin","last_name":"Plath"},{"last_name":"Riesch","first_name":"Rüdiger","full_name":"Riesch, Rüdiger"},{"last_name":"Schlupp","first_name":"Ingo","full_name":"Schlupp, Ingo"},{"last_name":"Grasse","first_name":"Anna V","id":"406F989C-F248-11E8-B48F-1D18A9856A87","full_name":"Grasse, Anna V"},{"first_name":"Gopi","full_name":"Munimanda, Gopi","last_name":"Munimanda"},{"first_name":"C","full_name":"Setzer, C","last_name":"Setzer"},{"last_name":"Penn","first_name":"Dustin","full_name":"Penn, Dustin"},{"full_name":"Moodley, Yoshan","first_name":"Yoshan","last_name":"Moodley"}],"publist_id":"5190","page":"960 - 974","type":"journal_article","article_processing_charge":"No","day":"12","fulldoi":"https://doi.org/10.1111/jeb.12370","language":[{"iso":"eng"}],"pmid":1,"external_id":{"pmid":["24725091"],"isi":["000334966800015"]},"date_published":"2014-04-12T00:00:00Z"},{"day":"09","type":"journal_article","article_processing_charge":"No","page":"1280 - 1292","pubrep_id":"573","scopus_import":"1","publist_id":"5189","author":[{"last_name":"Arikan","first_name":"Murat","full_name":"Arikan, Murat"},{"last_name":"Preiner","full_name":"Preiner, Reinhold","first_name":"Reinhold"},{"last_name":"Scheiblauer","full_name":"Scheiblauer, Claus","first_name":"Claus"},{"last_name":"Jeschke","full_name":"Jeschke, Stefan","first_name":"Stefan","id":"44D6411A-F248-11E8-B48F-1D18A9856A87"},{"last_name":"Wimmer","full_name":"Wimmer, Michael","first_name":"Michael"}],"external_id":{"isi":["000341566500006"]},"date_published":"2014-09-09T00:00:00Z","language":[{"iso":"eng"}],"has_accepted_license":"1","fulldoi":"https://doi.org/10.1109/TVCG.2014.2312011","ddc":["000"],"project":[{"name":"Deep Pictures: Creating Visual and Haptic Vector Images","grant_number":"P 24352-N23","_id":"25357BD2-B435-11E9-9278-68D0E5697425","call_identifier":"FWF"}],"publisher":"IEEE","status":"public","issue":"9","acknowledgement":"This research was supported by the Austrian Research Promotion Agency (FFG) project REPLICATE (no. 835948), the EU FP7 project HARVEST4D (no. 323567).","abstract":[{"text":"In this paper, we introduce a novel scene representation for the visualization of large-scale point clouds accompanied by a set of high-resolution photographs. Many real-world applications deal with very densely sampled point-cloud data, which are augmented with photographs that often reveal lighting variations and inaccuracies in registration. Consequently, the high-quality representation of the captured data, i.e., both point clouds and photographs together, is a challenging and time-consuming task. We propose a two-phase approach, in which the first (preprocessing) phase generates multiple overlapping surface patches and handles the problem of seamless texture generation locally for each patch. The second phase stitches these patches at render-time to produce a high-quality visualization of the data. As a result of the proposed localization of the global texturing problem, our algorithm is more than an order of magnitude faster than equivalent mesh-based texturing techniques. Furthermore, since our preprocessing phase requires only a minor fraction of the whole data set at once, we provide maximum flexibility when dealing with growing data sets.","lang":"eng"}],"title":"Large-scale point-cloud visualization through localized textured surface reconstruction","publication_status":"published","citation":{"short":"M. Arikan, R. Preiner, C. Scheiblauer, S. Jeschke, M. Wimmer, IEEE Transactions on Visualization and Computer Graphics 20 (2014) 1280–1292.","ieee":"M. Arikan, R. Preiner, C. Scheiblauer, S. Jeschke, and M. Wimmer, “Large-scale point-cloud visualization through localized textured surface reconstruction,” <i>IEEE Transactions on Visualization and Computer Graphics</i>, vol. 20, no. 9. IEEE, pp. 1280–1292, 2014.","chicago":"Arikan, Murat, Reinhold Preiner, Claus Scheiblauer, Stefan Jeschke, and Michael Wimmer. “Large-Scale Point-Cloud Visualization through Localized Textured Surface Reconstruction.” <i>IEEE Transactions on Visualization and Computer Graphics</i>. IEEE, 2014. <a href=\"https://doi.org/10.1109/TVCG.2014.2312011\">https://doi.org/10.1109/TVCG.2014.2312011</a>.","apa":"Arikan, M., Preiner, R., Scheiblauer, C., Jeschke, S., &#38; Wimmer, M. (2014). Large-scale point-cloud visualization through localized textured surface reconstruction. <i>IEEE Transactions on Visualization and Computer Graphics</i>. IEEE. <a href=\"https://doi.org/10.1109/TVCG.2014.2312011\">https://doi.org/10.1109/TVCG.2014.2312011</a>","ama":"Arikan M, Preiner R, Scheiblauer C, Jeschke S, Wimmer M. Large-scale point-cloud visualization through localized textured surface reconstruction. <i>IEEE Transactions on Visualization and Computer Graphics</i>. 2014;20(9):1280-1292. doi:<a href=\"https://doi.org/10.1109/TVCG.2014.2312011\">10.1109/TVCG.2014.2312011</a>","mla":"Arikan, Murat, et al. “Large-Scale Point-Cloud Visualization through Localized Textured Surface Reconstruction.” <i>IEEE Transactions on Visualization and Computer Graphics</i>, vol. 20, no. 9, IEEE, 2014, pp. 1280–92, doi:<a href=\"https://doi.org/10.1109/TVCG.2014.2312011\">10.1109/TVCG.2014.2312011</a>.","ista":"Arikan M, Preiner R, Scheiblauer C, Jeschke S, Wimmer M. 2014. Large-scale point-cloud visualization through localized textured surface reconstruction. IEEE Transactions on Visualization and Computer Graphics. 20(9), 1280–1292."},"volume":20,"year":"2014","doi":"10.1109/TVCG.2014.2312011","publication":"IEEE Transactions on Visualization and Computer Graphics","oa_version":"Submitted Version","intvolume":"        20","file_date_updated":"2020-07-14T12:45:20Z","oa":1,"file":[{"creator":"system","file_name":"IST-2016-573-v1+1_arikan-2014-pcvis-draft.pdf","file_id":"5297","date_updated":"2020-07-14T12:45:20Z","access_level":"open_access","content_type":"application/pdf","date_created":"2018-12-12T10:17:41Z","checksum":"5bf58942d2eb20adf03c7f9ea2e68124","file_size":13594598,"relation":"main_file"}],"date_updated":"2025-09-29T12:27:48Z","isi":1,"date_created":"2018-12-11T11:54:39Z","user_id":"317138e5-6ab7-11ef-aa6d-ffef3953e345","department":[{"_id":"ChWo"}],"_id":"1906","month":"09"}]