---
res:
  bibo_abstract:
  - Plant growth and development is well known to be both, flexible and dynamic. The
    high capacity for post-embryonic organ formation and tissue regeneration requires
    tightly regulated intercellular communication and coordinated tissue polarization.
    One of the most important drivers for patterning and polarity in plant development
    is the phytohormone auxin. Auxin has the unique characteristic to establish polarized
    channels for its own active directional cell to cell transport. This fascinating
    phenomenon is called auxin canalization. Those auxin transport channels are characterized
    by the expression and polar, subcellular localization of PIN auxin efflux carriers.
    PIN proteins have the ability to dynamically change their localization and auxin
    itself can affect this by interfering with trafficking. Most of the underlying
    molecular mechanisms of canalization still remain enigmatic. What is known so
    far is that canonical auxin signaling is indispensable but also other non-canonical
    signaling components are thought to play a role. In order to shed light into the
    mysteries auf auxin canalization this study revisits the branches of auxin signaling
    in detail. Further a new auxin analogue, PISA, is developed which triggers auxin-like
    responses but does not directly activate canonical transcriptional auxin signaling.
    We revisit the direct auxin effect on PIN trafficking where we found that, contradictory
    to previous observations, auxin is very specifically promoting endocytosis of
    PIN2 but has no overall effect on endocytosis. Further, we evaluate which cellular
    processes related to PIN subcellular dynamics are involved in the establishment
    of auxin conducting channels and the formation of vascular tissue. We are re-evaluating
    the function of AUXIN BINDING PROTEIN 1 (ABP1) and provide a comprehensive picture
    about its developmental phneotypes and involvement in auxin signaling and canalization.
    Lastly, we are focusing on the crosstalk between the hormone strigolactone (SL)
    and auxin and found that SL is interfering with essentially all processes involved
    in auxin canalization in a non-transcriptional manner. Lastly we identify a new
    way of SL perception and signaling which is emanating from mitochondria, is independent
    of canonical SL signaling and is modulating primary root growth.@eng
  bibo_authorlist:
  - foaf_Person:
      foaf_givenName: Michelle C
      foaf_name: Gallei, Michelle C
      foaf_surname: Gallei
      foaf_workInfoHomepage: http://www.librecat.org/personId=35A03822-F248-11E8-B48F-1D18A9856A87
    orcid: 0000-0003-1286-7368
  bibo_doi: 10.15479/at:ista:11626
  dct_date: 2022^xs_gYear
  dct_isPartOf:
  - http://id.crossref.org/issn/2663-337X
  - http://id.crossref.org/issn/978-3-99078-019-0
  dct_language: eng
  dct_publisher: Institute of Science and Technology Austria@
  dct_title: Auxin and strigolactone non-canonical signaling regulating development
    in Arabidopsis thaliana@
...