The time course of signaling at central glutamatergic synapses
Jonas PM. 2000. The time course of signaling at central glutamatergic synapses. Physiology. 15(2), 83–89.
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Journal Article
| Published
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Abstract
Glutamate is the main excitatory transmitter in the mammalian CNS, mediating fast synaptic transmission primarily by activation of AMPA-type glutamate receptor channels. Both synaptic structure and a cell-specific molecular switch in the AMPA receptor subunit expression are involved in the regulation of the synaptic signaling time course.
Publishing Year
Date Published
2000-04-01
Journal Title
Physiology
Publisher
American Physiological Society
Acknowledgement
I thank Drs. J.R.P. Geiger, K. Haverkampf, and B. Taskin for helpful discussions. This review is based on a lecture given in Lisbon in March 1998 on the occasion of the first award of the Medinfar European Prize in Physiology. My studies are supported by the Deutsche Forschungsgemeinschaft, the German Israeli Foundation, and the Human Frontiers Science Program Organization. I regret that many relevant papers could not be cited due to editorial restric-tions on the maximum number of references.
Volume
15
Issue
2
Page
83 - 89
ISSN
IST-REx-ID
Cite this
Jonas PM. The time course of signaling at central glutamatergic synapses. Physiology. 2000;15(2):83-89. doi:10.1152/physiologyonline.2000.15.2.83
Jonas, P. M. (2000). The time course of signaling at central glutamatergic synapses. Physiology. American Physiological Society. https://doi.org/10.1152/physiologyonline.2000.15.2.83
Jonas, Peter M. “The Time Course of Signaling at Central Glutamatergic Synapses.” Physiology. American Physiological Society, 2000. https://doi.org/10.1152/physiologyonline.2000.15.2.83.
P. M. Jonas, “The time course of signaling at central glutamatergic synapses,” Physiology, vol. 15, no. 2. American Physiological Society, pp. 83–89, 2000.
Jonas PM. 2000. The time course of signaling at central glutamatergic synapses. Physiology. 15(2), 83–89.
Jonas, Peter M. “The Time Course of Signaling at Central Glutamatergic Synapses.” Physiology, vol. 15, no. 2, American Physiological Society, 2000, pp. 83–89, doi:10.1152/physiologyonline.2000.15.2.83.
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