@article{21996,
  abstract     = {In this issue of Cell Host & Microbe, Osterman et al. discover aRES,1 a new family of bacterial immune proteins that deplete cellular NAD+, generating cleavage products that cannot be utilized by canonical phage NAD+ regeneration pathways. They identify the invader-specific trigger for aRES and characterize two distinct evolutionary countermeasures employed by phages to resist aRES.},
  author       = {Williams-Jones, Daniel and Bravo, Jack Peter Kelly},
  issn         = {1934-6069},
  journal      = {Cell Host & Microbe},
  number       = {6},
  pages        = {978--980},
  publisher    = {Elsevier},
  title        = {{NAD to the bone: How bacteria put phages under aRES-t … and how phages fight back}},
  doi          = {10.1016/j.chom.2026.05.013},
  volume       = {34},
  year         = {2026},
}